These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

165 related articles for article (PubMed ID: 21951640)

  • 1. Conserved cysteine residues in the mammalian lamin A tail are essential for cellular responses to ROS generation.
    Pekovic V; Gibbs-Seymour I; Markiewicz E; Alzoghaibi F; Benham AM; Edwards R; Wenhert M; von Zglinicki T; Hutchison CJ
    Aging Cell; 2011 Dec; 10(6):1067-79. PubMed ID: 21951640
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Sustained accumulation of prelamin A and depletion of lamin A/C both cause oxidative stress and mitochondrial dysfunction but induce different cell fates.
    Sieprath T; Corne TD; Nooteboom M; Grootaert C; Rajkovic A; Buysschaert B; Robijns J; Broers JL; Ramaekers FC; Koopman WJ; Willems PH; De Vos WH
    Nucleus; 2015; 6(3):236-46. PubMed ID: 25996284
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Insulin-like growth factor-1 attenuates oxidative stress-induced hepatocyte premature senescence in liver fibrogenesis via regulating nuclear p53-progerin interaction.
    Luo X; Jiang X; Li J; Bai Y; Li Z; Wei P; Sun S; Liang Y; Han S; Li X; Zhang B
    Cell Death Dis; 2019 Jun; 10(6):451. PubMed ID: 31171766
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Emery-Dreifuss Muscular Dystrophy-Associated Mutant Forms of Lamin A Recruit the Stress Responsive Protein Ankrd2 into the Nucleus, Affecting the Cellular Response to Oxidative Stress.
    Angori S; Capanni C; Faulkner G; Bean C; Boriani G; Lattanzi G; Cenni V
    Cell Physiol Biochem; 2017; 42(1):169-184. PubMed ID: 28531892
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Upregulated expression of lamin B receptor increases cell proliferation and suppresses genomic instability: implications for cellular immortalization.
    En A; Takemoto K; Yamakami Y; Nakabayashi K; Fujii M
    FEBS J; 2024 May; 291(10):2155-2171. PubMed ID: 38462947
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Embryonic senescence and laminopathies in a progeroid zebrafish model.
    Koshimizu E; Imamura S; Qi J; Toure J; Valdez DM; Carr CE; Hanai J; Kishi S
    PLoS One; 2011 Mar; 6(3):e17688. PubMed ID: 21479207
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Changes in vimentin, lamin A/C and mitofilin induce aberrant cell organization in fibroblasts from Fanconi anemia complementation group A (FA-A) patients.
    Capanni C; Bruschi M; Columbaro M; Cuccarolo P; Ravera S; Dufour C; Candiano G; Petretto A; Degan P; Cappelli E
    Biochimie; 2013 Oct; 95(10):1838-47. PubMed ID: 23831462
    [TBL] [Abstract][Full Text] [Related]  

  • 8. The last twenty residues in the head domain of mouse lamin A contain important structural elements for formation of head-to-tail polymers in vitro.
    Isobe K; Gohara R; Ueda T; Takasaki Y; Ando S
    Biosci Biotechnol Biochem; 2007 May; 71(5):1252-9. PubMed ID: 17485847
    [TBL] [Abstract][Full Text] [Related]  

  • 9. The tail domain of lamin B1 is more strongly modulated by divalent cations than lamin A.
    Ganesh S; Qin Z; Spagnol ST; Biegler MT; Coffey KA; Kalinowski A; Buehler MJ; Dahl KN
    Nucleus; 2015; 6(3):203-11. PubMed ID: 25807068
    [TBL] [Abstract][Full Text] [Related]  

  • 10. The R439C mutation in LMNA causes lamin oligomerization and susceptibility to oxidative stress.
    Verstraeten VL; Caputo S; van Steensel MA; Duband-Goulet I; Zinn-Justin S; Kamps M; Kuijpers HJ; Ostlund C; Worman HJ; Briedé JJ; Le Dour C; Marcelis CL; van Geel M; Steijlen PM; van den Wijngaard A; Ramaekers FC; Broers JL
    J Cell Mol Med; 2009 May; 13(5):959-71. PubMed ID: 19220582
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Nuclear lamins and progerin are dispensable for antioxidant Nrf2 response to arsenic and cadmium.
    Hashimoto K; Majumdar R; Tsuji Y
    Cell Signal; 2017 May; 33():69-78. PubMed ID: 28229933
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Lamin A safeguards the m
    Zhang J; Ao Y; Zhang Z; Mo Y; Peng L; Jiang Y; Wang Z; Liu B
    Aging Cell; 2020 Oct; 19(10):e13215. PubMed ID: 32813328
    [TBL] [Abstract][Full Text] [Related]  

  • 13. A drug-induced accelerated senescence (DIAS) is a possibility to study aging in time lapse.
    Alili L; Diekmann J; Giesen M; Holtkötter O; Brenneisen P
    Age (Dordr); 2014 Jun; 36(3):9658. PubMed ID: 24833306
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Protective effects of rosmarinic acid against hydrogen peroxide‑induced cellular senescence and the inflammatory response in normal human dermal fibroblasts.
    Hahn HJ; Kim KB; An IS; Ahn KJ; Han HJ
    Mol Med Rep; 2017 Dec; 16(6):9763-9769. PubMed ID: 29039587
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Progerin and telomere dysfunction collaborate to trigger cellular senescence in normal human fibroblasts.
    Cao K; Blair CD; Faddah DA; Kieckhaefer JE; Olive M; Erdos MR; Nabel EG; Collins FS
    J Clin Invest; 2011 Jul; 121(7):2833-44. PubMed ID: 21670498
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Cordyceps militaris extract protects human dermal fibroblasts against oxidative stress-induced apoptosis and premature senescence.
    Park JM; Lee JS; Lee KR; Ha SJ; Hong EK
    Nutrients; 2014 Sep; 6(9):3711-26. PubMed ID: 25230212
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Human lipodystrophies linked to mutations in A-type lamins and to HIV protease inhibitor therapy are both associated with prelamin A accumulation, oxidative stress and premature cellular senescence.
    Caron M; Auclair M; Donadille B; Béréziat V; Guerci B; Laville M; Narbonne H; Bodemer C; Lascols O; Capeau J; Vigouroux C
    Cell Death Differ; 2007 Oct; 14(10):1759-67. PubMed ID: 17612587
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Comparing lamin proteins post-translational relative stability using a 2A peptide-based system reveals elevated resistance of progerin to cellular degradation.
    Wu D; Yates PA; Zhang H; Cao K
    Nucleus; 2016 Nov; 7(6):585-596. PubMed ID: 27929926
    [TBL] [Abstract][Full Text] [Related]  

  • 19. A conserved splicing mechanism of the LMNA gene controls premature aging.
    Lopez-Mejia IC; Vautrot V; De Toledo M; Behm-Ansmant I; Bourgeois CF; Navarro CL; Osorio FG; Freije JM; Stévenin J; De Sandre-Giovannoli A; Lopez-Otin C; Lévy N; Branlant C; Tazi J
    Hum Mol Genet; 2011 Dec; 20(23):4540-55. PubMed ID: 21875900
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Incomplete processing of mutant lamin A in Hutchinson-Gilford progeria leads to nuclear abnormalities, which are reversed by farnesyltransferase inhibition.
    Glynn MW; Glover TW
    Hum Mol Genet; 2005 Oct; 14(20):2959-69. PubMed ID: 16126733
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 9.