These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

186 related articles for article (PubMed ID: 22539622)

  • 1. Release of apoptotic cytochrome C from mitochondria by dimethylarsinous acid occurs through interaction with voltage-dependent anion channel in vitro.
    Naranmandura H; Chen X; Tanaka M; Wang WW; Rehman K; Xu S; Chen Z; Chen SQ; Suzuki N
    Toxicol Sci; 2012 Jul; 128(1):137-46. PubMed ID: 22539622
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Mitochondria are the main target organelle for trivalent monomethylarsonous acid (MMA(III))-induced cytotoxicity.
    Naranmandura H; Xu S; Sawata T; Hao WH; Liu H; Bu N; Ogra Y; Lou YJ; Suzuki N
    Chem Res Toxicol; 2011 Jul; 24(7):1094-103. PubMed ID: 21648415
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Cd(2+)-induced cytochrome c release in apoptotic proximal tubule cells: role of mitochondrial permeability transition pore and Ca(2+) uniporter.
    Lee WK; Bork U; Gholamrezaei F; Thévenod F
    Am J Physiol Renal Physiol; 2005 Jan; 288(1):F27-39. PubMed ID: 15339793
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Trivalent methylated arsenical-induced phosphatidylserine exposure and apoptosis in platelets may lead to increased thrombus formation.
    Bae ON; Lim KM; Noh JY; Chung SM; Kim SH; Chung JH
    Toxicol Appl Pharmacol; 2009 Sep; 239(2):144-53. PubMed ID: 19167414
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC.
    Shimizu S; Narita M; Tsujimoto Y
    Nature; 1999 Jun; 399(6735):483-7. PubMed ID: 10365962
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Arsenic stimulates release of cytochrome c from isolated mitochondria via induction of mitochondrial permeability transition.
    Bustamante J; Nutt L; Orrenius S; Gogvadze V
    Toxicol Appl Pharmacol; 2005 Sep; 207(2 Suppl):110-6. PubMed ID: 15979664
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Mouse uterine epithelial apoptosis is associated with expression of mitochondrial voltage-dependent anion channels, release of cytochrome C from mitochondria, and the ratio of Bax to Bcl-2 or Bcl-X.
    Takagi-Morishita Y; Yamada N; Sugihara A; Iwasaki T; Tsujimura T; Terada N
    Biol Reprod; 2003 Apr; 68(4):1178-84. PubMed ID: 12606449
    [TBL] [Abstract][Full Text] [Related]  

  • 8. [Aging-related increase of sensitivity of the mitochondrial permeability transition pore to inductors in the rat heart].
    Sahach VF; Vavilova HL; Strutyns'ka NA; Rudyk OV
    Fiziol Zh (1994); 2004; 50(2):49-63. PubMed ID: 15174206
    [TBL] [Abstract][Full Text] [Related]  

  • 9. In self-defence: hexokinase promotes voltage-dependent anion channel closure and prevents mitochondria-mediated apoptotic cell death.
    Azoulay-Zohar H; Israelson A; Abu-Hamad S; Shoshan-Barmatz V
    Biochem J; 2004 Jan; 377(Pt 2):347-55. PubMed ID: 14561215
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Trivalent methylated arsenic metabolites induce apoptosis in human myeloid leukemic HL-60 cells through generation of reactive oxygen species.
    Rehman K; Fu YJ; Zhang YF; Wang QQ; Wu B; Wu Y; Zhou XY; Sun WH; Sun TF; Naranmandura H
    Metallomics; 2014 Aug; 6(8):1502-12. PubMed ID: 24870979
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Bax interacts with the voltage-dependent anion channel and mediates ethanol-induced apoptosis in rat hepatocytes.
    Adachi M; Higuchi H; Miura S; Azuma T; Inokuchi S; Saito H; Kato S; Ishii H
    Am J Physiol Gastrointest Liver Physiol; 2004 Sep; 287(3):G695-705. PubMed ID: 15044178
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Role of the alkali labile sites, reactive oxygen species and antioxidants in DNA damage induced by methylated trivalent metabolites of inorganic arsenic.
    Soto-Reyes E; Del Razo LM; Valverde M; Rojas E
    Biometals; 2005 Oct; 18(5):493-506. PubMed ID: 16333750
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Forced uptake of trivalent and pentavalent methylated and inorganic arsenic and its cyto-/genotoxicity in fibroblasts and hepatoma cells.
    Dopp E; Hartmann LM; von Recklinghausen U; Florea AM; Rabieh S; Zimmermann U; Shokouhi B; Yadav S; Hirner AV; Rettenmeier AW
    Toxicol Sci; 2005 Sep; 87(1):46-56. PubMed ID: 15947026
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Arsenite and its mono- and dimethylated trivalent metabolites enhance the formation of benzo[a]pyrene diol epoxide-DNA adducts in Xeroderma pigmentosum complementation group A cells.
    Shen S; Lee J; Cullen WR; Le XC; Weinfeld M
    Chem Res Toxicol; 2009 Feb; 22(2):382-90. PubMed ID: 19146383
    [TBL] [Abstract][Full Text] [Related]  

  • 15. The accumulation and toxicity of methylated arsenicals in endothelial cells: important roles of thiol compounds.
    Hirano S; Kobayashi Y; Cui X; Kanno S; Hayakawa T; Shraim A
    Toxicol Appl Pharmacol; 2004 Aug; 198(3):458-67. PubMed ID: 15276427
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Prooxidants open both the mitochondrial permeability transition pore and a low-conductance channel in the inner mitochondrial membrane.
    Kushnareva YE; Sokolove PM
    Arch Biochem Biophys; 2000 Apr; 376(2):377-88. PubMed ID: 10775426
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Uptake of inorganic and organic derivatives of arsenic associated with induced cytotoxic and genotoxic effects in Chinese hamster ovary (CHO) cells.
    Dopp E; Hartmann LM; Florea AM; von Recklinghausen U; Pieper R; Shokouhi B; Rettenmeier AW; Hirner AV; Obe G
    Toxicol Appl Pharmacol; 2004 Dec; 201(2):156-65. PubMed ID: 15541755
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Arsenic speciation in urine from acute promyelocytic leukemia patients undergoing arsenic trioxide treatment.
    Wang Z; Zhou J; Lu X; Gong Z; Le XC
    Chem Res Toxicol; 2004 Jan; 17(1):95-103. PubMed ID: 14727923
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Direct inhibition of the mitochondrial permeability transition pore: a possible mechanism for better neuroprotective effects of allopregnanolone over progesterone.
    Sayeed I; Parvez S; Wali B; Siemen D; Stein DG
    Brain Res; 2009 Mar; 1263():165-73. PubMed ID: 19368823
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Malonate induces cell death via mitochondrial potential collapse and delayed swelling through an ROS-dependent pathway.
    Fernandez-Gomez FJ; Galindo MF; Gómez-Lázaro M; Yuste VJ; Comella JX; Aguirre N; Jordán J
    Br J Pharmacol; 2005 Feb; 144(4):528-37. PubMed ID: 15655518
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 10.