455 related articles for article (PubMed ID: 21248468)
1. Senescence-associated heterochromatin foci are dispensable for cellular senescence, occur in a cell type- and insult-dependent manner and follow expression of p16(ink4a).
Kosar M; Bartkova J; Hubackova S; Hodny Z; Lukas J; Bartek J
Cell Cycle; 2011 Feb; 10(3):457-68. PubMed ID: 21248468
[TBL] [Abstract][Full Text] [Related]
2. BRG1 is required for formation of senescence-associated heterochromatin foci induced by oncogenic RAS or BRCA1 loss.
Tu Z; Zhuang X; Yao YG; Zhang R
Mol Cell Biol; 2013 May; 33(9):1819-29. PubMed ID: 23438604
[TBL] [Abstract][Full Text] [Related]
3. SAHF, to senesce or not to senesce?
Kocylowski MK; Halazonetis TD
Cell Cycle; 2011 Mar; 10(5):738-9. PubMed ID: 21311234
[No Abstract] [Full Text] [Related]
4. Interplay between oncogene-induced DNA damage response and heterochromatin in senescence and cancer.
Di Micco R; Sulli G; Dobreva M; Liontos M; Botrugno OA; Gargiulo G; dal Zuffo R; Matti V; d'Ario G; Montani E; Mercurio C; Hahn WC; Gorgoulis V; Minucci S; d'Adda di Fagagna F
Nat Cell Biol; 2011 Mar; 13(3):292-302. PubMed ID: 21336312
[TBL] [Abstract][Full Text] [Related]
5. MYC and RAS are unable to cooperate in overcoming cellular senescence and apoptosis in normal human fibroblasts.
Zhang F; Zakaria SM; Högqvist Tabor V; Singh M; Tronnersjö S; Goodwin J; Selivanova G; Bartek J; Castell A; Larsson LG
Cell Cycle; 2018; 17(24):2697-2715. PubMed ID: 30526305
[TBL] [Abstract][Full Text] [Related]
6. Mitogen-activated protein kinase p38 and retinoblastoma protein signalling is required for DNA damage-mediated formation of senescence-associated heterochromatic foci in tumour cells.
Zhang Y; Gao Y; Zhao L; Han L; Lu Y; Hou P; Shi X; Liu X; Tian B; Wang X; Huang B; Lu J
FEBS J; 2013 Sep; 280(18):4625-39. PubMed ID: 23859194
[TBL] [Abstract][Full Text] [Related]
7. Tumor suppressor and aging biomarker p16(INK4a) induces cellular senescence without the associated inflammatory secretory phenotype.
Coppé JP; Rodier F; Patil CK; Freund A; Desprez PY; Campisi J
J Biol Chem; 2011 Oct; 286(42):36396-403. PubMed ID: 21880712
[TBL] [Abstract][Full Text] [Related]
8. Human keratinocytes that express hTERT and also bypass a p16(INK4a)-enforced mechanism that limits life span become immortal yet retain normal growth and differentiation characteristics.
Dickson MA; Hahn WC; Ino Y; Ronfard V; Wu JY; Weinberg RA; Louis DN; Li FP; Rheinwald JG
Mol Cell Biol; 2000 Feb; 20(4):1436-47. PubMed ID: 10648628
[TBL] [Abstract][Full Text] [Related]
9. E2FBP1 antagonizes the p16(INK4A)-Rb tumor suppressor machinery for growth suppression and cellular senescence by regulating promyelocytic leukemia protein stability.
Fukuyo Y; Takahashi A; Hara E; Horikoshi N; Pandita TK; Nakajima T
Int J Oral Sci; 2011 Oct; 3(4):200-8. PubMed ID: 22010578
[TBL] [Abstract][Full Text] [Related]
10. The relative contributions of the p53 and pRb pathways in oncogene-induced melanocyte senescence.
Haferkamp S; Tran SL; Becker TM; Scurr LL; Kefford RF; Rizos H
Aging (Albany NY); 2009 May; 1(6):542-56. PubMed ID: 20157537
[TBL] [Abstract][Full Text] [Related]
11. Senescence delay and repression of p16INK4a by Lsh via recruitment of histone deacetylases in human diploid fibroblasts.
Zhou R; Han L; Li G; Tong T
Nucleic Acids Res; 2009 Aug; 37(15):5183-96. PubMed ID: 19561196
[TBL] [Abstract][Full Text] [Related]
12. Depletion of ERK2 but not ERK1 abrogates oncogenic Ras-induced senescence.
Shin J; Yang J; Lee JC; Baek KH
Cell Signal; 2013 Dec; 25(12):2540-7. PubMed ID: 23993963
[TBL] [Abstract][Full Text] [Related]
13. Bacterial intoxication evokes cellular senescence with persistent DNA damage and cytokine signalling.
Blazkova H; Krejcikova K; Moudry P; Frisan T; Hodny Z; Bartek J
J Cell Mol Med; 2010 Jan; 14(1-2):357-67. PubMed ID: 19650831
[TBL] [Abstract][Full Text] [Related]
14. CREG1 enhances p16(INK4a) -induced cellular senescence.
Moolmuang B; Tainsky MA
Cell Cycle; 2011 Feb; 10(3):518-30. PubMed ID: 21263217
[TBL] [Abstract][Full Text] [Related]
15. Direct evidence from siRNA-directed "knock down" that p16(INK4a) is required for human fibroblast senescence and for limiting ras-induced epithelial cell proliferation.
Bond J; Jones C; Haughton M; DeMicco C; Kipling D; Wynford-Thomas D
Exp Cell Res; 2004 Jan; 292(1):151-6. PubMed ID: 14720514
[TBL] [Abstract][Full Text] [Related]
16. p16(INK4a) suppression by glucose restriction contributes to human cellular lifespan extension through SIRT1-mediated epigenetic and genetic mechanisms.
Li Y; Tollefsbol TO
PLoS One; 2011 Feb; 6(2):e17421. PubMed ID: 21390332
[TBL] [Abstract][Full Text] [Related]
17. Senescence of human fibroblasts induced by oncogenic Raf.
Zhu J; Woods D; McMahon M; Bishop JM
Genes Dev; 1998 Oct; 12(19):2997-3007. PubMed ID: 9765202
[TBL] [Abstract][Full Text] [Related]
18. Molecular dissection of formation of senescence-associated heterochromatin foci.
Zhang R; Chen W; Adams PD
Mol Cell Biol; 2007 Mar; 27(6):2343-58. PubMed ID: 17242207
[TBL] [Abstract][Full Text] [Related]
19. Definition of pRB- and p53-dependent and -independent steps in HIRA/ASF1a-mediated formation of senescence-associated heterochromatin foci.
Ye X; Zerlanko B; Zhang R; Somaiah N; Lipinski M; Salomoni P; Adams PD
Mol Cell Biol; 2007 Apr; 27(7):2452-65. PubMed ID: 17242198
[TBL] [Abstract][Full Text] [Related]
20. Senescence delay of human diploid fibroblast induced by anti-sense p16INK4a expression.
Duan J; Zhang Z; Tong T
J Biol Chem; 2001 Dec; 276(51):48325-31. PubMed ID: 11606567
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
