176 related articles for article (PubMed ID: 2174110)
1. Hyperphosphorylation of the retinoblastoma gene product is determined by domains outside the simian virus 40 large-T-antigen-binding regions.
Hamel PA; Cohen BL; Sorce LM; Gallie BL; Phillips RA
Mol Cell Biol; 1990 Dec; 10(12):6586-95. PubMed ID: 2174110
[TBL] [Abstract][Full Text] [Related]
2. Functional implications of mutations within polyomavirus large T antigen Rb-binding domain: effects on pRb and p107 binding in vitro and immortalization activity in vivo.
Pilon AA; Desjardins P; Hassell JA; Mes-Masson AM
J Virol; 1996 Jul; 70(7):4457-65. PubMed ID: 8676470
[TBL] [Abstract][Full Text] [Related]
3. Simian virus 40 large T antigen alters the phosphorylation state of the RB-related proteins p130 and p107.
Stubdal H; Zalvide J; DeCaprio JA
J Virol; 1996 May; 70(5):2781-8. PubMed ID: 8627752
[TBL] [Abstract][Full Text] [Related]
4. Dissociation of retinoblastoma gene protein hyperphosphorylation and commitment to enter S phase.
Ogryzko VV; Hirai TH; Shih CE; Howard BH
J Virol; 1994 Jun; 68(6):3724-32. PubMed ID: 8189510
[TBL] [Abstract][Full Text] [Related]
5. Role of pRb-related proteins in simian virus 40 large-T-antigen-mediated transformation.
Zalvide J; DeCaprio JA
Mol Cell Biol; 1995 Oct; 15(10):5800-10. PubMed ID: 7565733
[TBL] [Abstract][Full Text] [Related]
6. Converting the JCV T antigen Rb binding domain to that of SV40 does not alter JCV's limited transforming activity but does eliminate viral viability.
Tavis JE; Trowbridge PW; Frisque RJ
Virology; 1994 Mar; 199(2):384-92. PubMed ID: 8122368
[TBL] [Abstract][Full Text] [Related]
7. Induction versus progression of brain tumor development: differential functions for the pRB- and p53-targeting domains of simian virus 40 T antigen.
Sáenz Robles MT; Symonds H; Chen J; Van Dyke T
Mol Cell Biol; 1994 Apr; 14(4):2686-98. PubMed ID: 8139568
[TBL] [Abstract][Full Text] [Related]
8. Nuclear binding of purified retinoblastoma gene product is determined by cell cycle-regulated phosphorylation.
Templeton DJ
Mol Cell Biol; 1992 Feb; 12(2):435-43. PubMed ID: 1310146
[TBL] [Abstract][Full Text] [Related]
9. Regions controlling hyperphosphorylation and conformation of the retinoblastoma gene product are independent of domains required for transcriptional repression.
Hamel PA; Gill RM; Phillips RA; Gallie BL
Oncogene; 1992 Apr; 7(4):693-701. PubMed ID: 1565466
[TBL] [Abstract][Full Text] [Related]
10. Adenovirus E1A, simian virus 40 tumor antigen, and human papillomavirus E7 protein share the capacity to disrupt the interaction between transcription factor E2F and the retinoblastoma gene product.
Chellappan S; Kraus VB; Kroger B; Munger K; Howley PM; Phelps WC; Nevins JR
Proc Natl Acad Sci U S A; 1992 May; 89(10):4549-53. PubMed ID: 1316611
[TBL] [Abstract][Full Text] [Related]
11. Definition of the minimal simian virus 40 large T antigen- and adenovirus E1A-binding domain in the retinoblastoma gene product.
Kaelin WG; Ewen ME; Livingston DM
Mol Cell Biol; 1990 Jul; 10(7):3761-9. PubMed ID: 2162480
[TBL] [Abstract][Full Text] [Related]
12. Mutations within the hamster polyomavirus large T antigen domain involved in pRb binding impair virus productive cycle and immortalization capacity.
Goutebroze L; de la Roche Saint Andre C; Scherneck S; Feunteun J
Oncogene; 1993 Mar; 8(3):685-93. PubMed ID: 8382359
[TBL] [Abstract][Full Text] [Related]
13. Host range and cell cycle activation properties of polyomavirus large T-antigen mutants defective in pRB binding.
Freund R; Bauer PH; Crissman HA; Bradbury EM; Benjamin TL
J Virol; 1994 Nov; 68(11):7227-34. PubMed ID: 7933105
[TBL] [Abstract][Full Text] [Related]
14. Polyomavirus large T mutants affected in retinoblastoma protein binding are defective in immortalization.
Larose A; Dyson N; Sullivan M; Harlow E; Bastin M
J Virol; 1991 May; 65(5):2308-13. PubMed ID: 2016761
[TBL] [Abstract][Full Text] [Related]
15. Overproduction of Rb protein after the G1/S boundary causes G2 arrest.
Karantza V; Maroo A; Fay D; Sedivy JM
Mol Cell Biol; 1993 Nov; 13(11):6640-52. PubMed ID: 8413260
[TBL] [Abstract][Full Text] [Related]
16. Loss of p19(ARF) eliminates the requirement for the pRB-binding motif in simian virus 40 large T antigen-mediated transformation.
Chao HH; Buchmann AM; DeCaprio JA
Mol Cell Biol; 2000 Oct; 20(20):7624-33. PubMed ID: 11003658
[TBL] [Abstract][Full Text] [Related]
17. DNA tumor virus oncoproteins and retinoblastoma gene mutations share the ability to relieve the cell's requirement for cyclin D1 function in G1.
Lukas J; Müller H; Bartkova J; Spitkovsky D; Kjerulff AA; Jansen-Dürr P; Strauss M; Bartek J
J Cell Biol; 1994 May; 125(3):625-38. PubMed ID: 8175885
[TBL] [Abstract][Full Text] [Related]
18. The DnaJ domain of polyomavirus large T antigen is required to regulate Rb family tumor suppressor function.
Sheng Q; Denis D; Ratnofsky M; Roberts TM; DeCaprio JA; Schaffhausen B
J Virol; 1997 Dec; 71(12):9410-6. PubMed ID: 9371601
[TBL] [Abstract][Full Text] [Related]
19. Inactivation of the retinoblastoma susceptibility protein is not sufficient for the transforming function of the conserved region 2-like domain of simian virus 40 large T antigen.
Christensen JB; Imperiale MJ
J Virol; 1995 Jun; 69(6):3945-8. PubMed ID: 7745751
[TBL] [Abstract][Full Text] [Related]
20. Biochemical characterization of phosphorylation site mutants of simian virus 40 large T antigen: evidence for interaction between amino- and carboxy-terminal domains.
Scheidtmann KH; Buck M; Schneider J; Kalderon D; Fanning E; Smith AE
J Virol; 1991 Mar; 65(3):1479-90. PubMed ID: 1847465
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]