142 related articles for article (PubMed ID: 20347420)
1. A soluble form of the pilus protein FimA targets the VDAC-hexokinase complex at mitochondria to suppress host cell apoptosis.
Sukumaran SK; Fu NY; Tin CB; Wan KF; Lee SS; Yu VC
Mol Cell; 2010 Mar; 37(6):768-83. PubMed ID: 20347420
[TBL] [Abstract][Full Text] [Related]
2. The VDAC1 N-terminus is essential both for apoptosis and the protective effect of anti-apoptotic proteins.
Abu-Hamad S; Arbel N; Calo D; Arzoine L; Israelson A; Keinan N; Ben-Romano R; Friedman O; Shoshan-Barmatz V
J Cell Sci; 2009 Jun; 122(Pt 11):1906-16. PubMed ID: 19461077
[TBL] [Abstract][Full Text] [Related]
3. In self-defence: hexokinase promotes voltage-dependent anion channel closure and prevents mitochondria-mediated apoptotic cell death.
Azoulay-Zohar H; Israelson A; Abu-Hamad S; Shoshan-Barmatz V
Biochem J; 2004 Jan; 377(Pt 2):347-55. PubMed ID: 14561215
[TBL] [Abstract][Full Text] [Related]
4. Alternative folding to a monomer or homopolymer is a common feature of the type 1 pilus subunit FimA from enteroinvasive bacteria.
Żyła DS; Prota AE; Capitani G; Glockshuber R
J Biol Chem; 2019 Jul; 294(27):10553-10563. PubMed ID: 31126987
[TBL] [Abstract][Full Text] [Related]
5. Hexokinase-I protection against apoptotic cell death is mediated via interaction with the voltage-dependent anion channel-1: mapping the site of binding.
Abu-Hamad S; Zaid H; Israelson A; Nahon E; Shoshan-Barmatz V
J Biol Chem; 2008 May; 283(19):13482-90. PubMed ID: 18308720
[TBL] [Abstract][Full Text] [Related]
6. The function of complexes between the outer mitochondrial membrane pore (VDAC) and the adenine nucleotide translocase in regulation of energy metabolism and apoptosis.
Vyssokikh MY; Brdiczka D
Acta Biochim Pol; 2003; 50(2):389-404. PubMed ID: 12833165
[TBL] [Abstract][Full Text] [Related]
7. The mitochondrial voltage-dependent anion channel 1 in tumor cells.
Shoshan-Barmatz V; Ben-Hail D; Admoni L; Krelin Y; Tripathi SS
Biochim Biophys Acta; 2015 Oct; 1848(10 Pt B):2547-75. PubMed ID: 25448878
[TBL] [Abstract][Full Text] [Related]
8. Mouse uterine epithelial apoptosis is associated with expression of mitochondrial voltage-dependent anion channels, release of cytochrome C from mitochondria, and the ratio of Bax to Bcl-2 or Bcl-X.
Takagi-Morishita Y; Yamada N; Sugihara A; Iwasaki T; Tsujimura T; Terada N
Biol Reprod; 2003 Apr; 68(4):1178-84. PubMed ID: 12606449
[TBL] [Abstract][Full Text] [Related]
9. Mapping the ruthenium red-binding site of the voltage-dependent anion channel-1.
Israelson A; Zaid H; Abu-Hamad S; Nahon E; Shoshan-Barmatz V
Cell Calcium; 2008 Feb; 43(2):196-204. PubMed ID: 17590433
[TBL] [Abstract][Full Text] [Related]
10. The voltage-dependent anion channel-1 modulates apoptotic cell death.
Zaid H; Abu-Hamad S; Israelson A; Nathan I; Shoshan-Barmatz V
Cell Death Differ; 2005 Jul; 12(7):751-60. PubMed ID: 15818409
[TBL] [Abstract][Full Text] [Related]
11. Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC.
Shimizu S; Narita M; Tsujimoto Y
Nature; 1999 Jun; 399(6735):483-7. PubMed ID: 10365962
[TBL] [Abstract][Full Text] [Related]
12. Dominant-negative VDAC1 mutants reveal oligomeric VDAC1 to be the active unit in mitochondria-mediated apoptosis.
Mader A; Abu-Hamad S; Arbel N; Gutiérrez-Aguilar M; Shoshan-Barmatz V
Biochem J; 2010 Jul; 429(1):147-55. PubMed ID: 20420578
[TBL] [Abstract][Full Text] [Related]
13. Intramolecular donor strand complementation in the E. coli type 1 pilus subunit FimA explains the existence of FimA monomers as off-pathway products of pilus assembly that inhibit host cell apoptosis.
Walczak MJ; Puorger C; Glockshuber R; Wider G
J Mol Biol; 2014 Feb; 426(3):542-9. PubMed ID: 24184277
[TBL] [Abstract][Full Text] [Related]
14. Bax and Bcl-xL independently regulate apoptotic changes of yeast mitochondria that require VDAC but not adenine nucleotide translocator.
Shimizu S; Shinohara Y; Tsujimoto Y
Oncogene; 2000 Sep; 19(38):4309-18. PubMed ID: 10980606
[TBL] [Abstract][Full Text] [Related]
15. Key regions of VDAC1 functioning in apoptosis induction and regulation by hexokinase.
Shoshan-Barmatz V; Zakar M; Rosenthal K; Abu-Hamad S
Biochim Biophys Acta; 2009 May; 1787(5):421-30. PubMed ID: 19094960
[TBL] [Abstract][Full Text] [Related]
16. Molecular model of hexokinase binding to the outer mitochondrial membrane porin (VDAC1): Implication for the design of new cancer therapies.
Rosano C
Mitochondrion; 2011 May; 11(3):513-9. PubMed ID: 21315184
[TBL] [Abstract][Full Text] [Related]
17. Knockout of VDAC1 in H9c2 Cells Promotes Oxidative Stress-Induced Cell Apoptosis through Decreased Mitochondrial Hexokinase II Binding and Enhanced Glycolytic Stress.
Yang M; Sun J; Stowe DF; Tajkhorshid E; Kwok WM; Camara AKS
Cell Physiol Biochem; 2020 Sep; 54(5):853-874. PubMed ID: 32901466
[TBL] [Abstract][Full Text] [Related]
18. The Voltage-dependent Anion Channel 1 Mediates Amyloid β Toxicity and Represents a Potential Target for Alzheimer Disease Therapy.
Smilansky A; Dangoor L; Nakdimon I; Ben-Hail D; Mizrachi D; Shoshan-Barmatz V
J Biol Chem; 2015 Dec; 290(52):30670-83. PubMed ID: 26542804
[TBL] [Abstract][Full Text] [Related]
19. Voltage-dependent anion channel 1 is involved in endostatin-induced endothelial cell apoptosis.
Yuan S; Fu Y; Wang X; Shi H; Huang Y; Song X; Li L; Song N; Luo Y
FASEB J; 2008 Aug; 22(8):2809-20. PubMed ID: 18381814
[TBL] [Abstract][Full Text] [Related]
20. Apoptosis is regulated by the VDAC1 N-terminal region and by VDAC oligomerization: release of cytochrome c, AIF and Smac/Diablo.
Shoshan-Barmatz V; Keinan N; Abu-Hamad S; Tyomkin D; Aram L
Biochim Biophys Acta; 2010; 1797(6-7):1281-91. PubMed ID: 20214874
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
