230 related articles for article (PubMed ID: 21420802)
1. Recent progress in understanding the pathogenesis of Clostridium perfringens type C infections.
Uzal FA; McClane BA
Vet Microbiol; 2011 Nov; 153(1-2):37-43. PubMed ID: 21420802
[TBL] [Abstract][Full Text] [Related]
2. Contact with enterocyte-like Caco-2 cells induces rapid upregulation of toxin production by Clostridium perfringens type C isolates.
Vidal JE; Ohtani K; Shimizu T; McClane BA
Cell Microbiol; 2009 Sep; 11(9):1306-28. PubMed ID: 19438515
[TBL] [Abstract][Full Text] [Related]
3. Beta toxin is essential for the intestinal virulence of Clostridium perfringens type C disease isolate CN3685 in a rabbit ileal loop model.
Sayeed S; Uzal FA; Fisher DJ; Saputo J; Vidal JE; Chen Y; Gupta P; Rood JI; McClane BA
Mol Microbiol; 2008 Jan; 67(1):15-30. PubMed ID: 18078439
[TBL] [Abstract][Full Text] [Related]
4. Epsilon-toxin production by Clostridium perfringens type D strain CN3718 is dependent upon the agr operon but not the VirS/VirR two-component regulatory system.
Chen J; Rood JI; McClane BA
mBio; 2011; 2(6):. PubMed ID: 22167225
[TBL] [Abstract][Full Text] [Related]
5. The VirS/VirR two-component system regulates the anaerobic cytotoxicity, intestinal pathogenicity, and enterotoxemic lethality of Clostridium perfringens type C isolate CN3685.
Ma M; Vidal J; Saputo J; McClane BA; Uzal F
mBio; 2011 Jan; 2(1):e00338-10. PubMed ID: 21264065
[TBL] [Abstract][Full Text] [Related]
6. Identifying the Basis for VirS/VirR Two-Component Regulatory System Control of Clostridium perfringens Beta-Toxin Production.
Mehdizadeh Gohari I; Li J; McClane BA
J Bacteriol; 2021 Aug; 203(18):e0027921. PubMed ID: 34228498
[TBL] [Abstract][Full Text] [Related]
7. Host cell-induced signaling causes Clostridium perfringens to upregulate production of toxins important for intestinal infections.
Chen J; Ma M; Uzal FA; McClane BA
Gut Microbes; 2014; 5(1):96-107. PubMed ID: 24061146
[TBL] [Abstract][Full Text] [Related]
8. Endothelial binding of beta toxin to small intestinal mucosal endothelial cells in early stages of experimentally induced Clostridium perfringens type C enteritis in pigs.
Schumacher VL; Martel A; Pasmans F; Van Immerseel F; Posthaus H
Vet Pathol; 2013 Jul; 50(4):626-9. PubMed ID: 23012387
[TBL] [Abstract][Full Text] [Related]
9. Evidence That VirS Is a Receptor for the Signaling Peptide of the Clostridium perfringens Agr-like Quorum Sensing System.
Li J; McClane BA
mBio; 2020 Sep; 11(5):. PubMed ID: 32934089
[TBL] [Abstract][Full Text] [Related]
10. The Agr-Like Quorum Sensing System Is Required for Pathogenesis of Necrotic Enteritis Caused by Clostridium perfringens in Poultry.
Yu Q; Lepp D; Mehdizadeh Gohari I; Wu T; Zhou H; Yin X; Yu H; Prescott JF; Nie SP; Xie MY; Gong J
Infect Immun; 2017 Jun; 85(6):. PubMed ID: 28373356
[No Abstract] [Full Text] [Related]
11. Characterization of toxin plasmids in Clostridium perfringens type C isolates.
Gurjar A; Li J; McClane BA
Infect Immun; 2010 Nov; 78(11):4860-9. PubMed ID: 20823204
[TBL] [Abstract][Full Text] [Related]
12. Baicalin-aluminum alleviates necrotic enteritis in broiler chickens by inhibiting virulence factors expression of
Liu J; Wu S; Zhao H; Ye C; Fu S; Liu Y; Liu T; Qiu Y
Front Cell Infect Microbiol; 2023; 13():1243819. PubMed ID: 37818042
[No Abstract] [Full Text] [Related]
13. Role of the Agr-like quorum-sensing system in regulating toxin production by Clostridium perfringens type B strains CN1793 and CN1795.
Chen J; McClane BA
Infect Immun; 2012 Sep; 80(9):3008-17. PubMed ID: 22689820
[TBL] [Abstract][Full Text] [Related]
14. CodY is a global regulator of virulence-associated properties for Clostridium perfringens type D strain CN3718.
Li J; Ma M; Sarker MR; McClane BA
mBio; 2013 Oct; 4(5):e00770-13. PubMed ID: 24105766
[TBL] [Abstract][Full Text] [Related]
15. TpeL-producing strains of Clostridium perfringens type A are highly virulent for broiler chicks.
Coursodon CF; Glock RD; Moore KL; Cooper KK; Songer JG
Anaerobe; 2012 Feb; 18(1):117-21. PubMed ID: 22019986
[TBL] [Abstract][Full Text] [Related]
16. Dissecting the contributions of Clostridium perfringens type C toxins to lethality in the mouse intravenous injection model.
Fisher DJ; Fernandez-Miyakawa ME; Sayeed S; Poon R; Adams V; Rood JI; Uzal FA; McClane BA
Infect Immun; 2006 Sep; 74(9):5200-10. PubMed ID: 16926413
[TBL] [Abstract][Full Text] [Related]
17. Characterization of toxin genes and quantitative analysis of netB in necrotic enteritis (NE)-producing and non-NE-producing Clostridium perfringens isolated from chickens.
Yang WY; Chou CH; Wang C
Anaerobe; 2018 Dec; 54():115-120. PubMed ID: 30170048
[TBL] [Abstract][Full Text] [Related]
18. CD31 (PECAM-1) Serves as the Endothelial Cell-Specific Receptor of Clostridium perfringens β-Toxin.
Bruggisser J; Tarek B; Wyder M; Müller P; von Ballmoos C; Witz G; Enzmann G; Deutsch U; Engelhardt B; Posthaus H
Cell Host Microbe; 2020 Jul; 28(1):69-78.e6. PubMed ID: 32497498
[TBL] [Abstract][Full Text] [Related]
19. The VirSR two-component signal transduction system regulates NetB toxin production in Clostridium perfringens.
Cheung JK; Keyburn AL; Carter GP; Lanckriet AL; Van Immerseel F; Moore RJ; Rood JI
Infect Immun; 2010 Jul; 78(7):3064-72. PubMed ID: 20457789
[TBL] [Abstract][Full Text] [Related]
20. Virulence for chickens of Clostridium perfringens isolated from poultry and other sources.
Cooper KK; Theoret JR; Stewart BA; Trinh HT; Glock RD; Songer JG
Anaerobe; 2010 Jun; 16(3):289-92. PubMed ID: 20193771
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
