BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

368 related articles for article (PubMed ID: 21248121)

  • 1. Axonal degeneration is mediated by the mitochondrial permeability transition pore.
    Barrientos SA; Martinez NW; Yoo S; Jara JS; Zamorano S; Hetz C; Twiss JL; Alvarez J; Court FA
    J Neurosci; 2011 Jan; 31(3):966-78. PubMed ID: 21248121
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Cyclophilin D deficiency rescues axonal mitochondrial transport in Alzheimer's neurons.
    Guo L; Du H; Yan S; Wu X; McKhann GM; Chen JX; Yan SS
    PLoS One; 2013; 8(1):e54914. PubMed ID: 23382999
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Axonal Degeneration Is Mediated by Necroptosis Activation.
    Arrázola MS; Saquel C; Catalán RJ; Barrientos SA; Hernandez DE; Martínez NW; Catenaccio A; Court FA
    J Neurosci; 2019 May; 39(20):3832-3844. PubMed ID: 30850513
    [TBL] [Abstract][Full Text] [Related]  

  • 4. The P66Shc/mitochondrial permeability transition pore pathway determines neurodegeneration.
    Savino C; Pelicci P; Giorgio M
    Oxid Med Cell Longev; 2013; 2013():719407. PubMed ID: 23766859
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Diapause formation and downregulation of insulin-like signaling via DAF-16/FOXO delays axonal degeneration and neuronal loss.
    Calixto A; Jara JS; Court FA
    PLoS Genet; 2012; 8(12):e1003141. PubMed ID: 23300463
    [TBL] [Abstract][Full Text] [Related]  

  • 6. MEC17-induced α-tubulin acetylation restores mitochondrial transport function and alleviates axonal injury after intracerebral hemorrhage in mice.
    Yang Y; Chen X; Feng Z; Cai X; Zhu X; Cao M; Yang L; Chen Y; Wang Y; Feng H
    J Neurochem; 2022 Jan; 160(1):51-63. PubMed ID: 34407220
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Mitochondrial permeability transition pore is a potential drug target for neurodegeneration.
    Rao VK; Carlson EA; Yan SS
    Biochim Biophys Acta; 2014 Aug; 1842(8):1267-72. PubMed ID: 24055979
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Chronic ethanol consumption enhances sensitivity to Ca(2+)-mediated opening of the mitochondrial permeability transition pore and increases cyclophilin D in liver.
    King AL; Swain TM; Dickinson DA; Lesort MJ; Bailey SM
    Am J Physiol Gastrointest Liver Physiol; 2010 Oct; 299(4):G954-66. PubMed ID: 20651005
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Selective Inhibition of the Mitochondrial Permeability Transition Pore Protects against Neurodegeneration in Experimental Multiple Sclerosis.
    Warne J; Pryce G; Hill JM; Shi X; Lennerås F; Puentes F; Kip M; Hilditch L; Walker P; Simone MI; Chan AW; Towers GJ; Coker AR; Duchen MR; Szabadkai G; Baker D; Selwood DL
    J Biol Chem; 2016 Feb; 291(9):4356-73. PubMed ID: 26679998
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Calcium release from intra-axonal endoplasmic reticulum leads to axon degeneration through mitochondrial dysfunction.
    Villegas R; Martinez NW; Lillo J; Pihan P; Hernandez D; Twiss JL; Court FA
    J Neurosci; 2014 May; 34(21):7179-89. PubMed ID: 24849352
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Cysteine 203 of cyclophilin D is critical for cyclophilin D activation of the mitochondrial permeability transition pore.
    Nguyen TT; Stevens MV; Kohr M; Steenbergen C; Sack MN; Murphy E
    J Biol Chem; 2011 Nov; 286(46):40184-92. PubMed ID: 21930693
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Not all mitochondrial carrier proteins support permeability transition pore formation: no involvement of uncoupling protein 1.
    Crichton PG; Parker N; Vidal-Puig AJ; Brand MD
    Biosci Rep; 2009 Dec; 30(3):187-92. PubMed ID: 19622065
    [TBL] [Abstract][Full Text] [Related]  

  • 13. The mitochondrial permeability transition pore regulates nitric oxide-mediated apoptosis of neurons induced by target deprivation.
    Martin LJ; Adams NA; Pan Y; Price A; Wong M
    J Neurosci; 2011 Jan; 31(1):359-70. PubMed ID: 21209222
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Mitochondrial permeability transition pore in Alzheimer's disease: cyclophilin D and amyloid beta.
    Du H; Yan SS
    Biochim Biophys Acta; 2010 Jan; 1802(1):198-204. PubMed ID: 19616093
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Mitochondria as a central sensor for axonal degenerative stimuli.
    Court FA; Coleman MP
    Trends Neurosci; 2012 Jun; 35(6):364-72. PubMed ID: 22578891
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Traumatic brain injury-induced axonal phenotypes react differently to treatment.
    Hånell A; Greer JE; McGinn MJ; Povlishock JT
    Acta Neuropathol; 2015 Feb; 129(2):317-32. PubMed ID: 25528329
    [TBL] [Abstract][Full Text] [Related]  

  • 17. An approach to experimental synaptic pathology using green fluorescent protein-transgenic mice and gene knockout mice to show mitochondrial permeability transition pore-driven excitotoxicity in interneurons and motoneurons.
    Martin LJ
    Toxicol Pathol; 2011 Jan; 39(1):220-33. PubMed ID: 21378209
    [TBL] [Abstract][Full Text] [Related]  

  • 18. HAX-1 regulates cyclophilin-D levels and mitochondria permeability transition pore in the heart.
    Lam CK; Zhao W; Liu GS; Cai WF; Gardner G; Adly G; Kranias EG
    Proc Natl Acad Sci U S A; 2015 Nov; 112(47):E6466-75. PubMed ID: 26553996
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Axonal degeneration induced by glutamate excitotoxicity is mediated by necroptosis.
    Hernández DE; Salvadores NA; Moya-Alvarado G; Catalán RJ; Bronfman FC; Court FA
    J Cell Sci; 2018 Nov; 131(22):. PubMed ID: 30333135
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Mitochondrial permeability transition pore component cyclophilin D distinguishes nigrostriatal dopaminergic death paradigms in the MPTP mouse model of Parkinson's disease.
    Thomas B; Banerjee R; Starkova NN; Zhang SF; Calingasan NY; Yang L; Wille E; Lorenzo BJ; Ho DJ; Beal MF; Starkov A
    Antioxid Redox Signal; 2012 May; 16(9):855-68. PubMed ID: 21529244
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 19.