These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
101 related articles for article (PubMed ID: 20837142)
41. Nonamyloid aggregates arising from mature copper/zinc superoxide dismutases resemble those observed in amyotrophic lateral sclerosis. Hwang YM; Stathopulos PB; Dimmick K; Yang H; Badiei HR; Tong MS; Rumfeldt JA; Chen P; Karanassios V; Meiering EM J Biol Chem; 2010 Dec; 285(53):41701-11. PubMed ID: 20974846 [TBL] [Abstract][Full Text] [Related]
42. Cu/Zn superoxide dismutase (SOD1) mutations associated with familial amyotrophic lateral sclerosis (ALS) affect cellular free radical release in the presence of oxidative stress. Cookson MR; Menzies FM; Manning P; Eggett CJ; Figlewicz DA; McNeil CJ; Shaw PJ Amyotroph Lateral Scler Other Motor Neuron Disord; 2002 Jun; 3(2):75-85. PubMed ID: 12215229 [TBL] [Abstract][Full Text] [Related]
43. Oligomerization of Cu,Zn-Superoxide Dismutase (SOD1) by Docosahexaenoic Acid and Its Hydroperoxides In Vitro: Aggregation Dependence on Fatty Acid Unsaturation and Thiols. Appolinário PP; Medinas DB; Chaves-Filho AB; Genaro-Mattos TC; Cussiol JR; Netto LE; Augusto O; Miyamoto S PLoS One; 2015; 10(4):e0125146. PubMed ID: 25928076 [TBL] [Abstract][Full Text] [Related]
44. Effects of Cellular Pathway Disturbances on Misfolded Superoxide Dismutase-1 in Fibroblasts Derived from ALS Patients. Keskin I; Forsgren E; Lange DJ; Weber M; Birve A; Synofzik M; Gilthorpe JD; Andersen PM; Marklund SL PLoS One; 2016; 11(2):e0150133. PubMed ID: 26919046 [TBL] [Abstract][Full Text] [Related]
45. Pro-oxidant copper-binding mode of the Apo form of ALS-linked SOD1 mutant H43R denatured at physiological temperature. Fujimaki N; Kitamura F; Takeuchi H Biochemistry; 2013 Aug; 52(31):5184-94. PubMed ID: 23837654 [TBL] [Abstract][Full Text] [Related]
46. Early steps in oxidation-induced SOD1 misfolding: implications for non-amyloid protein aggregation in familial ALS. Mulligan VK; Kerman A; Laister RC; Sharda PR; Arslan PE; Chakrabartty A J Mol Biol; 2012 Aug; 421(4-5):631-52. PubMed ID: 22542526 [TBL] [Abstract][Full Text] [Related]
47. Extracellular aggregated Cu/Zn superoxide dismutase activates microglia to give a cytotoxic phenotype. Roberts K; Zeineddine R; Corcoran L; Li W; Campbell IL; Yerbury JJ Glia; 2013 Mar; 61(3):409-19. PubMed ID: 23281114 [TBL] [Abstract][Full Text] [Related]
50. An RNAi strategy for treatment of amyotrophic lateral sclerosis caused by mutant Cu,Zn superoxide dismutase. Xia XG; Zhou H; Zhou S; Yu Y; Wu R; Xu Z J Neurochem; 2005 Jan; 92(2):362-7. PubMed ID: 15663483 [TBL] [Abstract][Full Text] [Related]
51. Local unfolding of Cu, Zn superoxide dismutase monomer determines the morphology of fibrillar aggregates. Ding F; Furukawa Y; Nukina N; Dokholyan NV J Mol Biol; 2012 Aug; 421(4-5):548-60. PubMed ID: 22210350 [TBL] [Abstract][Full Text] [Related]
52. Mechanism for transforming cytosolic SOD1 into integral membrane proteins of organelles by ALS-causing mutations. Lim L; Lee X; Song J Biochim Biophys Acta; 2015 Jan; 1848(1 Pt A):1-7. PubMed ID: 25306968 [TBL] [Abstract][Full Text] [Related]
53. Copper-zinc superoxide dismutase and amyotrophic lateral sclerosis. Valentine JS; Doucette PA; Zittin Potter S Annu Rev Biochem; 2005; 74():563-93. PubMed ID: 15952898 [TBL] [Abstract][Full Text] [Related]
54. Cu/Zn Superoxide Dismutase Forms Amyloid Fibrils under Near-Physiological Quiescent Conditions: The Roles of Disulfide Bonds and Effects of Denaturant. Khan MAI; Respondek M; Kjellström S; Deep S; Linse S; Akke M ACS Chem Neurosci; 2017 Sep; 8(9):2019-2026. PubMed ID: 28585802 [TBL] [Abstract][Full Text] [Related]
55. A structural modeling approach for the understanding of initiation and elongation of ALS-linked superoxide dismutase fibrils. Falconi M; Iacovelli F; Desideri A J Mol Model; 2013 Sep; 19(9):3695-704. PubMed ID: 23780345 [TBL] [Abstract][Full Text] [Related]
56. Functional features cause misfolding of the ALS-provoking enzyme SOD1. Nordlund A; Leinartaite L; Saraboji K; Aisenbrey C; Gröbner G; Zetterström P; Danielsson J; Logan DT; Oliveberg M Proc Natl Acad Sci U S A; 2009 Jun; 106(24):9667-72. PubMed ID: 19497878 [TBL] [Abstract][Full Text] [Related]
57. Trifluoroethanol Partially Unfolds G93A SOD1 Leading to Protein Aggregation: A Study by Native Mass Spectrometry and FPOP Protein Footprinting. Niu B; Mackness BC; Zitzewitz JA; Matthews CR; Gross ML Biochemistry; 2020 Oct; 59(39):3650-3659. PubMed ID: 32924445 [TBL] [Abstract][Full Text] [Related]
58. Computational insight into in silico analysis and molecular dynamics simulation of the dimer interface residues of ALS-linked hSOD1 forms in apo/holo states: a combined experimental and bioinformatic perspective. Zaji HD; Seyedalipour B; Hanun HM; Baziyar P; Hosseinkhani S; Akhlaghi M 3 Biotech; 2023 Mar; 13(3):92. PubMed ID: 36845075 [TBL] [Abstract][Full Text] [Related]
59. Mutation/metal deficiency in the "electrostatic loop" enhanced aggregation process in apo/holo SOD1 variants: implications for ALS diseases. Ashkaran F; Seyedalipour B; Baziyar P; Hosseinkhani S BMC Chem; 2024 Sep; 18(1):177. PubMed ID: 39300574 [TBL] [Abstract][Full Text] [Related]
60. Oxidative modifications and aggregation of Cu,Zn-superoxide dismutase associated with Alzheimer and Parkinson diseases. Choi J; Rees HD; Weintraub ST; Levey AI; Chin LS; Li L J Biol Chem; 2005 Mar; 280(12):11648-55. PubMed ID: 15659387 [TBL] [Abstract][Full Text] [Related] [Previous] [Next] [New Search]