These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

352 related articles for article (PubMed ID: 22039056)

  • 1. RECQL4, the protein mutated in Rothmund-Thomson syndrome, functions in telomere maintenance.
    Ghosh AK; Rossi ML; Singh DK; Dunn C; Ramamoorthy M; Croteau DL; Liu Y; Bohr VA
    J Biol Chem; 2012 Jan; 287(1):196-209. PubMed ID: 22039056
    [TBL] [Abstract][Full Text] [Related]  

  • 2. The RECQL4 protein, deficient in Rothmund-Thomson syndrome is active on telomeric D-loops containing DNA metabolism blocking lesions.
    Ferrarelli LK; Popuri V; Ghosh AK; Tadokoro T; Canugovi C; Hsu JK; Croteau DL; Bohr VA
    DNA Repair (Amst); 2013 Jul; 12(7):518-28. PubMed ID: 23683351
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Senescence induced by RECQL4 dysfunction contributes to Rothmund-Thomson syndrome features in mice.
    Lu H; Fang EF; Sykora P; Kulikowicz T; Zhang Y; Becker KG; Croteau DL; Bohr VA
    Cell Death Dis; 2014 May; 5(5):e1226. PubMed ID: 24832598
    [TBL] [Abstract][Full Text] [Related]  

  • 4. POT1 stimulates RecQ helicases WRN and BLM to unwind telomeric DNA substrates.
    Opresko PL; Mason PA; Podell ER; Lei M; Hickson ID; Cech TR; Bohr VA
    J Biol Chem; 2005 Sep; 280(37):32069-80. PubMed ID: 16030011
    [TBL] [Abstract][Full Text] [Related]  

  • 5. TRF1 negotiates TTAGGG repeat-associated replication problems by recruiting the BLM helicase and the TPP1/POT1 repressor of ATR signaling.
    Zimmermann M; Kibe T; Kabir S; de Lange T
    Genes Dev; 2014 Nov; 28(22):2477-91. PubMed ID: 25344324
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Human RECQL1 participates in telomere maintenance.
    Popuri V; Hsu J; Khadka P; Horvath K; Liu Y; Croteau DL; Bohr VA
    Nucleic Acids Res; 2014 May; 42(9):5671-88. PubMed ID: 24623817
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Werner syndrome protein suppresses the formation of large deletions during the replication of human telomeric sequences.
    Damerla RR; Knickelbein KE; Strutt S; Liu FJ; Wang H; Opresko PL
    Cell Cycle; 2012 Aug; 11(16):3036-44. PubMed ID: 22871734
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Telomere-binding protein TRF2 binds to and stimulates the Werner and Bloom syndrome helicases.
    Opresko PL; von Kobbe C; Laine JP; Harrigan J; Hickson ID; Bohr VA
    J Biol Chem; 2002 Oct; 277(43):41110-9. PubMed ID: 12181313
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Direct and indirect roles of RECQL4 in modulating base excision repair capacity.
    Schurman SH; Hedayati M; Wang Z; Singh DK; Speina E; Zhang Y; Becker K; Macris M; Sung P; Wilson DM; Croteau DL; Bohr VA
    Hum Mol Genet; 2009 Sep; 18(18):3470-83. PubMed ID: 19567405
    [TBL] [Abstract][Full Text] [Related]  

  • 10. ATM activation is impaired in human cells defective in RecQL4 helicase activity.
    Park SY; Kim H; Im JS; Lee JK
    Biochem Biophys Res Commun; 2019 Feb; 509(2):379-383. PubMed ID: 30594395
    [TBL] [Abstract][Full Text] [Related]  

  • 11. RECQ helicase RECQL4 participates in non-homologous end joining and interacts with the Ku complex.
    Shamanna RA; Singh DK; Lu H; Mirey G; Keijzers G; Salles B; Croteau DL; Bohr VA
    Carcinogenesis; 2014 Nov; 35(11):2415-24. PubMed ID: 24942867
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Sensitivity of RECQL4-deficient fibroblasts from Rothmund-Thomson syndrome patients to genotoxic agents.
    Jin W; Liu H; Zhang Y; Otta SK; Plon SE; Wang LL
    Hum Genet; 2008 Jul; 123(6):643-53. PubMed ID: 18504617
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Telomeric D-loops containing 8-oxo-2'-deoxyguanosine are preferred substrates for Werner and Bloom syndrome helicases and are bound by POT1.
    Ghosh A; Rossi ML; Aulds J; Croteau D; Bohr VA
    J Biol Chem; 2009 Nov; 284(45):31074-84. PubMed ID: 19734539
    [TBL] [Abstract][Full Text] [Related]  

  • 14. RECQL4 in genomic instability and aging.
    Croteau DL; Singh DK; Hoh Ferrarelli L; Lu H; Bohr VA
    Trends Genet; 2012 Dec; 28(12):624-31. PubMed ID: 22940096
    [TBL] [Abstract][Full Text] [Related]  

  • 15. The Werner syndrome helicase and exonuclease cooperate to resolve telomeric D loops in a manner regulated by TRF1 and TRF2.
    Opresko PL; Otterlei M; Graakjaer J; Bruheim P; Dawut L; Kølvraa S; May A; Seidman MM; Bohr VA
    Mol Cell; 2004 Jun; 14(6):763-74. PubMed ID: 15200954
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Sequence-specific processing of telomeric 3' overhangs by the Werner syndrome protein exonuclease activity.
    Li B; Reddy S; Comai L
    Aging (Albany NY); 2009 Mar; 1(3):289-302. PubMed ID: 20157518
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Telomeric protein TRF2 protects Holliday junctions with telomeric arms from displacement by the Werner syndrome helicase.
    Nora GJ; Buncher NA; Opresko PL
    Nucleic Acids Res; 2010 Jul; 38(12):3984-98. PubMed ID: 20215438
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Novel function of the Fanconi anemia group J or RECQ1 helicase to disrupt protein-DNA complexes in a replication protein A-stimulated manner.
    Sommers JA; Banerjee T; Hinds T; Wan B; Wold MS; Lei M; Brosh RM
    J Biol Chem; 2014 Jul; 289(29):19928-41. PubMed ID: 24895130
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Cooperation of DNA-PKcs and WRN helicase in the maintenance of telomeric D-loops.
    Kusumoto-Matsuo R; Opresko PL; Ramsden D; Tahara H; Bohr VA
    Aging (Albany NY); 2010 May; 2(5):274-84. PubMed ID: 20519774
    [TBL] [Abstract][Full Text] [Related]  

  • 20. The molecular role of the Rothmund-Thomson-, RAPADILINO- and Baller-Gerold-gene product, RECQL4: recent progress.
    Dietschy T; Shevelev I; Stagljar I
    Cell Mol Life Sci; 2007 Apr; 64(7-8):796-802. PubMed ID: 17364146
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 18.