These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

286 related articles for article (PubMed ID: 20380835)

  • 1. The role of macrophage-derived urokinase plasminogen activator in myocardial infarct repair: urokinase attenuates ventricular remodeling.
    Minami E; Castellani C; Malchodi L; Deem J; Bertko K; Meznarich J; Dishmon M; Murry CE; Stempien-Otero A
    J Mol Cell Cardiol; 2010 Sep; 49(3):516-24. PubMed ID: 20380835
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Cardiac macrophages adopt profibrotic/M2 phenotype in infarcted hearts: Role of urokinase plasminogen activator.
    Carlson S; Helterline D; Asbe L; Dupras S; Minami E; Farris S; Stempien-Otero A
    J Mol Cell Cardiol; 2017 Jul; 108():42-49. PubMed ID: 27262672
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Overexpression of urokinase by macrophages or deficiency of plasminogen activator inhibitor type 1 causes cardiac fibrosis in mice.
    Moriwaki H; Stempien-Otero A; Kremen M; Cozen AE; Dichek DA
    Circ Res; 2004 Sep; 95(6):637-44. PubMed ID: 15297377
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Mechanisms of cardiac fibrosis induced by urokinase plasminogen activator.
    Stempien-Otero A; Plawman A; Meznarich J; Dyamenahalli T; Otsuka G; Dichek DA
    J Biol Chem; 2006 Jun; 281(22):15345-51. PubMed ID: 16554301
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Urokinase plasminogen activator induces pro-fibrotic/m2 phenotype in murine cardiac macrophages.
    Meznarich J; Malchodi L; Helterline D; Ramsey SA; Bertko K; Plummer T; Plawman A; Gold E; Stempien-Otero A
    PLoS One; 2013; 8(3):e57837. PubMed ID: 23536772
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Plasminogen activator inhibitor-1 suppresses profibrotic responses in fibroblasts from fibrotic lungs.
    Marudamuthu AS; Shetty SK; Bhandary YP; Karandashova S; Thompson M; Sathish V; Florova G; Hogan TB; Pabelick CM; Prakash YS; Tsukasaki Y; Fu J; Ikebe M; Idell S; Shetty S
    J Biol Chem; 2015 Apr; 290(15):9428-41. PubMed ID: 25648892
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Lgr4 Governs a Pro-Inflammatory Program in Macrophages to Antagonize Post-Infarction Cardiac Repair.
    Huang CK; Dai D; Xie H; Zhu Z; Hu J; Su M; Liu M; Lu L; Shen W; Ning G; Wang J; Zhang R; Yan X
    Circ Res; 2020 Sep; 127(8):953-973. PubMed ID: 32600176
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Bone marrow-derived CXCR4+ cells mobilized by macrophage colony-stimulating factor participate in the reduction of infarct area and improvement of cardiac remodeling after myocardial infarction in mice.
    Morimoto H; Takahashi M; Shiba Y; Izawa A; Ise H; Hongo M; Hatake K; Motoyoshi K; Ikeda U
    Am J Pathol; 2007 Sep; 171(3):755-66. PubMed ID: 17640962
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Protective role of 5-azacytidine on myocardial infarction is associated with modulation of macrophage phenotype and inhibition of fibrosis.
    Kim YS; Kang WS; Kwon JS; Hong MH; Jeong HY; Jeong HC; Jeong MH; Ahn Y
    J Cell Mol Med; 2014 Jun; 18(6):1018-27. PubMed ID: 24571348
    [TBL] [Abstract][Full Text] [Related]  

  • 10. THP-1 macrophage membrane-bound plasmin activity is up-regulated by transforming growth factor-beta 1 via increased expression of urokinase and the urokinase receptor.
    Falcone DJ; McCaffrey TA; Mathew J; McAdam K; Borth W
    J Cell Physiol; 1995 Aug; 164(2):334-43. PubMed ID: 7622580
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Tissue factor cytoplasmic domain exacerbates post-infarct left ventricular remodeling via orchestrating cardiac inflammation and angiogenesis.
    Chong SY; Zharkova O; Yatim SMJM; Wang X; Lim XC; Huang C; Tan CY; Jiang J; Ye L; Tan MS; Angeli V; Versteeg HH; Dewerchin M; Carmeliet P; Lam CSP; Chan MY; de Kleijn DPV; Wang JW
    Theranostics; 2021; 11(19):9243-9261. PubMed ID: 34646369
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Targeted deletion of class A macrophage scavenger receptor increases the risk of cardiac rupture after experimental myocardial infarction.
    Tsujita K; Kaikita K; Hayasaki T; Honda T; Kobayashi H; Sakashita N; Suzuki H; Kodama T; Ogawa H; Takeya M
    Circulation; 2007 Apr; 115(14):1904-11. PubMed ID: 17389263
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Selective abrogation of the uPA-uPAR interaction in vivo reveals a novel role in suppression of fibrin-associated inflammation.
    Connolly BM; Choi EY; Gårdsvoll H; Bey AL; Currie BM; Chavakis T; Liu S; Molinolo A; Ploug M; Leppla SH; Bugge TH
    Blood; 2010 Sep; 116(9):1593-603. PubMed ID: 20466854
    [TBL] [Abstract][Full Text] [Related]  

  • 14. The plasminogen-MMP system is more activated in the scar than in viable myocardium 3 months post-MI in the rat.
    Gaertner R; Jacob MP; Prunier F; Angles-Cano E; Mercadier JJ; Michel JB
    J Mol Cell Cardiol; 2005 Jan; 38(1):193-204. PubMed ID: 15623436
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Urokinase-type plasminogen activator and macrophages are required for skeletal muscle hypertrophy in mice.
    DiPasquale DM; Cheng M; Billich W; Huang SA; van Rooijen N; Hornberger TA; Koh TJ
    Am J Physiol Cell Physiol; 2007 Oct; 293(4):C1278-85. PubMed ID: 17652428
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Binding of anti-SSA antibodies to apoptotic fetal cardiocytes stimulates urokinase plasminogen activator (uPA)/uPA receptor-dependent activation of TGF-β and potentiates fibrosis.
    Briassouli P; Rifkin D; Clancy RM; Buyon JP
    J Immunol; 2011 Nov; 187(10):5392-401. PubMed ID: 22013113
    [TBL] [Abstract][Full Text] [Related]  

  • 17. uPA, uPAR and TGFβ₁ expression during early and late post myocardial infarction period in rat myocardium.
    Stavropoulou A; Philippou A; Halapas A; Sourla A; Pissimissis N; Koutsilieris M
    In Vivo; 2010; 24(5):647-52. PubMed ID: 20952728
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Early and delayed tranilast treatment reduces pathological fibrosis following myocardial infarction.
    See F; Watanabe M; Kompa AR; Wang BH; Boyle AJ; Kelly DJ; Gilbert RE; Krum H
    Heart Lung Circ; 2013 Feb; 22(2):122-32. PubMed ID: 22986349
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Exogenous CXCL4 infusion inhibits macrophage phagocytosis by limiting CD36 signalling to enhance post-myocardial infarction cardiac dilation and mortality.
    Lindsey ML; Jung M; Yabluchanskiy A; Cannon PL; Iyer RP; Flynn ER; DeLeon-Pennell KY; Valerio FM; Harrison CL; Ripplinger CM; Hall ME; Ma Y
    Cardiovasc Res; 2019 Feb; 115(2):395-408. PubMed ID: 30169632
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Urokinase plasminogen activator upregulates paraoxonase 2 expression in macrophages via an NADPH oxidase-dependent mechanism.
    Fuhrman B; Khateeb J; Shiner M; Nitzan O; Karry R; Volkova N; Aviram M
    Arterioscler Thromb Vasc Biol; 2008 Jul; 28(7):1361-7. PubMed ID: 18436804
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 15.