BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

233 related articles for article (PubMed ID: 20626435)

  • 1. Downregulation and upregulation of glial connexins may cause synaptic imbalances responsible for the pathophysiology of bipolar disorder.
    Mitterauer BJ
    CNS Neurosci Ther; 2011 Oct; 17(5):281-93. PubMed ID: 20626435
    [TBL] [Abstract][Full Text] [Related]  

  • 2. The syncytiopathy hypothesis of depression: downregulation of glial connexins may protract synaptic information processing and cause memory impairment.
    Mitterauer BJ
    Med Hypotheses; 2010 Mar; 74(3):497-502. PubMed ID: 19900766
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Imbalance of glial-neuronal interaction in synapses: a possible mechanism of the pathophysiology of bipolar disorder.
    Mitterauer B
    Neuroscientist; 2004 Jun; 10(3):199-206. PubMed ID: 15155059
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Glial-Neuronal Interaction in Synapses: A Possible Mechanism of the Pathophysiology of Bipolar Disorder.
    Wartchow KM; Scaini G; Quevedo J
    Adv Exp Med Biol; 2023; 1411():191-208. PubMed ID: 36949311
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Possible role of glia in cognitive impairment in schizophrenia.
    Mitterauer BJ
    CNS Neurosci Ther; 2011 Oct; 17(5):333-44. PubMed ID: 21951367
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Balancing and imbalancing effects of astrocytic receptors in tripartite synapses. Common pathophysiological model of mental disorders and epilepsy.
    Mitterauer BJ
    Med Hypotheses; 2015 Apr; 84(4):315-20. PubMed ID: 25655220
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Synaptic imbalances in endogenous psychoses.
    Mitterauer BJ
    Biosystems; 2010 May; 100(2):113-21. PubMed ID: 20176076
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Nonfunctional glial proteins in tripartite synapses: a pathophysiological model of schizophrenia.
    Mitterauer B
    Neuroscientist; 2005 Jun; 11(3):192-8. PubMed ID: 16013114
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Glial pathology in bipolar disorder: potential therapeutic implications.
    Dong XH; Zhen XC
    CNS Neurosci Ther; 2015 May; 21(5):393-7. PubMed ID: 25753128
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Glial ATP and Large Pore Channels Modulate Synaptic Strength in Response to Chronic Inactivity.
    Rafael A; Cairus A; Tizzoni M; Abudara V; Vitureira N
    Mol Neurobiol; 2020 Jun; 57(6):2856-2869. PubMed ID: 32388797
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Emerging complexities in identity and function of glial connexins.
    Theis M; Söhl G; Eiberger J; Willecke K
    Trends Neurosci; 2005 Apr; 28(4):188-95. PubMed ID: 15808353
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Disorders of human consciousness in the Tri-partite synapses.
    Miterauer B; Baer W
    Med Hypotheses; 2020 Mar; 136():109523. PubMed ID: 31927223
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Physiological Functions of Glial Cell Hemichannels.
    Orellana JA
    Adv Exp Med Biol; 2016; 949():93-108. PubMed ID: 27714686
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Hemichannels: new pathways for gliotransmitter release.
    Montero TD; Orellana JA
    Neuroscience; 2015 Feb; 286():45-59. PubMed ID: 25475761
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Glial cells as key elements in the pathophysiology and treatment of bipolar disorder.
    Keshavarz M
    Acta Neuropsychiatr; 2017 Jun; 29(3):140-152. PubMed ID: 27772534
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Altered expression of apoptotic factors and synaptic markers in postmortem brain from bipolar disorder patients.
    Kim HW; Rapoport SI; Rao JS
    Neurobiol Dis; 2010 Mar; 37(3):596-603. PubMed ID: 19945534
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Cellular plasticity cascades in the pathophysiology and treatment of bipolar disorder.
    Schloesser RJ; Huang J; Klein PS; Manji HK
    Neuropsychopharmacology; 2008 Jan; 33(1):110-33. PubMed ID: 17912251
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Glial Connexins and Pannexins in the Healthy and Diseased Brain.
    Giaume C; Naus CC; Sáez JC; Leybaert L
    Physiol Rev; 2021 Jan; 101(1):93-145. PubMed ID: 32326824
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Connexin 30 controls the extension of astrocytic processes into the synaptic cleft through an unconventional non-channel function.
    Clasadonte J; Haydon PG
    Neurosci Bull; 2014 Dec; 30(6):1045-1048. PubMed ID: 25342075
    [No Abstract]   [Full Text] [Related]  

  • 20. Loss of function of glial gap junctions may cause severe cognitive impairments in schizophrenia.
    Mitterauer B
    Med Hypotheses; 2009 Sep; 73(3):393-7. PubMed ID: 19435655
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 12.