These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

160 related articles for article (PubMed ID: 21616139)

  • 1. P66Shc mediated ferritin degradation--a novel mechanism of ROS formation.
    Borkowska A; Sielicka-Dudzin A; Herman-Antosiewicz A; Halon M; Wozniak M; Antosiewicz J
    Free Radic Biol Med; 2011 Aug; 51(3):658-63. PubMed ID: 21616139
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Diallyl trisulfide-induced prostate cancer cell death is associated with Akt/PKB dephosphorylation mediated by P-p66shc.
    Borkowska A; Sielicka-Dudzin A; Herman-Antosiewicz A; Wozniak M; Fedeli D; Falcioni G; Antosiewicz J
    Eur J Nutr; 2012 Oct; 51(7):817-25. PubMed ID: 22020565
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Diallyl trisulfide is more cytotoxic to prostate cancer cells PC-3 than to noncancerous epithelial cell line PNT1A: a possible role of p66Shc signaling axis.
    Borkowska A; Knap N; Antosiewicz J
    Nutr Cancer; 2013; 65(5):711-7. PubMed ID: 23859039
    [TBL] [Abstract][Full Text] [Related]  

  • 4. c-Jun NH(2)-terminal kinase signaling axis regulates diallyl trisulfide-induced generation of reactive oxygen species and cell cycle arrest in human prostate cancer cells.
    Antosiewicz J; Herman-Antosiewicz A; Marynowski SW; Singh SV
    Cancer Res; 2006 May; 66(10):5379-86. PubMed ID: 16707465
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Impact of JNK1, JNK2, and ligase Itch on reactive oxygen species formation and survival of prostate cancer cells treated with diallyl trisulfide.
    Sielicka-Dudzin A; Borkowska A; Herman-Antosiewicz A; Wozniak M; Jozwik A; Fedeli D; Antosiewicz J
    Eur J Nutr; 2012 Aug; 51(5):573-81. PubMed ID: 21863240
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Diallyl trisulfide-induced G(2)-M phase cell cycle arrest in human prostate cancer cells is caused by reactive oxygen species-dependent destruction and hyperphosphorylation of Cdc 25 C.
    Xiao D; Herman-Antosiewicz A; Antosiewicz J; Xiao H; Brisson M; Lazo JS; Singh SV
    Oncogene; 2005 Sep; 24(41):6256-68. PubMed ID: 15940258
    [TBL] [Abstract][Full Text] [Related]  

  • 7. [P66shc action on resistance of colon carcinoma RKO cells to oxidative stress].
    Galimov ER; Sidorenko AS; Tereshkova AV; Pletiushkina OIu; Cherniak BV; Chumakov PM
    Mol Biol (Mosk); 2012; 46(1):139-46. PubMed ID: 22642111
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Reactive oxygen species induced by p66Shc longevity protein mediate nongenomic androgen action via tyrosine phosphorylation signaling to enhance tumorigenicity of prostate cancer cells.
    Veeramani S; Chou YW; Lin FC; Muniyan S; Lin FF; Kumar S; Xie Y; Lele SM; Tu Y; Lin MF
    Free Radic Biol Med; 2012 Jul; 53(1):95-108. PubMed ID: 22561705
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Diallyl trisulfide induces apoptosis in human breast cancer cells through ROS-mediated activation of JNK and AP-1.
    Na HK; Kim EH; Choi MA; Park JM; Kim DH; Surh YJ
    Biochem Pharmacol; 2012 Nov; 84(10):1241-50. PubMed ID: 22981381
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Diallyl trisulfide-induced apoptosis in human prostate cancer cells involves c-Jun N-terminal kinase and extracellular-signal regulated kinase-mediated phosphorylation of Bcl-2.
    Xiao D; Choi S; Johnson DE; Vogel VG; Johnson CS; Trump DL; Lee YJ; Singh SV
    Oncogene; 2004 Jul; 23(33):5594-606. PubMed ID: 15184882
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Allyl sulfides inhibit cell growth of skin cancer cells through induction of DNA damage mediated G2/M arrest and apoptosis.
    Wang HC; Yang JH; Hsieh SC; Sheen LY
    J Agric Food Chem; 2010 Jun; 58(11):7096-103. PubMed ID: 20459099
    [TBL] [Abstract][Full Text] [Related]  

  • 12. The mitochondrial reactive oxygen species regulator p66Shc controls PDGF-induced signaling and migration through protein tyrosine phosphatase oxidation.
    Frijhoff J; Dagnell M; Augsten M; Beltrami E; Giorgio M; Östman A
    Free Radic Biol Med; 2014 Mar; 68():268-77. PubMed ID: 24378437
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Diallyl trisufide (DATS) suppresses high glucose-induced cardiomyocyte apoptosis by inhibiting JNK/NFκB signaling via attenuating ROS generation.
    Kuo WW; Wang WJ; Tsai CY; Way CL; Hsu HH; Chen LM
    Int J Cardiol; 2013 Sep; 168(1):270-80. PubMed ID: 23158927
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Effect of diallyl trisulfide derivatives on the induction of apoptosis in human prostate cancer PC-3 cells.
    Chen M; Li B; Zhao X; Zuo H; He X; Li Z; Liu X; Chen L
    Mol Cell Biochem; 2012 Apr; 363(1-2):75-84. PubMed ID: 22143535
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Diallyl trisulfide suppresses growth of PC-3 human prostate cancer xenograft in vivo in association with Bax and Bak induction.
    Xiao D; Lew KL; Kim YA; Zeng Y; Hahm ER; Dhir R; Singh SV
    Clin Cancer Res; 2006 Nov; 12(22):6836-43. PubMed ID: 17121905
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Diallyl trisulfide selectively causes Bax- and Bak-mediated apoptosis in human lung cancer cells.
    Xiao D; Zeng Y; Hahm ER; Kim YA; Ramalingam S; Singh SV
    Environ Mol Mutagen; 2009 Apr; 50(3):201-12. PubMed ID: 18800351
    [TBL] [Abstract][Full Text] [Related]  

  • 17. JNK/p66Shc/ITCH Signaling Pathway Mediates Angiotensin II-induced Ferritin Degradation and Labile Iron Pool Increase.
    Borkowska A; Popowska U; Spodnik J; Herman-Antosiewicz A; Woźniak M; Antosiewicz J
    Nutrients; 2020 Feb; 12(3):. PubMed ID: 32121405
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Mitochondrial redox signaling by p66Shc is involved in regulating androgenic growth stimulation of human prostate cancer cells.
    Veeramani S; Yuan TC; Lin FF; Lin MF
    Oncogene; 2008 Aug; 27(37):5057-68. PubMed ID: 18504439
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Changes in skeletal muscle iron metabolism outpace amyotrophic lateral sclerosis onset in transgenic rats bearing the G93A hmSOD1 gene mutation.
    Halon M; Kaczor JJ; Ziolkowski W; Flis DJ; Borkowska A; Popowska U; Nyka W; Wozniak M; Antosiewicz J
    Free Radic Res; 2014 Nov; 48(11):1363-70. PubMed ID: 25175826
    [TBL] [Abstract][Full Text] [Related]  

  • 20. p53/p66Shc-mediated signaling contributes to the progression of non-alcoholic steatohepatitis in humans and mice.
    Tomita K; Teratani T; Suzuki T; Oshikawa T; Yokoyama H; Shimamura K; Nishiyama K; Mataki N; Irie R; Minamino T; Okada Y; Kurihara C; Ebinuma H; Saito H; Shimizu I; Yoshida Y; Hokari R; Sugiyama K; Hatsuse K; Yamamoto J; Kanai T; Miura S; Hibi T
    J Hepatol; 2012 Oct; 57(4):837-43. PubMed ID: 22641095
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 8.