215 related articles for article (PubMed ID: 22493079)
1. TASK-3 channel deletion in mice recapitulates low-renin essential hypertension.
Guagliardo NA; Yao J; Hu C; Schertz EM; Tyson DA; Carey RM; Bayliss DA; Barrett PQ
Hypertension; 2012 May; 59(5):999-1005. PubMed ID: 22493079
[TBL] [Abstract][Full Text] [Related]
2. Severe hyperaldosteronism in neonatal Task3 potassium channel knockout mice is associated with activation of the intraadrenal renin-angiotensin system.
Bandulik S; Tauber P; Penton D; Schweda F; Tegtmeier I; Sterner C; Lalli E; Lesage F; Hartmann M; Barhanin J; Warth R
Endocrinology; 2013 Aug; 154(8):2712-22. PubMed ID: 23698720
[TBL] [Abstract][Full Text] [Related]
3. Adrenal Tissue-Specific Deletion of TASK Channels Causes Aldosterone-Driven Angiotensin II-Independent Hypertension.
Guagliardo NA; Yao J; Stipes EJ; Cechova S; Le TH; Bayliss DA; Breault DT; Barrett PQ
Hypertension; 2019 Feb; 73(2):407-414. PubMed ID: 30580687
[TBL] [Abstract][Full Text] [Related]
4. TASK channel deletion in mice causes primary hyperaldosteronism.
Davies LA; Hu C; Guagliardo NA; Sen N; Chen X; Talley EM; Carey RM; Bayliss DA; Barrett PQ
Proc Natl Acad Sci U S A; 2008 Feb; 105(6):2203-8. PubMed ID: 18250325
[TBL] [Abstract][Full Text] [Related]
5. Dopamine reduces aldosterone and 18-hydroxycorticosterone response to angiotensin II in patients with essential low-renin hypertension and idiopathic hyperaldosteronism.
Witzgall H; Lorenz R; von Werder K; Weber PC
Clin Sci (Lond); 1985 Mar; 68(3):291-9. PubMed ID: 3882312
[TBL] [Abstract][Full Text] [Related]
6. The syndromes of low-renin hypertension: "separating the wheat from the chaff".
Kater CE; Biglieri EG
Arq Bras Endocrinol Metabol; 2004 Oct; 48(5):674-81. PubMed ID: 15761538
[TBL] [Abstract][Full Text] [Related]
7. Hypertension: renin-angiotensin-aldosterone system alterations.
Te Riet L; van Esch JH; Roks AJ; van den Meiracker AH; Danser AH
Circ Res; 2015 Mar; 116(6):960-75. PubMed ID: 25767283
[TBL] [Abstract][Full Text] [Related]
8. Altered adrenal sensitivity to angiotensin II in low-renin essential hypertension.
Fisher ND; Hurwitz S; Ferri C; Jeunemaitre X; Hollenberg NK; Williams GH
Hypertension; 1999 Sep; 34(3):388-94. PubMed ID: 10489382
[TBL] [Abstract][Full Text] [Related]
9. Intrinsic Adrenal TWIK-Related Acid-Sensitive TASK Channel Dysfunction Produces Spontaneous Calcium Oscillations Sufficient to Drive AngII (Angiotensin II)-Unresponsive Hyperaldosteronism.
Gancayco CA; Gerding MR; Breault DT; Beenhakker MP; Barrett PQ; Guagliardo NA
Hypertension; 2022 Nov; 79(11):2552-2564. PubMed ID: 36129175
[TBL] [Abstract][Full Text] [Related]
10. QT interval in patients with primary aldosteronism and low-renin essential hypertension.
Maule S; Mulatero P; Milan A; Leotta G; Caserta M; Bertello C; Rabbia F; Veglio F
J Hypertens; 2006 Dec; 24(12):2459-64. PubMed ID: 17082730
[TBL] [Abstract][Full Text] [Related]
11. Salt-sensitive blood pressure in mice with increased expression of aldosterone synthase.
Makhanova N; Hagaman J; Kim HS; Smithies O
Hypertension; 2008 Jan; 51(1):134-40. PubMed ID: 18039983
[TBL] [Abstract][Full Text] [Related]
12. Effect of angiotensin II and converting enzyme inhibitor (captopril) on blood pressure, plasma renin activity and aldosterone in primary aldosteronism.
Mantero F; Fallo F; Opocher G; Armanini D; Boscaro M; Scaroni C
Clin Sci (Lond); 1981 Dec; 61 Suppl 7():289s-293s. PubMed ID: 7032817
[TBL] [Abstract][Full Text] [Related]
13. The pressor response to angiotensin II in patients with low renin essential hypertension.
Marks AD; Marks DB; Kim YN; Moctezuma J; Adlin EV; Channick BJ
Circ Res; 1978 Jun; 42(6):864-9. PubMed ID: 657448
[TBL] [Abstract][Full Text] [Related]
14. [Studies on abnormalities of adrenal steroidogenesis in essential hypertension, primary aldosteronism and renovascular hypertension: responses of plasma steroids to angiotensin III].
Itoh N
Nihon Naibunpi Gakkai Zasshi; 1988 May; 64(5):419-38. PubMed ID: 3410145
[TBL] [Abstract][Full Text] [Related]
15. Task3 potassium channel gene invalidation causes low renin and salt-sensitive arterial hypertension.
Penton D; Bandulik S; Schweda F; Haubs S; Tauber P; Reichold M; Cong LD; El Wakil A; Budde T; Lesage F; Lalli E; Zennaro MC; Warth R; Barhanin J
Endocrinology; 2012 Oct; 153(10):4740-8. PubMed ID: 22878402
[TBL] [Abstract][Full Text] [Related]
16. Angiotensin-independent mechanism for aldosterone synthesis during chronic extracellular fluid volume depletion.
Okubo S; Niimura F; Nishimura H; Takemoto F; Fogo A; Matsusaka T; Ichikawa I
J Clin Invest; 1997 Mar; 99(5):855-60. PubMed ID: 9062342
[TBL] [Abstract][Full Text] [Related]
17. Hyperaldosteronism after decreased renal K+ excretion in KCNMB2 knockout mice.
Larsen CK; Jensen IS; Sorensen MV; de Bruijn PI; Bleich M; Praetorius HA; Leipziger J
Am J Physiol Renal Physiol; 2016 May; 310(10):F1035-46. PubMed ID: 26962098
[TBL] [Abstract][Full Text] [Related]
18. [New data on mineralocorticoid hormones. Physiopathological implications].
Corvol P; Jeunemaitre X
Presse Med; 1995 Sep; 24(27):1235-7. PubMed ID: 7501603
[TBL] [Abstract][Full Text] [Related]
19. [Differences of blood plasma renin activity, angiotensin II and aldosterone levels in essential or secondary hypertension].
Song AL; Zeng ZP; Tong AL; Lu L; Chen S; Li M; Fu CL; Wang YH; Sun ML
Zhonghua Nei Ke Za Zhi; 2012 Apr; 51(4):294-8. PubMed ID: 22781950
[TBL] [Abstract][Full Text] [Related]
20. Enhanced Ca
Seidel E; Schewe J; Zhang J; Dinh HA; Forslund SK; Markó L; Hellmig N; Peters J; Muller DN; Lifton RP; Nottoli T; Stölting G; Scholl UI
Proc Natl Acad Sci U S A; 2021 Apr; 118(17):. PubMed ID: 33879608
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]