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6. Elimination of proliferating cells unmasks the shift from senescence to quiescence caused by rapamycin. Leontieva OV; Demidenko ZN; Gudkov AV; Blagosklonny MV PLoS One; 2011; 6(10):e26126. PubMed ID: 22022534 [TBL] [Abstract][Full Text] [Related]
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9. Paradoxical suppression of cellular senescence by p53. Demidenko ZN; Korotchkina LG; Gudkov AV; Blagosklonny MV Proc Natl Acad Sci U S A; 2010 May; 107(21):9660-4. PubMed ID: 20457898 [TBL] [Abstract][Full Text] [Related]
10. Rapamycin inhibits the secretory phenotype of senescent cells by a Nrf2-independent mechanism. Wang R; Yu Z; Sunchu B; Shoaf J; Dang I; Zhao S; Caples K; Bradley L; Beaver LM; Ho E; Löhr CV; Perez VI Aging Cell; 2017 Jun; 16(3):564-574. PubMed ID: 28371119 [TBL] [Abstract][Full Text] [Related]
11. Hypoxia suppresses conversion from proliferative arrest to cellular senescence. Leontieva OV; Natarajan V; Demidenko ZN; Burdelya LG; Gudkov AV; Blagosklonny MV Proc Natl Acad Sci U S A; 2012 Aug; 109(33):13314-8. PubMed ID: 22847439 [TBL] [Abstract][Full Text] [Related]
12. DNA damaging agents and p53 do not cause senescence in quiescent cells, while consecutive re-activation of mTOR is associated with conversion to senescence. Leontieva OV; Blagosklonny MV Aging (Albany NY); 2010 Dec; 2(12):924-35. PubMed ID: 21212465 [TBL] [Abstract][Full Text] [Related]