278 related articles for article (PubMed ID: 21097884)
1. Cooperation of RAD51 and RAD54 in regression of a model replication fork.
Bugreev DV; Rossi MJ; Mazin AV
Nucleic Acids Res; 2011 Mar; 39(6):2153-64. PubMed ID: 21097884
[TBL] [Abstract][Full Text] [Related]
2. Strand invasion by HLTF as a mechanism for template switch in fork rescue.
Burkovics P; Sebesta M; Balogh D; Haracska L; Krejci L
Nucleic Acids Res; 2014 Feb; 42(3):1711-20. PubMed ID: 24198246
[TBL] [Abstract][Full Text] [Related]
3. Rad51 protein stimulates the branch migration activity of Rad54 protein.
Rossi MJ; Mazin AV
J Biol Chem; 2008 Sep; 283(36):24698-706. PubMed ID: 18617519
[TBL] [Abstract][Full Text] [Related]
4. Regression of replication forks stalled by leading-strand template damage: I. Both RecG and RuvAB catalyze regression, but RuvC cleaves the holliday junctions formed by RecG preferentially.
Gupta S; Yeeles JT; Marians KJ
J Biol Chem; 2014 Oct; 289(41):28376-87. PubMed ID: 25138216
[TBL] [Abstract][Full Text] [Related]
5. Fork Cleavage-Religation Cycle and Active Transcription Mediate Replication Restart after Fork Stalling at Co-transcriptional R-Loops.
Chappidi N; Nascakova Z; Boleslavska B; Zellweger R; Isik E; Andrs M; Menon S; Dobrovolna J; Balbo Pogliano C; Matos J; Porro A; Lopes M; Janscak P
Mol Cell; 2020 Feb; 77(3):528-541.e8. PubMed ID: 31759821
[TBL] [Abstract][Full Text] [Related]
6. Rad54 protein promotes branch migration of Holliday junctions.
Bugreev DV; Mazina OM; Mazin AV
Nature; 2006 Aug; 442(7102):590-3. PubMed ID: 16862129
[TBL] [Abstract][Full Text] [Related]
7. SUMO modification regulates BLM and RAD51 interaction at damaged replication forks.
Ouyang KJ; Woo LL; Zhu J; Huo D; Matunis MJ; Ellis NA
PLoS Biol; 2009 Dec; 7(12):e1000252. PubMed ID: 19956565
[TBL] [Abstract][Full Text] [Related]
8. Nek1 Regulates Rad54 to Orchestrate Homologous Recombination and Replication Fork Stability.
Spies J; Waizenegger A; Barton O; Sürder M; Wright WD; Heyer WD; Löbrich M
Mol Cell; 2016 Jun; 62(6):903-917. PubMed ID: 27264870
[TBL] [Abstract][Full Text] [Related]
9. Polarity and bypass of DNA heterology during branch migration of Holliday junctions by human RAD54, BLM, and RECQ1 proteins.
Mazina OM; Rossi MJ; Deakyne JS; Huang F; Mazin AV
J Biol Chem; 2012 Apr; 287(15):11820-32. PubMed ID: 22356911
[TBL] [Abstract][Full Text] [Related]
10. Regression of replication forks stalled by leading-strand template damage: II. Regression by RecA is inhibited by SSB.
Gupta S; Yeeles JT; Marians KJ
J Biol Chem; 2014 Oct; 289(41):28388-98. PubMed ID: 25138217
[TBL] [Abstract][Full Text] [Related]
11. The MMS22L-TONSL heterodimer directly promotes RAD51-dependent recombination upon replication stress.
Piwko W; Mlejnkova LJ; Mutreja K; Ranjha L; Stafa D; Smirnov A; Brodersen MM; Zellweger R; Sturzenegger A; Janscak P; Lopes M; Peter M; Cejka P
EMBO J; 2016 Dec; 35(23):2584-2601. PubMed ID: 27797818
[TBL] [Abstract][Full Text] [Related]
12. From strand exchange to branch migration; bypassing of non-homologous sequences by human Rad51 and Rad54.
Urena DE; Zhang Z; Tsai YC; Wang YZ; Chen J
J Mol Biol; 2011 Jan; 405(1):77-91. PubMed ID: 21056573
[TBL] [Abstract][Full Text] [Related]
13. Werner syndrome protein participates in a complex with RAD51, RAD54, RAD54B and ATR in response to ICL-induced replication arrest.
Otterlei M; Bruheim P; Ahn B; Bussen W; Karmakar P; Baynton K; Bohr VA
J Cell Sci; 2006 Dec; 119(Pt 24):5137-46. PubMed ID: 17118963
[TBL] [Abstract][Full Text] [Related]
14. RAD54 N-terminal domain is a DNA sensor that couples ATP hydrolysis with branch migration of Holliday junctions.
Goyal N; Rossi MJ; Mazina OM; Chi Y; Moritz RL; Clurman BE; Mazin AV
Nat Commun; 2018 Jan; 9(1):34. PubMed ID: 29295984
[TBL] [Abstract][Full Text] [Related]
15. DNA2 drives processing and restart of reversed replication forks in human cells.
Thangavel S; Berti M; Levikova M; Pinto C; Gomathinayagam S; Vujanovic M; Zellweger R; Moore H; Lee EH; Hendrickson EA; Cejka P; Stewart S; Lopes M; Vindigni A
J Cell Biol; 2015 Mar; 208(5):545-62. PubMed ID: 25733713
[TBL] [Abstract][Full Text] [Related]
16. RADX prevents genome instability by confining replication fork reversal to stalled forks.
Krishnamoorthy A; Jackson J; Mohamed T; Adolph M; Vindigni A; Cortez D
Mol Cell; 2021 Jul; 81(14):3007-3017.e5. PubMed ID: 34107305
[TBL] [Abstract][Full Text] [Related]
17. Hydroxyurea-stalled replication forks become progressively inactivated and require two different RAD51-mediated pathways for restart and repair.
Petermann E; Orta ML; Issaeva N; Schultz N; Helleday T
Mol Cell; 2010 Feb; 37(4):492-502. PubMed ID: 20188668
[TBL] [Abstract][Full Text] [Related]
18. A fork in the road: Where homologous recombination and stalled replication fork protection part ways.
Tye S; Ronson GE; Morris JR
Semin Cell Dev Biol; 2021 May; 113():14-26. PubMed ID: 32653304
[TBL] [Abstract][Full Text] [Related]
19. RADX Modulates RAD51 Activity to Control Replication Fork Protection.
Bhat KP; Krishnamoorthy A; Dungrawala H; Garcin EB; Modesti M; Cortez D
Cell Rep; 2018 Jul; 24(3):538-545. PubMed ID: 30021152
[TBL] [Abstract][Full Text] [Related]
20. The Bloom's syndrome helicase can promote the regression of a model replication fork.
Ralf C; Hickson ID; Wu L
J Biol Chem; 2006 Aug; 281(32):22839-46. PubMed ID: 16766518
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]