674 related articles for article (PubMed ID: 21914486)
1. The Aβ oligomer hypothesis for synapse failure and memory loss in Alzheimer's disease.
Ferreira ST; Klein WL
Neurobiol Learn Mem; 2011 Nov; 96(4):529-43. PubMed ID: 21914486
[TBL] [Abstract][Full Text] [Related]
2. Synaptotoxic amyloid-β oligomers: a molecular basis for the cause, diagnosis, and treatment of Alzheimer's disease?
Klein WL
J Alzheimers Dis; 2013; 33 Suppl 1():S49-65. PubMed ID: 22785404
[TBL] [Abstract][Full Text] [Related]
3. Is Alzheimer's disease a result of presynaptic failure? Synaptic dysfunctions induced by oligomeric beta-amyloid.
Nimmrich V; Ebert U
Rev Neurosci; 2009; 20(1):1-12. PubMed ID: 19526730
[TBL] [Abstract][Full Text] [Related]
4. Amyloid β oligomers in Alzheimer's disease pathogenesis, treatment, and diagnosis.
Viola KL; Klein WL
Acta Neuropathol; 2015 Feb; 129(2):183-206. PubMed ID: 25604547
[TBL] [Abstract][Full Text] [Related]
5. Intraneuronal beta-amyloid accumulation and synapse pathology in Alzheimer's disease.
Gouras GK; Tampellini D; Takahashi RH; Capetillo-Zarate E
Acta Neuropathol; 2010 May; 119(5):523-41. PubMed ID: 20354705
[TBL] [Abstract][Full Text] [Related]
6. Synaptic Mitochondria: An Early Target of Amyloid-β and Tau in Alzheimer's Disease.
Torres AK; Jara C; Park-Kang HS; Polanco CM; Tapia D; Alarcón F; de la Peña A; Llanquinao J; Vargas-Mardones G; Indo JA; Inestrosa NC; Tapia-Rojas C
J Alzheimers Dis; 2021; 84(4):1391-1414. PubMed ID: 34719499
[TBL] [Abstract][Full Text] [Related]
7. Dispersible amyloid β-protein oligomers, protofibrils, and fibrils represent diffusible but not soluble aggregates: their role in neurodegeneration in amyloid precursor protein (APP) transgenic mice.
Rijal Upadhaya A; Capetillo-Zarate E; Kosterin I; Abramowski D; Kumar S; Yamaguchi H; Walter J; Fändrich M; Staufenbiel M; Thal DR
Neurobiol Aging; 2012 Nov; 33(11):2641-60. PubMed ID: 22305478
[TBL] [Abstract][Full Text] [Related]
8. Amyloid-β induced signaling by cellular prion protein and Fyn kinase in Alzheimer disease.
Um JW; Strittmatter SM
Prion; 2013; 7(1):37-41. PubMed ID: 22987042
[TBL] [Abstract][Full Text] [Related]
9. Alzheimer's therapeutics targeting amyloid beta 1-42 oligomers II: Sigma-2/PGRMC1 receptors mediate Abeta 42 oligomer binding and synaptotoxicity.
Izzo NJ; Xu J; Zeng C; Kirk MJ; Mozzoni K; Silky C; Rehak C; Yurko R; Look G; Rishton G; Safferstein H; Cruchaga C; Goate A; Cahill MA; Arancio O; Mach RH; Craven R; Head E; LeVine H; Spires-Jones TL; Catalano SM
PLoS One; 2014; 9(11):e111899. PubMed ID: 25390692
[TBL] [Abstract][Full Text] [Related]
10. RAPGEF2 mediates oligomeric Aβ-induced synaptic loss and cognitive dysfunction in the 3xTg-AD mouse model of Alzheimer's disease.
Jang YN; Jang H; Kim GH; Noh JE; Chang KA; Lee KJ
Neuropathol Appl Neurobiol; 2021 Aug; 47(5):625-639. PubMed ID: 33345400
[TBL] [Abstract][Full Text] [Related]
11. Monomeric amyloid-β reduced amyloid-β oligomer-induced synapse damage in neuronal cultures.
Bate C; Williams A
Neurobiol Dis; 2018 Mar; 111():48-58. PubMed ID: 29272738
[TBL] [Abstract][Full Text] [Related]
12. Human Brain-Derived Aβ Oligomers Bind to Synapses and Disrupt Synaptic Activity in a Manner That Requires APP.
Wang Z; Jackson RJ; Hong W; Taylor WM; Corbett GT; Moreno A; Liu W; Li S; Frosch MP; Slutsky I; Young-Pearse TL; Spires-Jones TL; Walsh DM
J Neurosci; 2017 Dec; 37(49):11947-11966. PubMed ID: 29101243
[TBL] [Abstract][Full Text] [Related]
13. [Involvement of beta-amyloid in the etiology of Alzheimer's disease].
Tomiyama T
Brain Nerve; 2010 Jul; 62(7):691-9. PubMed ID: 20675873
[TBL] [Abstract][Full Text] [Related]
14. Human tau increases amyloid β plaque size but not amyloid β-mediated synapse loss in a novel mouse model of Alzheimer's disease.
Jackson RJ; Rudinskiy N; Herrmann AG; Croft S; Kim JM; Petrova V; Ramos-Rodriguez JJ; Pitstick R; Wegmann S; Garcia-Alloza M; Carlson GA; Hyman BT; Spires-Jones TL
Eur J Neurosci; 2016 Dec; 44(12):3056-3066. PubMed ID: 27748574
[TBL] [Abstract][Full Text] [Related]
15. Soluble amyloid beta peptide concentration as a predictor of synaptic change in Alzheimer's disease.
Lue LF; Kuo YM; Roher AE; Brachova L; Shen Y; Sue L; Beach T; Kurth JH; Rydel RE; Rogers J
Am J Pathol; 1999 Sep; 155(3):853-62. PubMed ID: 10487842
[TBL] [Abstract][Full Text] [Related]
16. Accumulation of cellular prion protein within β-amyloid oligomer plaques in aged human brains.
Takahashi RH; Yokotsuka M; Tobiume M; Sato Y; Hasegawa H; Nagao T; Gouras GK
Brain Pathol; 2021 Sep; 31(5):e12941. PubMed ID: 33624334
[TBL] [Abstract][Full Text] [Related]
17. Non-Fibrillar Oligomeric Amyloid-β within Synapses.
Pickett EK; Koffie RM; Wegmann S; Henstridge CM; Herrmann AG; Colom-Cadena M; Lleo A; Kay KR; Vaught M; Soberman R; Walsh DM; Hyman BT; Spires-Jones TL
J Alzheimers Dis; 2016 May; 53(3):787-800. PubMed ID: 27258414
[TBL] [Abstract][Full Text] [Related]
18. β-Amyloid peptides and amyloid plaques in Alzheimer's disease.
Gouras GK; Olsson TT; Hansson O
Neurotherapeutics; 2015 Jan; 12(1):3-11. PubMed ID: 25371168
[TBL] [Abstract][Full Text] [Related]
19. Stabilized Low-n Amyloid-β Oligomers Induce Robust Novel Object Recognition Deficits Associated with Inflammatory, Synaptic, and GABAergic Dysfunction in the Rat.
Watremez W; Jackson J; Almari B; McLean SL; Grayson B; Neill JC; Fischer N; Allouche A; Koziel V; Pillot T; Harte MK
J Alzheimers Dis; 2018; 62(1):213-226. PubMed ID: 29439327
[TBL] [Abstract][Full Text] [Related]
20. Tooth Loss Induces Memory Impairment and Gliosis in App Knock-In Mouse Models of Alzheimer's Disease.
Taslima F; Jung CG; Zhou C; Abdelhamid M; Abdullah M; Goto T; Saito T; Saido TC; Michikawa M
J Alzheimers Dis; 2021; 80(4):1687-1704. PubMed ID: 33720883
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]