296 related articles for article (PubMed ID: 20523339)
1. Ginsenoside Rb1 protects cardiomyocytes against CoCl2-induced apoptosis in neonatal rats by inhibiting mitochondria permeability transition pore opening.
Kong HL; Li ZQ; Zhao YJ; Zhao SM; Zhu L; Li T; Fu Y; Li HJ
Acta Pharmacol Sin; 2010 Jun; 31(6):687-95. PubMed ID: 20523339
[TBL] [Abstract][Full Text] [Related]
2. Hydrogen sulfide protects cardiomyocytes from hypoxia/reoxygenation-induced apoptosis by preventing GSK-3beta-dependent opening of mPTP.
Yao LL; Huang XW; Wang YG; Cao YX; Zhang CC; Zhu YC
Am J Physiol Heart Circ Physiol; 2010 May; 298(5):H1310-9. PubMed ID: 20154265
[TBL] [Abstract][Full Text] [Related]
3. Anti-hypoxic effect of ginsenoside Rbl on neonatal rat cardiomyocytes is mediated through the specific activation of glucose transporter-4 ex vivo.
Kong HL; Wang JP; Li ZQ; Zhao SM; Dong J; Zhang WW
Acta Pharmacol Sin; 2009 Apr; 30(4):396-403. PubMed ID: 19305424
[TBL] [Abstract][Full Text] [Related]
4. Cardioprotection of ginsenoside Rb1 against ischemia/reperfusion injury is associated with mitochondrial permeability transition pore opening inhibition.
Li YH; Li YY; Fan GW; Yu JH; Duan ZZ; Wang LY; Yu B
Chin J Integr Med; 2016 Jan; ():. PubMed ID: 26740222
[TBL] [Abstract][Full Text] [Related]
5. Ginsenoside Rb1 Ameliorates Autophagy of Hypoxia Cardiomyocytes from Neonatal Rats via AMP-Activated Protein Kinase Pathway.
Dai SN; Hou AJ; Zhao SM; Chen XM; Huang HT; Chen BH; Kong HL
Chin J Integr Med; 2019 Jul; 25(7):521-528. PubMed ID: 30088211
[TBL] [Abstract][Full Text] [Related]
6. Anti-hypertrophic effect of NHE-1 inhibition involves GSK-3beta-dependent attenuation of mitochondrial dysfunction.
Javadov S; Rajapurohitam V; Kilić A; Zeidan A; Choi A; Karmazyn M
J Mol Cell Cardiol; 2009 Jun; 46(6):998-1007. PubMed ID: 19318234
[TBL] [Abstract][Full Text] [Related]
7. Apelin-APJ effects of ginsenoside-Rb1 depending on hypoxia-induced factor 1α in hypoxia neonatal cardiomyocytes.
Kong HL; Li ZQ; Zhao SM; Yuan L; Miao ZL; Liu Y; Guan RM
Chin J Integr Med; 2015 Feb; 21(2):139-46. PubMed ID: 24893658
[TBL] [Abstract][Full Text] [Related]
8. Salvianolic Acid A Protects Neonatal Cardiomyocytes Against Hypoxia/Reoxygenation-Induced Injury by Preserving Mitochondrial Function and Activating Akt/GSK-3β Signals.
Li XL; Fan JP; Liu JX; Liang LN
Chin J Integr Med; 2019 Jan; 25(1):23-30. PubMed ID: 28197936
[TBL] [Abstract][Full Text] [Related]
9. Mechanism for resveratrol-induced cardioprotection against reperfusion injury involves glycogen synthase kinase 3beta and mitochondrial permeability transition pore.
Xi J; Wang H; Mueller RA; Norfleet EA; Xu Z
Eur J Pharmacol; 2009 Feb; 604(1-3):111-6. PubMed ID: 19135050
[TBL] [Abstract][Full Text] [Related]
10. Cardioprotective effect of morphine and a blocker of glycogen synthase kinase 3 beta, SB216763 [3-(2,4-dichlorophenyl)-4(1-methyl-1H-indol-3-yl)-1H-pyrrole-2,5-dione], via inhibition of the mitochondrial permeability transition pore.
Obame FN; Plin-Mercier C; Assaly R; Zini R; Dubois-Randé JL; Berdeaux A; Morin D
J Pharmacol Exp Ther; 2008 Jul; 326(1):252-8. PubMed ID: 18434587
[TBL] [Abstract][Full Text] [Related]
11. Accelerated recovery of mitochondrial membrane potential by GSK-3β inactivation affords cardiomyocytes protection from oxidant-induced necrosis.
Sunaga D; Tanno M; Kuno A; Ishikawa S; Ogasawara M; Yano T; Miki T; Miura T
PLoS One; 2014; 9(11):e112529. PubMed ID: 25390651
[TBL] [Abstract][Full Text] [Related]
12. Morphine prevents the mitochondrial permeability transition pore opening through NO/cGMP/PKG/Zn2+/GSK-3beta signal pathway in cardiomyocytes.
Xi J; Tian W; Zhang L; Jin Y; Xu Z
Am J Physiol Heart Circ Physiol; 2010 Feb; 298(2):H601-7. PubMed ID: 19966058
[TBL] [Abstract][Full Text] [Related]
13. Sappanone A alleviates hypoxia/reoxygenation-induced cardiomyocytes injury through inhibition of mitochondrial apoptosis and activation of PI3K-Akt-Gsk-3β pathway.
Shi X; Tao G; Ji L; Tian G
Biosci Rep; 2020 Feb; 40(2):. PubMed ID: 32095825
[TBL] [Abstract][Full Text] [Related]
14. [Resveratrol attenuates oxidant-induced mitochondrial damage in embryonic rat cardiomyocytes via inactivating GSK-3β].
He YG; Sun YJ; Xie YX; Zheng H; Zhang YD; Guo J; Xi JK
Zhonghua Xin Xue Guan Bing Za Zhi; 2012 Oct; 40(10):858-63. PubMed ID: 23302675
[TBL] [Abstract][Full Text] [Related]
15. Erinacine Facilitates the Opening of the Mitochondrial Permeability Transition Pore Through the Inhibition of the PI3K/ Akt/GSK-3β Signaling Pathway in Human Hepatocellular Carcinoma.
Zhou LJ; Mo YB; Bu X; Wang JJ; Bai J; Zhang JW; Cheng AB; Ma JH; Wang YW; Xie YX
Cell Physiol Biochem; 2018; 50(3):851-867. PubMed ID: 30355923
[TBL] [Abstract][Full Text] [Related]
16. Rosuvastatin protects isolated hearts against ischemia-reperfusion injury: role of Akt-GSK-3β, metabolic environment, and mitochondrial permeability transition pore.
Vélez DE; Mestre-Cordero VE; Hermann R; Perego J; Harriet S; Fernandez-Pazos MLM; Mourglia J; Marina-Prendes MG
J Physiol Biochem; 2020 Feb; 76(1):85-98. PubMed ID: 31916218
[TBL] [Abstract][Full Text] [Related]
17. N6-(3-iodobenzyl)-adenosine-5'-N-methylcarboxamide confers cardioprotection at reperfusion by inhibiting mitochondrial permeability transition pore opening via glycogen synthase kinase 3 beta.
Park SS; Zhao H; Jang Y; Mueller RA; Xu Z
J Pharmacol Exp Ther; 2006 Jul; 318(1):124-31. PubMed ID: 16611852
[TBL] [Abstract][Full Text] [Related]
18. Rosuvastatin postconditioning protects isolated hearts against ischemia-reperfusion injury: The role of radical oxygen species, PI3K-Akt-GSK-3β pathway, and mitochondrial permeability transition pore.
Liu CW; Yang F; Cheng SZ; Liu Y; Wan LH; Cong HL
Cardiovasc Ther; 2017 Feb; 35(1):3-9. PubMed ID: 27580017
[TBL] [Abstract][Full Text] [Related]
19. Ethanol prevents oxidant-induced mitochondrial permeability transition pore opening in cardiac cells.
Zhou K; Zhang L; Xi J; Tian W; Xu Z
Alcohol Alcohol; 2009; 44(1):20-4. PubMed ID: 19033378
[TBL] [Abstract][Full Text] [Related]
20. Endoplasmic reticulum stress in diabetic hearts abolishes erythropoietin-induced myocardial protection by impairment of phospho-glycogen synthase kinase-3beta-mediated suppression of mitochondrial permeability transition.
Miki T; Miura T; Hotta H; Tanno M; Yano T; Sato T; Terashima Y; Takada A; Ishikawa S; Shimamoto K
Diabetes; 2009 Dec; 58(12):2863-72. PubMed ID: 19755525
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
