BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

409 related articles for article (PubMed ID: 22287626)

  • 1. Evolution and function of CAG/polyglutamine repeats in protein-protein interaction networks.
    Schaefer MH; Wanker EE; Andrade-Navarro MA
    Nucleic Acids Res; 2012 May; 40(10):4273-87. PubMed ID: 22287626
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Correlation of inter-locus polyglutamine toxicity with CAG•CTG triplet repeat expandability and flanking genomic DNA GC content.
    Nestor CE; Monckton DG
    PLoS One; 2011; 6(12):e28260. PubMed ID: 22163004
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Coiled-coil structure-dependent interactions between polyQ proteins and Foxo lead to dendrite pathology and behavioral defects.
    Kwon MJ; Han MH; Bagley JA; Hyeon DY; Ko BS; Lee YM; Cha IJ; Kim SY; Kim DY; Kim HM; Hwang D; Lee SB; Jan YN
    Proc Natl Acad Sci U S A; 2018 Nov; 115(45):E10748-E10757. PubMed ID: 30348793
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Polyglutamine Repeats in Neurodegenerative Diseases.
    Lieberman AP; Shakkottai VG; Albin RL
    Annu Rev Pathol; 2019 Jan; 14():1-27. PubMed ID: 30089230
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Polyglutamine expansion diseases: More than simple repeats.
    Silva A; de Almeida AV; Macedo-Ribeiro S
    J Struct Biol; 2018 Feb; 201(2):139-154. PubMed ID: 28928079
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Increased aggregation of polyleucine compared with that of polyglutamine in dentatorubral-pallidoluysian atrophy protein.
    Suzuki Y; Jin C; Yazawa I
    Neurosci Lett; 2013 Sep; 552():156-61. PubMed ID: 23933208
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Polyglutamine disease proteins: Commonalities and differences in interaction profiles and pathological effects.
    Bonsor M; Ammar O; Schnoegl S; Wanker EE; Silva Ramos E
    Proteomics; 2024 Jun; 24(12-13):e2300114. PubMed ID: 38615323
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Interaction of expanded polyglutamine stretches with nuclear transcription factors leads to aberrant transcriptional regulation in polyglutamine diseases.
    Shimohata T; Onodera O; Tsuji S
    Neuropathology; 2000 Dec; 20(4):326-33. PubMed ID: 11211059
    [TBL] [Abstract][Full Text] [Related]  

  • 9. A peptidylic inhibitor for neutralizing expanded
    Zhang Q; Chen ZS; An Y; Liu H; Hou Y; Li W; Lau KF; Koon AC; Ngo JCK; Chan HYE
    RNA; 2018 Apr; 24(4):486-498. PubMed ID: 29295891
    [TBL] [Abstract][Full Text] [Related]  

  • 10. The interplay between PolyQ and protein context delays aggregation by forming a reservoir of protofibrils.
    Bulone D; Masino L; Thomas DJ; San Biagio PL; Pastore A
    PLoS One; 2006 Dec; 1(1):e111. PubMed ID: 17205115
    [TBL] [Abstract][Full Text] [Related]  

  • 11. The features of polyglutamine regions depend on their evolutionary stability.
    Mier P; Andrade-Navarro MA
    BMC Evol Biol; 2020 May; 20(1):59. PubMed ID: 32448113
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Proteins Containing Expanded Polyglutamine Tracts and Neurodegenerative Disease.
    Adegbuyiro A; Sedighi F; Pilkington AW; Groover S; Legleiter J
    Biochemistry; 2017 Mar; 56(9):1199-1217. PubMed ID: 28170216
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Polyglutamine Repeats in Viruses.
    Schein CH
    Mol Neurobiol; 2019 May; 56(5):3664-3675. PubMed ID: 30182336
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Expanded polyglutamine stretches lead to aberrant transcriptional regulation in polyglutamine diseases.
    Shimohata T; Onodera O; Tsuji S
    Hum Cell; 2001 Mar; 14(1):17-25. PubMed ID: 11436350
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Evidence for a recruitment and sequestration mechanism in Huntington's disease.
    Preisinger E; Jordan BM; Kazantsev A; Housman D
    Philos Trans R Soc Lond B Biol Sci; 1999 Jun; 354(1386):1029-34. PubMed ID: 10434302
    [TBL] [Abstract][Full Text] [Related]  

  • 16. CAG Expansions Are Genetically Stable and Form Nontoxic Aggregates in Cells Lacking Endogenous Polyglutamine Proteins.
    Zurawel AA; Kabeche R; DiGregorio SE; Deng L; Menon KM; Opalko H; Duennwald ML; Moseley JB; Supattapone S
    mBio; 2016 Sep; 7(5):. PubMed ID: 27677791
    [TBL] [Abstract][Full Text] [Related]  

  • 17. [The Role of Mutant RNA in the Pathogenesis of Huntington's Disease and Other Polyglutamine Diseases].
    Bogomazova AN; Eremeev AV; Pozmogova GE; Lagarkova MA
    Mol Biol (Mosk); 2019; 53(6):954-967. PubMed ID: 31876275
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Association of polyalanine and polyglutamine coiled coils mediates expansion disease-related protein aggregation and dysfunction.
    Pelassa I; Corà D; Cesano F; Monje FJ; Montarolo PG; Fiumara F
    Hum Mol Genet; 2014 Jul; 23(13):3402-20. PubMed ID: 24497578
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Polyglutamine expansion mutation yields a pathological epitope linked to nucleation of protein aggregate: determinant of Huntington's disease onset.
    Sugaya K; Matsubara S; Kagamihara Y; Kawata A; Hayashi H
    PLoS One; 2007 Jul; 2(7):e635. PubMed ID: 17653262
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Current understanding on the pathogenesis of polyglutamine diseases.
    He XH; Lin F; Qin ZH
    Neurosci Bull; 2010 Jun; 26(3):247-56. PubMed ID: 20502504
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 21.