273 related articles for article (PubMed ID: 19635034)
1. Expression of constitutively-active aryl hydrocarbon receptor in T-cells enhances the down-regulation of CD62L, but does not alter expression of CD25 or suppress the allogeneic CTL response.
Funatake CJ; Ao K; Suzuki T; Murai H; Yamamoto M; Fujii-Kuriyama Y; Kerkvliet NI; Nohara K
J Immunotoxicol; 2009 Sep; 6(3):194-203. PubMed ID: 19635034
[TBL] [Abstract][Full Text] [Related]
2. T lymphocytes are direct, aryl hydrocarbon receptor (AhR)-dependent targets of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD): AhR expression in both CD4+ and CD8+ T cells is necessary for full suppression of a cytotoxic T lymphocyte response by TCDD.
Kerkvliet NI; Shepherd DM; Baecher-Steppan L
Toxicol Appl Pharmacol; 2002 Dec; 185(2):146-52. PubMed ID: 12490139
[TBL] [Abstract][Full Text] [Related]
3. 2,3,7,8-Tetrachlorodibenzo-p-dioxin alters the differentiation of alloreactive CD8+ T cells toward a regulatory T cell phenotype by a mechanism that is dependent on aryl hydrocarbon receptor in CD4+ T cells.
Funatake CJ; Marshall NB; Kerkvliet NI
J Immunotoxicol; 2008 Jan; 5(1):81-91. PubMed ID: 18382861
[TBL] [Abstract][Full Text] [Related]
4. Cutting edge: activation of the aryl hydrocarbon receptor by 2,3,7,8-tetrachlorodibenzo-p-dioxin generates a population of CD4+ CD25+ cells with characteristics of regulatory T cells.
Funatake CJ; Marshall NB; Steppan LB; Mourich DV; Kerkvliet NI
J Immunol; 2005 Oct; 175(7):4184-8. PubMed ID: 16177056
[TBL] [Abstract][Full Text] [Related]
5. Activation of aryl hydrocarbon receptor by TCDD prevents diabetes in NOD mice and increases Foxp3+ T cells in pancreatic lymph nodes.
Kerkvliet NI; Steppan LB; Vorachek W; Oda S; Farrer D; Wong CP; Pham D; Mourich DV
Immunotherapy; 2009 Jul; 1(4):539-47. PubMed ID: 20174617
[TBL] [Abstract][Full Text] [Related]
6. Activation of the aryl hydrocarbon receptor reduces the number of precursor and effector T cells, but preserves thymic CD4+CD25+Foxp3+ regulatory T cells.
Schulz VJ; Smit JJ; Bol-Schoenmakers M; van Duursen MB; van den Berg M; Pieters RH
Toxicol Lett; 2012 Nov; 215(2):100-9. PubMed ID: 23041608
[TBL] [Abstract][Full Text] [Related]
7. Suppression of acute graft-versus-host response by TCDD is independent of the CTLA-4-IFN-γ-IDO pathway.
Rohlman D; Punj S; Pennington J; Bradford S; Kerkvliet NI
Toxicol Sci; 2013 Sep; 135(1):81-90. PubMed ID: 23798565
[TBL] [Abstract][Full Text] [Related]
8. Suppression of experimental autoimmune uveoretinitis by inducing differentiation of regulatory T cells via activation of aryl hydrocarbon receptor.
Zhang L; Ma J; Takeuchi M; Usui Y; Hattori T; Okunuki Y; Yamakawa N; Kezuka T; Kuroda M; Goto H
Invest Ophthalmol Vis Sci; 2010 Apr; 51(4):2109-17. PubMed ID: 20007828
[TBL] [Abstract][Full Text] [Related]
9. Functional characterization and gene expression analysis of CD4+ CD25+ regulatory T cells generated in mice treated with 2,3,7,8-tetrachlorodibenzo-p-dioxin.
Marshall NB; Vorachek WR; Steppan LB; Mourich DV; Kerkvliet NI
J Immunol; 2008 Aug; 181(4):2382-91. PubMed ID: 18684927
[TBL] [Abstract][Full Text] [Related]
10. Constitutively active aryl hydrocarbon receptor expressed in T cells increases immunization-induced IFN-gamma production in mice but does not suppress T(h)2-cytokine production or antibody production.
Nohara K; Suzuki T; Ao K; Murai H; Miyamoto Y; Inouye K; Pan X; Motohashi H; Fujii-Kuriyama Y; Yamamoto M; Tohyama C
Int Immunol; 2009 Jul; 21(7):769-77. PubMed ID: 19461128
[TBL] [Abstract][Full Text] [Related]
11. Effects of Developmental Activation of the Aryl Hydrocarbon Receptor by 2,3,7,8-Tetrachlorodibenzo-p-dioxin on Long-term Self-renewal of Murine Hematopoietic Stem Cells.
Laiosa MD; Tate ER; Ahrenhoerster LS; Chen Y; Wang D
Environ Health Perspect; 2016 Jul; 124(7):957-65. PubMed ID: 26495820
[TBL] [Abstract][Full Text] [Related]
12. A chimeric aryl hydrocarbon receptor knockout mouse model indicates that aryl hydrocarbon receptor activation in hematopoietic cells contributes to the hepatic lesions induced by 2,3,7, 8-tetrachlorodibenzo-p-dioxin.
Thurmond TS; Silverstone AE; Baggs RB; Quimby FW; Staples JE; Gasiewicz TA
Toxicol Appl Pharmacol; 1999 Jul; 158(1):33-40. PubMed ID: 10387930
[TBL] [Abstract][Full Text] [Related]
13. Thymic alterations induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin are strictly dependent on aryl hydrocarbon receptor activation in hemopoietic cells.
Staples JE; Murante FG; Fiore NC; Gasiewicz TA; Silverstone AE
J Immunol; 1998 Apr; 160(8):3844-54. PubMed ID: 9558089
[TBL] [Abstract][Full Text] [Related]
14. Activation of the aryl hydrocarbon receptor diminishes the memory response to homotypic influenza virus infection but does not impair host resistance.
Lawrence BP; Vorderstrasse BA
Toxicol Sci; 2004 Jun; 79(2):304-14. PubMed ID: 14976337
[TBL] [Abstract][Full Text] [Related]
15. Aryl hydrocarbon receptor-deficient mice generate normal immune responses to model antigens and are resistant to TCDD-induced immune suppression.
Vorderstrasse BA; Steppan LB; Silverstone AE; Kerkvliet NI
Toxicol Appl Pharmacol; 2001 Mar; 171(3):157-64. PubMed ID: 11243915
[TBL] [Abstract][Full Text] [Related]
16. Inhibition of TC-1 cytokine production, effector cytotoxic T lymphocyte development and alloantibody production by 2,3,7,8-tetrachlorodibenzo-p-dioxin.
Kerkvliet NI; Baecher-Steppan L; Shepherd DM; Oughton JA; Vorderstrasse BA; DeKrey GK
J Immunol; 1996 Sep; 157(6):2310-9. PubMed ID: 8805628
[TBL] [Abstract][Full Text] [Related]
17. CTL hyporesponsiveness induced by 2,3,7, 8-tetrachlorodibenzo-p-dioxin: role of cytokines and apoptosis.
Prell RA; Dearstyne E; Steppan LG; Vella AT; Kerkvliet NI
Toxicol Appl Pharmacol; 2000 Aug; 166(3):214-21. PubMed ID: 10906285
[TBL] [Abstract][Full Text] [Related]
18. Aryl hydrocarbon receptor expression and activity in cerebellar granule neuroblasts: implications for development and dioxin neurotoxicity.
Williamson MA; Gasiewicz TA; Opanashuk LA
Toxicol Sci; 2005 Feb; 83(2):340-8. PubMed ID: 15537747
[TBL] [Abstract][Full Text] [Related]
19. Aryl hydrocarbon receptor activation in genital tubercle, palate, and other embryonic tissues in 2,3,7, 8-tetrachlorodibenzo-p-dioxin-responsive lacZ mice.
Willey JJ; Stripp BR; Baggs RB; Gasiewicz TA
Toxicol Appl Pharmacol; 1998 Jul; 151(1):33-44. PubMed ID: 9705885
[TBL] [Abstract][Full Text] [Related]
20. Treatment of mice with 2,3,7,8-tetrachlorodibenzo-p-dioxin leads to aryl hydrocarbon receptor-dependent nuclear translocation of NF-kappaB and expression of Fas ligand in thymic stromal cells and consequent apoptosis in T cells.
Camacho IA; Singh N; Hegde VL; Nagarkatti M; Nagarkatti PS
J Immunol; 2005 Jul; 175(1):90-103. PubMed ID: 15972635
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
