BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

270 related articles for article (PubMed ID: 22174687)

  • 1. Sialidases affect the host cell adherence and epsilon toxin-induced cytotoxicity of Clostridium perfringens type D strain CN3718.
    Li J; Sayeed S; Robertson S; Chen J; McClane BA
    PLoS Pathog; 2011 Dec; 7(12):e1002429. PubMed ID: 22174687
    [TBL] [Abstract][Full Text] [Related]  

  • 2. NanI Sialidase, CcpA, and CodY Work Together To Regulate Epsilon Toxin Production by Clostridium perfringens Type D Strain CN3718.
    Li J; Freedman JC; McClane BA
    J Bacteriol; 2015 Oct; 197(20):3339-53. PubMed ID: 26260460
    [TBL] [Abstract][Full Text] [Related]  

  • 3. The Sialidases of Clostridium perfringens type D strain CN3718 differ in their properties and sensitivities to inhibitors.
    Li J; McClane BA
    Appl Environ Microbiol; 2014 Mar; 80(5):1701-9. PubMed ID: 24375134
    [TBL] [Abstract][Full Text] [Related]  

  • 4. CodY is a global regulator of virulence-associated properties for Clostridium perfringens type D strain CN3718.
    Li J; Ma M; Sarker MR; McClane BA
    mBio; 2013 Oct; 4(5):e00770-13. PubMed ID: 24105766
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Epsilon-toxin production by Clostridium perfringens type D strain CN3718 is dependent upon the agr operon but not the VirS/VirR two-component regulatory system.
    Chen J; Rood JI; McClane BA
    mBio; 2011; 2(6):. PubMed ID: 22167225
    [TBL] [Abstract][Full Text] [Related]  

  • 6. NanJ Is the Major Sialidase for Clostridium perfringens Type F Food Poisoning Strain 01E809.
    Li J; Pradhan A; McClane BA
    Infect Immun; 2023 Jun; 91(6):e0005323. PubMed ID: 37212696
    [TBL] [Abstract][Full Text] [Related]  

  • 7. The NanI and NanJ sialidases of Clostridium perfringens are not essential for virulence.
    Chiarezza M; Lyras D; Pidot SJ; Flores-Díaz M; Awad MM; Kennedy CL; Cordner LM; Phumoonna T; Poon R; Hughes ML; Emmins JJ; Alape-Girón A; Rood JI
    Infect Immun; 2009 Oct; 77(10):4421-8. PubMed ID: 19651873
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Contributions of NanI sialidase to Caco-2 cell adherence by Clostridium perfringens type A and C strains causing human intestinal disease.
    Li J; McClane BA
    Infect Immun; 2014 Nov; 82(11):4620-30. PubMed ID: 25135687
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Native or Proteolytically Activated NanI Sialidase Enhances the Binding and Cytotoxic Activity of Clostridium perfringens Enterotoxin and Beta Toxin.
    Theoret JR; Li J; Navarro MA; Garcia JP; Uzal FA; McClane BA
    Infect Immun; 2018 Jan; 86(1):. PubMed ID: 29038129
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Clostridium perfringens Sialidases: Potential Contributors to Intestinal Pathogenesis and Therapeutic Targets.
    Li J; Uzal FA; McClane BA
    Toxins (Basel); 2016 Nov; 8(11):. PubMed ID: 27869757
    [No Abstract]   [Full Text] [Related]  

  • 11. NanR, a Transcriptional Regulator That Binds to the Promoters of Genes Involved in Sialic Acid Metabolism in the Anaerobic Pathogen Clostridium perfringens.
    Therit B; Cheung JK; Rood JI; Melville SB
    PLoS One; 2015; 10(7):e0133217. PubMed ID: 26197388
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Sialidases From
    Wang YH
    Front Cell Infect Microbiol; 2019; 9():462. PubMed ID: 31998664
    [No Abstract]   [Full Text] [Related]  

  • 13. NanI Sialidase Is an Important Contributor to Clostridium perfringens Type F Strain F4969 Intestinal Colonization in Mice.
    Navarro MA; Li J; McClane BA; Morrell E; Beingesser J; Uzal FA
    Infect Immun; 2018 Dec; 86(12):. PubMed ID: 30297524
    [No Abstract]   [Full Text] [Related]  

  • 14. NanH Is Produced by Sporulating Cultures of Clostridium perfringens Type F Food Poisoning Strains and Enhances the Cytotoxicity of C. perfringens Enterotoxin.
    Li J; McClane BA
    mSphere; 2021 Apr; 6(2):. PubMed ID: 33910991
    [No Abstract]   [Full Text] [Related]  

  • 15. NanI sialidase contributes to toxin expression and host cell binding of Clostridium perfringens type G strain CP56 in vitro.
    Van Damme L; Callens C; Dargatz M; Flügel M; Hark S; Thiemann F; Pelzer S; Ducatelle R; Van Immerseel F; Goossens E
    Vet Microbiol; 2022 Mar; 266():109371. PubMed ID: 35176607
    [TBL] [Abstract][Full Text] [Related]  

  • 16. NanR Regulates
    Li J; Evans DR; Freedman JC; McClane BA
    Infect Immun; 2017 Sep; 85(9):. PubMed ID: 28652312
    [No Abstract]   [Full Text] [Related]  

  • 17. NanI Sialidase Can Support the Growth and Survival of Clostridium perfringens Strain F4969 in the Presence of Sialyated Host Macromolecules (Mucin) or Caco-2 Cells.
    Li J; McClane BA
    Infect Immun; 2018 Feb; 86(2):. PubMed ID: 29203541
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Regulation of sialidase production in Clostridium perfringens by the orphan sensor histidine kinase ReeS.
    Hiscox TJ; Harrison PF; Chakravorty A; Choo JM; Ohtani K; Shimizu T; Cheung JK; Rood JI
    PLoS One; 2013; 8(9):e73525. PubMed ID: 24023881
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Epsilon toxin is essential for the virulence of Clostridium perfringens type D infection in sheep, goats, and mice.
    Garcia JP; Adams V; Beingesser J; Hughes ML; Poon R; Lyras D; Hill A; McClane BA; Rood JI; Uzal FA
    Infect Immun; 2013 Jul; 81(7):2405-14. PubMed ID: 23630957
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Reevaluation of whether a Functional Agr-like Quorum-Sensing System Is Necessary for Production of Wild-Type Levels of Epsilon-Toxin by Clostridium perfringens Type D Strains.
    Mehdizadeh Gohari I; Li J; Rood JI; McClane BA
    mBio; 2022 Apr; 13(2):e0049622. PubMed ID: 35319233
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 14.