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4. Epigenetic repression of p16(INK4A) by latent Epstein-Barr virus requires the interaction of EBNA3A and EBNA3C with CtBP. Skalska L; White RE; Franz M; Ruhmann M; Allday MJ PLoS Pathog; 2010 Jun; 6(6):e1000951. PubMed ID: 20548956 [TBL] [Abstract][Full Text] [Related]
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6. Two Epstein-Barr virus (EBV) oncoproteins cooperate to repress expression of the proapoptotic tumour-suppressor Bim: clues to the pathogenesis of Burkitt's lymphoma. Anderton E; Yee J; Smith P; Crook T; White RE; Allday MJ Oncogene; 2008 Jan; 27(4):421-33. PubMed ID: 17653091 [TBL] [Abstract][Full Text] [Related]
7. Epigenetic silencing of Bim transcription by Spi-1/PU.1 promotes apoptosis resistance in leukaemia. Ridinger-Saison M; Evanno E; Gallais I; Rimmelé P; Selimoglu-Buet D; Sapharikas E; Moreau-Gachelin F; Guillouf C Cell Death Differ; 2013 Sep; 20(9):1268-78. PubMed ID: 23852375 [TBL] [Abstract][Full Text] [Related]
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10. EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency. Styles CT; Bazot Q; Parker GA; White RE; Paschos K; Allday MJ PLoS Biol; 2017 Aug; 15(8):e2001992. PubMed ID: 28771465 [TBL] [Abstract][Full Text] [Related]
11. Epstein-Barr virus nuclear antigen 3C recruits histone deacetylase activity and associates with the corepressors mSin3A and NCoR in human B-cell lines. Knight JS; Lan K; Subramanian C; Robertson ES J Virol; 2003 Apr; 77(7):4261-72. PubMed ID: 12634383 [TBL] [Abstract][Full Text] [Related]
12. Epstein-Barr Virus Nuclear Antigen 3 (EBNA3) Proteins Regulate EBNA2 Binding to Distinct RBPJ Genomic Sites. Wang A; Welch R; Zhao B; Ta T; Keleş S; Johannsen E J Virol; 2015 Dec; 90(6):2906-19. PubMed ID: 26719268 [TBL] [Abstract][Full Text] [Related]
13. EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells. Kalchschmidt JS; Gillman AC; Paschos K; Bazot Q; Kempkes B; Allday MJ PLoS Pathog; 2016 Jan; 12(1):e1005383. PubMed ID: 26751214 [TBL] [Abstract][Full Text] [Related]
14. Epstein-Barr virus nuclear antigen 3C interacts with histone deacetylase to repress transcription. Radkov SA; Touitou R; Brehm A; Rowe M; West M; Kouzarides T; Allday MJ J Virol; 1999 Jul; 73(7):5688-97. PubMed ID: 10364319 [TBL] [Abstract][Full Text] [Related]
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16. Epstein-Barr virus nuclear antigen 3C is a powerful repressor of transcription when tethered to DNA. Bain M; Watson RJ; Farrell PJ; Allday MJ J Virol; 1996 Apr; 70(4):2481-9. PubMed ID: 8642676 [TBL] [Abstract][Full Text] [Related]
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18. MYC activation and BCL2L11 silencing by a tumour virus through the large-scale reconfiguration of enhancer-promoter hubs. Wood CD; Veenstra H; Khasnis S; Gunnell A; Webb HM; Shannon-Lowe C; Andrews S; Osborne CS; West MJ Elife; 2016 Aug; 5():. PubMed ID: 27490482 [TBL] [Abstract][Full Text] [Related]
19. Epstein-Barr virus EBNA3A and EBNA3C proteins both repress RBP-J kappa-EBNA2-activated transcription by inhibiting the binding of RBP-J kappa to DNA. Waltzer L; Perricaudet M; Sergeant A; Manet E J Virol; 1996 Sep; 70(9):5909-15. PubMed ID: 8709211 [TBL] [Abstract][Full Text] [Related]
20. Transcriptional repression by the Epstein-Barr virus EBNA3A protein tethered to DNA does not require RBP-Jkappa. Bourillot PY; Waltzer L; Sergeant A; Manet E J Gen Virol; 1998 Feb; 79 ( Pt 2)():363-70. PubMed ID: 9472621 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]