119 related articles for article (PubMed ID: 19915346)
1. [Molecular pathogenesis of leukemias].
Kurokawa M
Rinsho Ketsueki; 2009 Oct; 50(10):1382-8. PubMed ID: 19915346
[No Abstract] [Full Text] [Related]
2. Cooperation of BCR-ABL and AML1/MDS1/EVI1 in blocking myeloid differentiation and rapid induction of an acute myelogenous leukemia.
Cuenco GM; Ren R
Oncogene; 2001 Dec; 20(57):8236-48. PubMed ID: 11781838
[TBL] [Abstract][Full Text] [Related]
3. Leukemogenesis by CBF oncoproteins.
Friedman AD
Leukemia; 1999 Dec; 13(12):1932-42. PubMed ID: 10602413
[TBL] [Abstract][Full Text] [Related]
4. Intergenic splicing of MDS1 and EVI1 occurs in normal tissues as well as in myeloid leukemia and produces a new member of the PR domain family.
Fears S; Mathieu C; Zeleznik-Le N; Huang S; Rowley JD; Nucifora G
Proc Natl Acad Sci U S A; 1996 Feb; 93(4):1642-7. PubMed ID: 8643684
[TBL] [Abstract][Full Text] [Related]
5. Both AML1 and EVI1 oncogenic components are required for the cooperation of AML1/MDS1/EVI1 with BCR/ABL in the induction of acute myelogenous leukemia in mice.
Cuenco GM; Ren R
Oncogene; 2004 Jan; 23(2):569-79. PubMed ID: 14724585
[TBL] [Abstract][Full Text] [Related]
6. Chromatin remodeling and leukemia: new therapeutic paradigms.
Redner RL; Wang J; Liu JM
Blood; 1999 Jul; 94(2):417-28. PubMed ID: 10397708
[No Abstract] [Full Text] [Related]
7. Extending the repertoire of the mixed-lineage leukemia gene MLL in leukemogenesis.
Daser A; Rabbitts TH
Genes Dev; 2004 May; 18(9):965-74. PubMed ID: 15132992
[No Abstract] [Full Text] [Related]
8. RUNX1/EVI1, which blocks myeloid differentiation, inhibits CCAAT-enhancer binding protein alpha function.
Tokita K; Maki K; Mitani K
Cancer Sci; 2007 Nov; 98(11):1752-7. PubMed ID: 17894555
[TBL] [Abstract][Full Text] [Related]
9. Development of megakaryoblastic leukaemia in Runx1-Evi1 knock-in chimaeric mouse.
Maki K; Yamagata T; Yamazaki I; Oda H; Mitani K
Leukemia; 2006 Aug; 20(8):1458-60. PubMed ID: 16761014
[No Abstract] [Full Text] [Related]
10. The role of EVI1 in normal and leukemic cells.
Buonamici S; Chakraborty S; Senyuk V; Nucifora G
Blood Cells Mol Dis; 2003; 31(2):206-12. PubMed ID: 12972028
[TBL] [Abstract][Full Text] [Related]
11. AML1-Evi-1 specifically transforms hematopoietic stem cells through fusion of the entire Evi-1 sequence to AML1.
Takeshita M; Ichikawa M; Nitta E; Goyama S; Asai T; Ogawa S; Chiba S; Kurokawa M
Leukemia; 2008 Jun; 22(6):1241-9. PubMed ID: 18337762
[TBL] [Abstract][Full Text] [Related]
12. The AML1 gene: a transcription factor involved in the pathogenesis of myeloid and lymphoid leukemias.
Lo Coco F; Pisegna S; Diverio D
Haematologica; 1997; 82(3):364-70. PubMed ID: 9234595
[TBL] [Abstract][Full Text] [Related]
13. [Study of genes involved in chronic myeloid leukemia with t (3; 21) (q26; q22) in blastic crisis].
Liu XP; Zhang MR; Dai Y; Zhang L; Li R; Hao YS; Xiao ZJ
Zhonghua Xue Ye Xue Za Zhi; 2006 May; 27(5):310-3. PubMed ID: 16875579
[TBL] [Abstract][Full Text] [Related]
14. Deregulated expression of EVI1 defines a poor prognostic subset of MLL-rearranged acute myeloid leukemias: a study of the German-Austrian Acute Myeloid Leukemia Study Group and the Dutch-Belgian-Swiss HOVON/SAKK Cooperative Group.
Gröschel S; Schlenk RF; Engelmann J; Rockova V; Teleanu V; Kühn MW; Eiwen K; Erpelinck C; Havermans M; Lübbert M; Germing U; Schmidt-Wolf IG; Beverloo HB; Schuurhuis GJ; Ossenkoppele GJ; Schlegelberger B; Verdonck LF; Vellenga E; Verhoef G; Vandenberghe P; Pabst T; Bargetzi M; Krauter J; Ganser A; Valk PJ; Löwenberg B; Döhner K; Döhner H; Delwel R
J Clin Oncol; 2013 Jan; 31(1):95-103. PubMed ID: 23008312
[TBL] [Abstract][Full Text] [Related]
15. [Molecular mechanisms in the development of acute myeloid leukemia].
Ichikawa M; Kurokawa M
Nihon Rinsho; 2009 Oct; 67(10):1889-93. PubMed ID: 19860185
[TBL] [Abstract][Full Text] [Related]
16. Molecular cytogenetic analysis of gene rearrangements in childhood acute lymphoblastic leukemia.
Woo HY; Kim DW; Park H; Seong KW; Koo HH; Kim SH
J Korean Med Sci; 2005 Feb; 20(1):36-41. PubMed ID: 15716599
[TBL] [Abstract][Full Text] [Related]
17. Self-fusion of the ALL1 gene. A new genetic mechanism for acute leukemia.
Schichman SA; Canaani E; Croce CM
JAMA; 1995 Feb; 273(7):571-6. PubMed ID: 7837391
[No Abstract] [Full Text] [Related]
18. Activation of EVI1 transcription by the LEF1/β-catenin complex with p53-alteration in myeloid blast crisis of chronic myeloid leukemia.
Manachai N; Saito Y; Nakahata S; Bahirvani AG; Osato M; Morishita K
Biochem Biophys Res Commun; 2017 Jan; 482(4):994-1000. PubMed ID: 27908728
[TBL] [Abstract][Full Text] [Related]
19. The leukaemia-associated transcription factors EVI-1 and MDS1/EVI1 repress transcription and interact with histone deacetylase.
Vinatzer U; Taplick J; Seiser C; Fonatsch C; Wieser R
Br J Haematol; 2001 Sep; 114(3):566-73. PubMed ID: 11552981
[TBL] [Abstract][Full Text] [Related]
20. Consistent intergenic splicing and production of multiple transcripts between AML1 at 21q22 and unrelated genes at 3q26 in (3;21)(q26;q22) translocations.
Nucifora G; Begy CR; Kobayashi H; Roulston D; Claxton D; Pedersen-Bjergaard J; Parganas E; Ihle JN; Rowley JD
Proc Natl Acad Sci U S A; 1994 Apr; 91(9):4004-8. PubMed ID: 8171026
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]