These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
106 related articles for article (PubMed ID: 2140387)
1. Requirement of the thymus for the recovery of anti-alloantigen helper T cells from tolerance induced by intravenous presensitization with allogeneic cells. Kitagawa S; Sato S; Azuma T; Hori S; Hamaoka T; Fujiwara H J Immunol; 1990 Jun; 144(11):4139-46. PubMed ID: 2140387 [TBL] [Abstract][Full Text] [Related]
2. Tolerance induction of allo-class II H-2 antigen-reactive L3T4+ helper T cells and prolonged survival of the corresponding class II H-2-disparate skin graft. Hori S; Sato S; Kitagawa S; Azuma T; Kokudo S; Hamaoka T; Fujiwara H J Immunol; 1989 Sep; 143(5):1447-52. PubMed ID: 2527264 [TBL] [Abstract][Full Text] [Related]
3. Property of class I H-2 alloantigen-reactive Lyt-2+ helper T cell subset. Abrogation of its proliferative and IL-2-producing capacities by intravenous injection of class I H-2-disparate allogeneic cells. Sato S; Azuma T; Shimizu J; Shima J; Kitagawa S; Hamaoka T; Fujiwara H J Immunol; 1988 Aug; 141(3):721-7. PubMed ID: 2456330 [TBL] [Abstract][Full Text] [Related]
4. Tolerance induction of allo-class I H-2 antigen-reactive Lyt-2+ helper T cells and prolonged survival of the corresponding class I H-2-disparate skin graft. Azuma T; Sato S; Kitagawa S; Hori S; Kokudo S; Hamaoka T; Fujiwara H J Immunol; 1989 Jul; 143(1):1-8. PubMed ID: 2525143 [TBL] [Abstract][Full Text] [Related]
5. Cell-cell interaction in graft rejection responses: induction of anti-allo-class I H-2 tolerance is prevented by immune responses against allo-class II H-2 antigens coexpressed on tolerogen. Hori S; Kitagawa S; Iwata H; Ochiai T; Isono K; Hamaoka T; Fujiwara H J Exp Med; 1992 Jan; 175(1):99-109. PubMed ID: 1730930 [TBL] [Abstract][Full Text] [Related]
6. Induction of anti-allo-class I H-2 tolerance by inactivation of CD8+ helper T cells, and reversal of tolerance through introduction of third-party helper T cells. Kitagawa S; Sato S; Hori S; Hamaoka T; Fujiwara H J Exp Med; 1990 Jul; 172(1):105-13. PubMed ID: 2141624 [TBL] [Abstract][Full Text] [Related]
7. Heterogeneity of CD4+ T cells involved in anti-allo-class I H-2 immune responses. Functional discrimination between the major proliferating cells and helper cells assisting cytotoxic T cell responses. Kitagawa S; Sato S; Azuma T; Shimizu J; Hamaoka T; Fujiwara H J Immunol; 1991 Apr; 146(8):2513-21. PubMed ID: 1673140 [TBL] [Abstract][Full Text] [Related]
8. Tumor allograft rejection is mainly mediated by CD8+ cytotoxic T lymphocytes stimulated with class I alloantigens in cooperation with CD4+ helper T cells recognizing class II alloantigens. Tomita Y; Mayumi H; Eto M; Nomoto K J Immunol; 1990 Mar; 144(6):2425-35. PubMed ID: 1968930 [TBL] [Abstract][Full Text] [Related]
9. Reactivity of hybridomas derived from T cells activated in vivo during graft-versus-host disease. Leibnitz RR; Lipsky PE; Thiele DL J Immunol; 1994 Dec; 153(11):4959-68. PubMed ID: 7963559 [TBL] [Abstract][Full Text] [Related]
10. Protection from T helper cell-mediated graft-versus-host disease by the presence of an MHC class I alloantigen is associated with perturbation of MHC class II-restricted responses by class I-derived peptides. Leibnitz RR; Lipsky PE; Thiele DL J Immunol; 1995 Aug; 155(4):1784-95. PubMed ID: 7636234 [TBL] [Abstract][Full Text] [Related]
11. Role of the thymus in repopulating alloantigen-reactive T cells after its functional elimination by intravenous presensitization with allogeneic cells. Sato S; Kitagawa S; Hori S; Azuma T; Hamaoka T; Fujiwara H Transplant Proc; 1991 Feb; 23(1 Pt 1):152-4. PubMed ID: 1824970 [No Abstract] [Full Text] [Related]
12. Assessment of alloreactive T cell subpopulations of aged mice in vivo. CD4+ but not CD8+ T cell-mediated rejection response declines with advanced age. Rosenberg AS; Sechler JM; Horvath JA; Maniero TG; Bloom ET Eur J Immunol; 1994 Jun; 24(6):1312-6. PubMed ID: 7911422 [TBL] [Abstract][Full Text] [Related]
13. The lymphoid cell populations required for induction of tolerance of different subsets of alloantigen-reactive T cells. Sato S; Iwata H; Kitagawa S; Kokudo S; Okuno K; Hamaoka T; Fujiwara H Transplantation; 1991 Nov; 52(5):862-7. PubMed ID: 1835197 [TBL] [Abstract][Full Text] [Related]
14. Nonresponsiveness to the male antigen H-Y in H-2 I-A-mutant B6.C-H-2bm12 is not caused by defective antigen presentation. de Waal LP; de Hoop J; Stukart MJ; Gleichmann H; Melvold RW; Melief CJ J Immunol; 1983 Feb; 130(2):655-60. PubMed ID: 6401303 [TBL] [Abstract][Full Text] [Related]
15. Allosuppressor and allohelper T cells in acute and chronic graft-vs.-host disease. II. F1 recipients carrying mutations at H-2K and/or I-A. Rolink AG; Pals ST; Gleichmann E J Exp Med; 1983 Feb; 157(2):755-71. PubMed ID: 6218218 [TBL] [Abstract][Full Text] [Related]
16. Graft-vs-host reaction limited to a class II MHC difference results in a selective deficiency in L3T4+ but not in Lyt-2+ T helper cell function. Moser M; Mizuochi T; Sharrow SO; Singer A; Shearer GM J Immunol; 1987 Mar; 138(5):1355-62. PubMed ID: 2949013 [TBL] [Abstract][Full Text] [Related]
17. Role of L3T4+ and Lyt-2+ donor cells in graft-versus-host immune deficiency induced across a class I, class II, or whole H-2 difference. Moser M; Sharrow SO; Shearer GM J Immunol; 1988 Apr; 140(8):2600-8. PubMed ID: 3258615 [TBL] [Abstract][Full Text] [Related]
18. Immunogenicity of the mutated H-2Kbm1 antigen(s). Test of thyroid graft rejection between B6.C-H-2bm1 and C57BL/6 mice following reciprocal immunization with normal versus malignant cells. Isakov N; Segal S Immunobiology; 1983 Dec; 165(5):485-99. PubMed ID: 6363278 [TBL] [Abstract][Full Text] [Related]
19. Antigen-presenting T cells. I. Class I alloantigen (bm1)-bearing T lymphoblasts efficiently stimulate a primary clonal response of Lyt-2+ cytotoxic lymphocyte precursors of B6 origin. Reimann J; Bellan A; Kabelitz D J Immunol; 1987 Feb; 138(4):1042-50. PubMed ID: 3492539 [TBL] [Abstract][Full Text] [Related]
20. The role of major histocompatibility complex and non-major histocompatibility complex encoded antigens in generation of bile duct lesions during hepatic graft-vs.-host responses mediated by helper or cytotoxic T cells. Williams FH; Thiele DL Hepatology; 1994 Apr; 19(4):980-8. PubMed ID: 8138273 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]