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7. Clinical characteristics of recessive and dominant congenital hyperinsulinism due to mutation(s) in the ABCC8/KCNJ11 genes encoding the ATP-sensitive potasium channel in the pancreatic beta cell. Oçal G; Flanagan SE; Hacihamdioğlu B; Berberoğlu M; Siklar Z; Ellard S; Savas Erdeve S; Okulu E; Akin IM; Atasay B; Arsan S; Yağmurlu A J Pediatr Endocrinol Metab; 2011; 24(11-12):1019-23. PubMed ID: 22308858 [TBL] [Abstract][Full Text] [Related]
8. ABCC8 (SUR1) and KCNJ11 (KIR6.2) mutations in persistent hyperinsulinemic hypoglycemia of infancy and evaluation of different therapeutic measures. Darendeliler F; Fournet JC; Baş F; Junien C; Gross MS; Bundak R; Saka N; Günöz H J Pediatr Endocrinol Metab; 2002; 15(7):993-1000. PubMed ID: 12199344 [TBL] [Abstract][Full Text] [Related]
9. Monoallelic ABCC8 mutations are a common cause of diazoxide-unresponsive diffuse form of congenital hyperinsulinism. Saint-Martin C; Zhou Q; Martin GM; Vaury C; Leroy G; Arnoux JB; de Lonlay P; Shyng SL; Bellanné-Chantelot C Clin Genet; 2015 May; 87(5):448-54. PubMed ID: 24814349 [TBL] [Abstract][Full Text] [Related]
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11. Characterization of ABCC8 and KCNJ11 gene mutations and phenotypes in Korean patients with congenital hyperinsulinism. Park SE; Flanagan SE; Hussain K; Ellard S; Shin CH; Yang SW Eur J Endocrinol; 2011 Jun; 164(6):919-26. PubMed ID: 21422196 [TBL] [Abstract][Full Text] [Related]
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13. Clinical and molecular characterisation of 300 patients with congenital hyperinsulinism. Kapoor RR; Flanagan SE; Arya VB; Shield JP; Ellard S; Hussain K Eur J Endocrinol; 2013 Apr; 168(4):557-64. PubMed ID: 23345197 [TBL] [Abstract][Full Text] [Related]
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17. Genotype-phenotype correlation in Taiwanese children with diazoxide-unresponsive congenital hyperinsulinism. Lee CT; Tsai WH; Chang CC; Chen PC; Fann CS; Chang HK; Liu SY; Wu MZ; Chiu PC; Hsu WM; Yang WS; Lai LP; Tsai WY; Yang SB; Chen PL Front Endocrinol (Lausanne); 2023; 14():1283907. PubMed ID: 38033998 [TBL] [Abstract][Full Text] [Related]
18. Genotype-phenotype correlations in children with congenital hyperinsulinism due to recessive mutations of the adenosine triphosphate-sensitive potassium channel genes. Henwood MJ; Kelly A; Macmullen C; Bhatia P; Ganguly A; Thornton PS; Stanley CA J Clin Endocrinol Metab; 2005 Feb; 90(2):789-94. PubMed ID: 15562009 [TBL] [Abstract][Full Text] [Related]
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