657 related articles for article (PubMed ID: 21834058)
1. Stress signaling from the endoplasmic reticulum: A central player in the pathogenesis of amyotrophic lateral sclerosis.
Walker AK; Atkin JD
IUBMB Life; 2011 Sep; 63(9):754-63. PubMed ID: 21834058
[TBL] [Abstract][Full Text] [Related]
2. ER stress and unfolded protein response in amyotrophic lateral sclerosis.
Kanekura K; Suzuki H; Aiso S; Matsuoka M
Mol Neurobiol; 2009 Apr; 39(2):81-9. PubMed ID: 19184563
[TBL] [Abstract][Full Text] [Related]
3. Overexpression of human mutated G93A SOD1 changes dynamics of the ER mitochondria calcium cycle specifically in mouse embryonic motor neurons.
Lautenschläger J; Prell T; Ruhmer J; Weidemann L; Witte OW; Grosskreutz J
Exp Neurol; 2013 Sep; 247():91-100. PubMed ID: 23578819
[TBL] [Abstract][Full Text] [Related]
4. Rab1-dependent ER-Golgi transport dysfunction is a common pathogenic mechanism in SOD1, TDP-43 and FUS-associated ALS.
Soo KY; Halloran M; Sundaramoorthy V; Parakh S; Toth RP; Southam KA; McLean CA; Lock P; King A; Farg MA; Atkin JD
Acta Neuropathol; 2015 Nov; 130(5):679-97. PubMed ID: 26298469
[TBL] [Abstract][Full Text] [Related]
5. Superoxide dismutase 1 mutants related to amyotrophic lateral sclerosis induce endoplasmic stress in neuro2a cells.
Oh YK; Shin KS; Yuan J; Kang SJ
J Neurochem; 2008 Feb; 104(4):993-1005. PubMed ID: 18233996
[TBL] [Abstract][Full Text] [Related]
6. The unfolded protein response in models of human mutant G93A amyotrophic lateral sclerosis.
Prell T; Lautenschläger J; Witte OW; Carri MT; Grosskreutz J
Eur J Neurosci; 2012 Mar; 35(5):652-60. PubMed ID: 22390177
[TBL] [Abstract][Full Text] [Related]
7. Endoplasmic reticulum stress is accompanied by activation of NF-κB in amyotrophic lateral sclerosis.
Prell T; Lautenschläger J; Weidemann L; Ruhmer J; Witte OW; Grosskreutz J
J Neuroimmunol; 2014 May; 270(1-2):29-36. PubMed ID: 24666819
[TBL] [Abstract][Full Text] [Related]
8. Widespread aggregation of mutant VAPB associated with ALS does not cause motor neuron degeneration or modulate mutant SOD1 aggregation and toxicity in mice.
Qiu L; Qiao T; Beers M; Tan W; Wang H; Yang B; Xu Z
Mol Neurodegener; 2013 Jan; 8():1. PubMed ID: 23281774
[TBL] [Abstract][Full Text] [Related]
9. ERp57 is protective against mutant SOD1-induced cellular pathology in amyotrophic lateral sclerosis.
Parakh S; Jagaraj CJ; Vidal M; Ragagnin AMG; Perri ER; Konopka A; Toth RP; Galper J; Blair IP; Thomas CJ; Walker AK; Yang S; Spencer DM; Atkin JD
Hum Mol Genet; 2018 Apr; 27(8):1311-1331. PubMed ID: 29409023
[TBL] [Abstract][Full Text] [Related]
10. The UPR-PERK pathway is not a promising therapeutic target for mutant SOD1-induced ALS.
Dzhashiashvili Y; Monckton CP; Shah HS; Kunjamma RB; Popko B
Neurobiol Dis; 2019 Jul; 127():527-544. PubMed ID: 30923003
[TBL] [Abstract][Full Text] [Related]
11. Endoplasmic reticulum stress and the ER mitochondrial calcium cycle in amyotrophic lateral sclerosis.
Lautenschlaeger J; Prell T; Grosskreutz J
Amyotroph Lateral Scler; 2012 Feb; 13(2):166-77. PubMed ID: 22292840
[TBL] [Abstract][Full Text] [Related]
12. Guanabenz delays the onset of disease symptoms, extends lifespan, improves motor performance and attenuates motor neuron loss in the SOD1 G93A mouse model of amyotrophic lateral sclerosis.
Jiang HQ; Ren M; Jiang HZ; Wang J; Zhang J; Yin X; Wang SY; Qi Y; Wang XD; Feng HL
Neuroscience; 2014 Sep; 277():132-8. PubMed ID: 24699224
[TBL] [Abstract][Full Text] [Related]
13. Protein disulphide isomerase protects against protein aggregation and is S-nitrosylated in amyotrophic lateral sclerosis.
Walker AK; Farg MA; Bye CR; McLean CA; Horne MK; Atkin JD
Brain; 2010 Jan; 133(Pt 1):105-16. PubMed ID: 19903735
[TBL] [Abstract][Full Text] [Related]
14. Motor neuron disease-associated mutant vesicle-associated membrane protein-associated protein (VAP) B recruits wild-type VAPs into endoplasmic reticulum-derived tubular aggregates.
Teuling E; Ahmed S; Haasdijk E; Demmers J; Steinmetz MO; Akhmanova A; Jaarsma D; Hoogenraad CC
J Neurosci; 2007 Sep; 27(36):9801-15. PubMed ID: 17804640
[TBL] [Abstract][Full Text] [Related]
15. Extracellular wildtype and mutant SOD1 induces ER-Golgi pathology characteristic of amyotrophic lateral sclerosis in neuronal cells.
Sundaramoorthy V; Walker AK; Yerbury J; Soo KY; Farg MA; Hoang V; Zeineddine R; Spencer D; Atkin JD
Cell Mol Life Sci; 2013 Nov; 70(21):4181-95. PubMed ID: 23765103
[TBL] [Abstract][Full Text] [Related]
16. Neuroprotective effects of the Sigma-1 receptor (S1R) agonist PRE-084, in a mouse model of motor neuron disease not linked to SOD1 mutation.
Peviani M; Salvaneschi E; Bontempi L; Petese A; Manzo A; Rossi D; Salmona M; Collina S; Bigini P; Curti D
Neurobiol Dis; 2014 Feb; 62():218-32. PubMed ID: 24141020
[TBL] [Abstract][Full Text] [Related]
17. Mutant SOD1 accumulation in sensory neurons does not associate with endoplasmic reticulum stress features: Implications for differential vulnerability of sensory and motor neurons to SOD1 toxicity.
Taiana M; Sassone J; Lauria G
Neurosci Lett; 2016 Aug; 627():107-14. PubMed ID: 27241719
[TBL] [Abstract][Full Text] [Related]
18. The role of heat shock proteins in Amyotrophic Lateral Sclerosis: The therapeutic potential of Arimoclomol.
Kalmar B; Lu CH; Greensmith L
Pharmacol Ther; 2014 Jan; 141(1):40-54. PubMed ID: 23978556
[TBL] [Abstract][Full Text] [Related]
19. Mutant SOD1 inhibits ER-Golgi transport in amyotrophic lateral sclerosis.
Atkin JD; Farg MA; Soo KY; Walker AK; Halloran M; Turner BJ; Nagley P; Horne MK
J Neurochem; 2014 Apr; 129(1):190-204. PubMed ID: 24134191
[TBL] [Abstract][Full Text] [Related]
20. Derlin-1 overexpression ameliorates mutant SOD1-induced endoplasmic reticulum stress by reducing mutant SOD1 accumulation.
Mori A; Yamashita S; Uchino K; Suga T; Ikeda T; Takamatsu K; Ishizaki M; Koide T; Kimura E; Mita S; Maeda Y; Hirano T; Uchino M
Neurochem Int; 2011 Feb; 58(3):344-53. PubMed ID: 21185345
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]