247 related articles for article (PubMed ID: 21558983)
1. Neutrophils, not monocyte/macrophages, are the major splenic source of postburn IL-10.
Noel G; Wang Q; Schwemberger S; Hanson C; Giacalone N; Haar L; Ogle CK
Shock; 2011 Aug; 36(2):149-55. PubMed ID: 21558983
[TBL] [Abstract][Full Text] [Related]
2. Postburn monocytes are the major producers of TNF-alpha in the heterogeneous splenic macrophage population.
Noel G; Guo X; Wang Q; Schwemberger S; Byrum D; Ogle C
Shock; 2007 Mar; 27(3):312-9. PubMed ID: 17304113
[TBL] [Abstract][Full Text] [Related]
3. Interaction between the innate and adaptive immune systems is required to survive sepsis and control inflammation after injury.
Shelley O; Murphy T; Paterson H; Mannick JA; Lederer JA
Shock; 2003 Aug; 20(2):123-9. PubMed ID: 12865655
[TBL] [Abstract][Full Text] [Related]
4. In vivo IL-18 supplementation ameliorates lethal acute lung injury in burn-primed endotoxemic mice: a novel anti-inflammatory role of IL-18.
Sekine K; Fujishima S; Sasaki J; Ishizaka A; Aiso S; Aikawa N
Shock; 2009 Nov; 32(5):554-62. PubMed ID: 19197224
[TBL] [Abstract][Full Text] [Related]
5. Molecular or pharmacologic inhibition of the CD14 signaling pathway protects against burn-related myocardial inflammation and dysfunction.
Barber RC; Maass DL; White DJ; Chang LY; Horton JW
Shock; 2008 Dec; 30(6):705-13. PubMed ID: 18461018
[TBL] [Abstract][Full Text] [Related]
6. A ribonucleotide reductase inhibitor reverses burn-induced inflammatory defects.
Noel G; Wang Q; Osterburg A; Schwemberger S; James L; Haar L; Giacalone N; Thomas I; Ogle C
Shock; 2010 Nov; 34(5):535-44. PubMed ID: 20386495
[TBL] [Abstract][Full Text] [Related]
7. The effector component of the cytotoxic T-lymphocyte response has a biphasic pattern after burn injury.
Hunt JP; Hunter CT; Brownstein MR; Giannopoulos A; Hultman CS; deSerres S; Bracey L; Frelinger J; Meyer AA
J Surg Res; 1998 Dec; 80(2):243-51. PubMed ID: 9878320
[TBL] [Abstract][Full Text] [Related]
8. Skewing the Th cell phenotype toward Th1 alters the maturation of tumor-infiltrating mononuclear phagocytes.
Nonaka K; Saio M; Suwa T; Frey AB; Umemura N; Imai H; Ouyang GF; Osada S; Balazs M; Adany R; Kawaguchi Y; Yoshida K; Takami T
J Leukoc Biol; 2008 Sep; 84(3):679-88. PubMed ID: 18566103
[TBL] [Abstract][Full Text] [Related]
9. Regulation of macrophage IL-10 production postinjury via beta2 integrin signaling and the P38 MAP kinase pathway.
Schwacha MG; Chaudry IH; Alexander M
Shock; 2003 Dec; 20(6):529-35. PubMed ID: 14625477
[TBL] [Abstract][Full Text] [Related]
10. Monocytes/macrophages and/or neutrophils are the target of IL-10 in the LPS endotoxemia model.
Pils MC; Pisano F; Fasnacht N; Heinrich JM; Groebe L; Schippers A; Rozell B; Jack RS; Müller W
Eur J Immunol; 2010 Feb; 40(2):443-8. PubMed ID: 19941312
[TBL] [Abstract][Full Text] [Related]
11. Increased IL-10 production during spontaneous apoptosis of monocytes.
Bzowska M; Guzik K; Barczyk K; Ernst M; Flad HD; Pryjma J
Eur J Immunol; 2002 Jul; 32(7):2011-20. PubMed ID: 12115622
[TBL] [Abstract][Full Text] [Related]
12. Pathogenic role of interleukin-6 in the development of sepsis. Part I: Study in a standardized contact burn murine model.
Pallua N; von Heimburg D
Crit Care Med; 2003 May; 31(5):1490-4. PubMed ID: 12771623
[TBL] [Abstract][Full Text] [Related]
13. Regulation of the postburn wound inflammatory response by gammadelta T-cells.
Daniel T; Thobe BM; Chaudry IH; Choudhry MA; Hubbard WJ; Schwacha MG
Shock; 2007 Sep; 28(3):278-83. PubMed ID: 17545947
[TBL] [Abstract][Full Text] [Related]
14. Modulation of TNFalpha, IL-10 and IL-12p40 levels by a ceramide-1-phosphate analog, PCERA-1, in vivo and ex vivo in primary macrophages.
Avni D; Goldsmith M; Ernst O; Mashiach R; Tuntland T; Meijler MM; Gray NS; Rosen H; Zor T
Immunol Lett; 2009 Mar; 123(1):1-8. PubMed ID: 19185589
[TBL] [Abstract][Full Text] [Related]
15. A central role for CD95 (Fas) in T-cell reactivity after injury.
Kell MR; Shelley O; Mannick JA; Guo Z; Lederer JA
Surgery; 2000 Aug; 128(2):159-64. PubMed ID: 10922986
[TBL] [Abstract][Full Text] [Related]
16. [Induction mechanism of shock: applying the etiology in judgment of the cause of death in forensic practice].
Tanaka N
Nihon Hoigaku Zasshi; 2004 Sep; 58(2):130-40. PubMed ID: 15526767
[TBL] [Abstract][Full Text] [Related]
17. Moesin: a potential LPS receptor on human monocytes.
Amar S; Oyaisu K; Li L; Van Dyke T
J Endotoxin Res; 2001; 7(4):281-6. PubMed ID: 11717582
[TBL] [Abstract][Full Text] [Related]
18. Effects of prolactin level on burn-induced aberrations in myelopoiesis.
Dugan AL; Schwemberger S; Babcock GF; Buckley D; Buckley AR; Ogle CK; Horseman ND
Shock; 2004 Feb; 21(2):151-9. PubMed ID: 14752289
[TBL] [Abstract][Full Text] [Related]
19. Pathogenic role of interleukin-6 in the development of sepsis. Part II: Significance of anti-interleukin-6 and anti-soluble interleukin-6 receptor-alpha antibodies in a standardized murine contact burn model.
Pallua N; Low JF; von Heimburg D
Crit Care Med; 2003 May; 31(5):1495-501. PubMed ID: 12771624
[TBL] [Abstract][Full Text] [Related]
20. Human polymorphonuclear leukocytes derived from chronically inflamed tissue express inflammatory cytokines in vivo.
Takeichi O; Saito I; Tsurumachi T; Saito T; Moro I
Cell Immunol; 1994 Jul; 156(2):296-309. PubMed ID: 8025949
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
