311 related articles for article (PubMed ID: 20430035)
1. The opposing effects of CCN2 and CCN5 on the development of cardiac hypertrophy and fibrosis.
Yoon PO; Lee MA; Cha H; Jeong MH; Kim J; Jang SP; Choi BY; Jeong D; Yang DK; Hajjar RJ; Park WJ
J Mol Cell Cardiol; 2010 Aug; 49(2):294-303. PubMed ID: 20430035
[TBL] [Abstract][Full Text] [Related]
2. CCN2 and CCN5 exerts opposing effect on fibroblast proliferation and transdifferentiation induced by TGF-β.
Xu H; Li P; Liu M; Liu C; Sun Z; Guo X; Zhang Y
Clin Exp Pharmacol Physiol; 2015 Nov; 42(11):1207-19. PubMed ID: 26218313
[TBL] [Abstract][Full Text] [Related]
3. CCN5 overexpression inhibits profibrotic phenotypes via the PI3K/Akt signaling pathway in lung fibroblasts isolated from patients with idiopathic pulmonary fibrosis and in an in vivo model of lung fibrosis.
Zhang L; Li Y; Liang C; Yang W
Int J Mol Med; 2014 Feb; 33(2):478-86. PubMed ID: 24276150
[TBL] [Abstract][Full Text] [Related]
4. CCN5 attenuates profibrotic phenotypes of fibroblasts through the Smad6-CCN2 pathway: Potential role in epidural fibrosis.
Xu H; Liu C; Sun Z; Guo X; Zhang Y; Liu M; Li P
Int J Mol Med; 2015 Jul; 36(1):123-9. PubMed ID: 25901787
[TBL] [Abstract][Full Text] [Related]
5. CCN2/CTGF attenuates myocardial hypertrophy and cardiac dysfunction upon chronic pressure-overload.
Gravning J; Ahmed MS; von Lueder TG; Edvardsen T; Attramadal H
Int J Cardiol; 2013 Oct; 168(3):2049-56. PubMed ID: 23452880
[TBL] [Abstract][Full Text] [Related]
6. Cucurbitacin I Attenuates Cardiomyocyte Hypertrophy via Inhibition of Connective Tissue Growth Factor (CCN2) and TGF- β/Smads Signalings.
Jeong MH; Kim SJ; Kang H; Park KW; Park WJ; Yang SY; Yang DK
PLoS One; 2015; 10(8):e0136236. PubMed ID: 26296085
[TBL] [Abstract][Full Text] [Related]
7. The matricellular protein CCN5 prevents adverse atrial structural and electrical remodelling.
Lee MA; Raad N; Song MH; Yoo J; Lee M; Jang SP; Kwak TH; Kook H; Choi EK; Cha TJ; Hajjar RJ; Jeong D; Park WJ
J Cell Mol Med; 2020 Oct; 24(20):11768-11778. PubMed ID: 32885578
[TBL] [Abstract][Full Text] [Related]
8. The carboxyl-terminal TSP1-homology domain is the biologically active effector peptide of matricellular protein CCN5 that counteracts profibrotic CCN2.
Zolfaghari S; Kaasbøll OJ; Monsen VT; Sredic B; Hagelin EMV; Attramadal H
J Biol Chem; 2023 Jan; 299(1):102803. PubMed ID: 36529291
[TBL] [Abstract][Full Text] [Related]
9. Matricellular Protein CCN5 Reverses Established Cardiac Fibrosis.
Jeong D; Lee MA; Li Y; Yang DK; Kho C; Oh JG; Hong G; Lee A; Song MH; LaRocca TJ; Chen J; Liang L; Mitsuyama S; D'Escamard V; Kovacic JC; Kwak TH; Hajjar RJ; Park WJ
J Am Coll Cardiol; 2016 Apr; 67(13):1556-1568. PubMed ID: 27150688
[TBL] [Abstract][Full Text] [Related]
10. CTGF/CCN2 is an autocrine regulator of cardiac fibrosis.
Dorn LE; Petrosino JM; Wright P; Accornero F
J Mol Cell Cardiol; 2018 Aug; 121():205-211. PubMed ID: 30040954
[TBL] [Abstract][Full Text] [Related]
11. Single-target RNA interference for the blockade of multiple interacting proinflammatory and profibrotic pathways in cardiac fibroblasts.
Tank J; Lindner D; Wang X; Stroux A; Gilke L; Gast M; Zietsch C; Skurk C; Scheibenbogen C; Klingel K; Lassner D; Kühl U; Schultheiss HP; Westermann D; Poller W
J Mol Cell Cardiol; 2014 Jan; 66():141-56. PubMed ID: 24239602
[TBL] [Abstract][Full Text] [Related]
12. Yin and Yang Part Deux: CCN5 inhibits the pro-fibrotic effects of CCN2.
Leask A
J Cell Commun Signal; 2010 Oct; 4(3):155-6. PubMed ID: 21063505
[TBL] [Abstract][Full Text] [Related]
13. Wnt signaling is critical for maladaptive cardiac hypertrophy and accelerates myocardial remodeling.
Malekar P; Hagenmueller M; Anyanwu A; Buss S; Streit MR; Weiss CS; Wolf D; Riffel J; Bauer A; Katus HA; Hardt SE
Hypertension; 2010 Apr; 55(4):939-45. PubMed ID: 20177000
[TBL] [Abstract][Full Text] [Related]
14. Zerumbone prevents pressure overload-induced left ventricular systolic dysfunction by inhibiting cardiac hypertrophy and fibrosis.
Sari N; Katanasaka Y; Sugiyama Y; Sunagawa Y; Miyazaki Y; Funamoto M; Shimizu S; Shimizu K; Murakami A; Mori K; Wada H; Hasegawa K; Morimoto T
Phytomedicine; 2021 Nov; 92():153744. PubMed ID: 34563985
[TBL] [Abstract][Full Text] [Related]
15. Growth/differentiation factor 1 alleviates pressure overload-induced cardiac hypertrophy and dysfunction.
Zhang Y; Zhang XF; Gao L; Liu Y; Jiang DS; Chen K; Yang Q; Fan GC; Zhang XD; Huang C
Biochim Biophys Acta; 2014 Feb; 1842(2):232-44. PubMed ID: 24275554
[TBL] [Abstract][Full Text] [Related]
16. Diacylglycerol kinase-epsilon restores cardiac dysfunction under chronic pressure overload: a new specific regulator of Galpha(q) signaling cascade.
Niizeki T; Takeishi Y; Kitahara T; Arimoto T; Ishino M; Bilim O; Suzuki S; Sasaki T; Nakajima O; Walsh RA; Goto K; Kubota I
Am J Physiol Heart Circ Physiol; 2008 Jul; 295(1):H245-55. PubMed ID: 18487437
[TBL] [Abstract][Full Text] [Related]
17. Cardiac-specific overexpression of farnesyl pyrophosphate synthase induces cardiac hypertrophy and dysfunction in mice.
Yang J; Mou Y; Wu T; Ye Y; Jiang JC; Zhao CZ; Zhu HH; Du CQ; Zhou L; Hu SJ
Cardiovasc Res; 2013 Mar; 97(3):490-9. PubMed ID: 23180723
[TBL] [Abstract][Full Text] [Related]
18. Targeted disruption of Hspa4 gene leads to cardiac hypertrophy and fibrosis.
Mohamed BA; Barakat AZ; Zimmermann WH; Bittner RE; Mühlfeld C; Hünlich M; Engel W; Maier LS; Adham IM
J Mol Cell Cardiol; 2012 Oct; 53(4):459-68. PubMed ID: 22884543
[TBL] [Abstract][Full Text] [Related]
19. Increased connective tissue growth factor relative to brain natriuretic peptide as a determinant of myocardial fibrosis.
Koitabashi N; Arai M; Kogure S; Niwano K; Watanabe A; Aoki Y; Maeno T; Nishida T; Kubota S; Takigawa M; Kurabayashi M
Hypertension; 2007 May; 49(5):1120-7. PubMed ID: 17372041
[TBL] [Abstract][Full Text] [Related]
20. CCN2 plays a key role in extracellular matrix gene expression in severe hypertrophic cardiomyopathy and heart failure.
Tsoutsman T; Wang X; Garchow K; Riser B; Twigg S; Semsarian C
J Mol Cell Cardiol; 2013 Sep; 62():164-78. PubMed ID: 23756156
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]