314 related articles for article (PubMed ID: 22615584)
1. Deletion of PTH rescues skeletal abnormalities and high osteopontin levels in Klotho-/- mice.
Yuan Q; Sato T; Densmore M; Saito H; Schüler C; Erben RG; Lanske B
PLoS Genet; 2012; 8(5):e1002726. PubMed ID: 22615584
[TBL] [Abstract][Full Text] [Related]
2. Klotho ablation converts the biochemical and skeletal alterations in FGF23 (R176Q) transgenic mice to a Klotho-deficient phenotype.
Bai X; Dinghong Q; Miao D; Goltzman D; Karaplis AC
Am J Physiol Endocrinol Metab; 2009 Jan; 296(1):E79-88. PubMed ID: 18984852
[TBL] [Abstract][Full Text] [Related]
3. Increased osteopontin contributes to inhibition of bone mineralization in FGF23-deficient mice.
Yuan Q; Jiang Y; Zhao X; Sato T; Densmore M; Schüler C; Erben RG; McKee MD; Lanske B
J Bone Miner Res; 2014 Mar; 29(3):693-704. PubMed ID: 24038141
[TBL] [Abstract][Full Text] [Related]
4. Klotho expression in long bones regulates FGF23 production during renal failure.
Kaludjerovic J; Komaba H; Sato T; Erben RG; Baron R; Olauson H; Larsson TE; Lanske B
FASEB J; 2017 May; 31(5):2050-2064. PubMed ID: 28183805
[TBL] [Abstract][Full Text] [Related]
5. In vivo evidence for an interplay of FGF23/Klotho/PTH axis on the phosphate handling in renal proximal tubules.
Ide N; Ye R; Courbebaisse M; Olauson H; Densmore MJ; Larsson TE; Hanai JI; Lanske B
Am J Physiol Renal Physiol; 2018 Nov; 315(5):F1261-F1270. PubMed ID: 29993278
[TBL] [Abstract][Full Text] [Related]
6. FGF23 Regulates Bone Mineralization in a 1,25(OH)2 D3 and Klotho-Independent Manner.
Murali SK; Roschger P; Zeitz U; Klaushofer K; Andrukhova O; Erben RG
J Bone Miner Res; 2016 Jan; 31(1):129-42. PubMed ID: 26235988
[TBL] [Abstract][Full Text] [Related]
7. PTH ablation ameliorates the anomalies of Fgf23-deficient mice by suppressing the elevated vitamin D and calcium levels.
Yuan Q; Sitara D; Sato T; Densmore M; Saito H; Schüler C; Erben RG; Lanske B
Endocrinology; 2011 Nov; 152(11):4053-61. PubMed ID: 21896668
[TBL] [Abstract][Full Text] [Related]
8. Soluble Klotho causes hypomineralization in Klotho-deficient mice.
Minamizaki T; Konishi Y; Sakurai K; Yoshioka H; Aubin JE; Kozai K; Yoshiko Y
J Endocrinol; 2018 Jun; 237(3):285-300. PubMed ID: 29632215
[TBL] [Abstract][Full Text] [Related]
9. Parathyroid-specific deletion of Klotho unravels a novel calcineurin-dependent FGF23 signaling pathway that regulates PTH secretion.
Olauson H; Lindberg K; Amin R; Sato T; Jia T; Goetz R; Mohammadi M; Andersson G; Lanske B; Larsson TE
PLoS Genet; 2013; 9(12):e1003975. PubMed ID: 24348262
[TBL] [Abstract][Full Text] [Related]
10. FGF-23/Klotho signaling is not essential for the phosphaturic and anabolic functions of PTH.
Yuan Q; Sato T; Densmore M; Saito H; Schüler C; Erben RG; Lanske B
J Bone Miner Res; 2011 Sep; 26(9):2026-35. PubMed ID: 21590742
[TBL] [Abstract][Full Text] [Related]
11. Klotho lacks a vitamin D independent physiological role in glucose homeostasis, bone turnover, and steady-state PTH secretion in vivo.
Anour R; Andrukhova O; Ritter E; Zeitz U; Erben RG
PLoS One; 2012; 7(2):e31376. PubMed ID: 22319626
[TBL] [Abstract][Full Text] [Related]
12. Fgf23 and parathyroid hormone signaling interact in kidney and bone.
Andrukhova O; Streicher C; Zeitz U; Erben RG
Mol Cell Endocrinol; 2016 Nov; 436():224-39. PubMed ID: 27498418
[TBL] [Abstract][Full Text] [Related]
13. Targeted deletion of Klotho in kidney distal tubule disrupts mineral metabolism.
Olauson H; Lindberg K; Amin R; Jia T; Wernerson A; Andersson G; Larsson TE
J Am Soc Nephrol; 2012 Oct; 23(10):1641-51. PubMed ID: 22878961
[TBL] [Abstract][Full Text] [Related]
14. Parathyroid hormone 1 receptor is essential to induce FGF23 production and maintain systemic mineral ion homeostasis.
Fan Y; Bi R; Densmore MJ; Sato T; Kobayashi T; Yuan Q; Zhou X; Erben RG; Lanske B
FASEB J; 2016 Jan; 30(1):428-40. PubMed ID: 26428657
[TBL] [Abstract][Full Text] [Related]
15. [Discovery of alpha-Klotho and FGF23 unveiled new insight into calcium and phosphate homeostasis].
Nabeshima Y
Clin Calcium; 2008 Jul; 18(7):923-34. PubMed ID: 18591743
[TBL] [Abstract][Full Text] [Related]
16. Bone mineralization is regulated by signaling cross talk between molecular factors of local and systemic origin: the role of fibroblast growth factor 23.
Sapir-Koren R; Livshits G
Biofactors; 2014; 40(6):555-68. PubMed ID: 25352227
[TBL] [Abstract][Full Text] [Related]
17. The parathyroid is a target organ for FGF23 in rats.
Ben-Dov IZ; Galitzer H; Lavi-Moshayoff V; Goetz R; Kuro-o M; Mohammadi M; Sirkis R; Naveh-Many T; Silver J
J Clin Invest; 2007 Dec; 117(12):4003-8. PubMed ID: 17992255
[TBL] [Abstract][Full Text] [Related]
18. Upregulation of calcitriol during pregnancy and skeletal recovery after lactation do not require parathyroid hormone.
Kirby BJ; Ma Y; Martin HM; Buckle Favaro KL; Karaplis AC; Kovacs CS
J Bone Miner Res; 2013 Sep; 28(9):1987-2000. PubMed ID: 23505097
[TBL] [Abstract][Full Text] [Related]
19. Osteo-renal cross-talk and phosphate metabolism by the FGF23-Klotho system.
Ohnishi M; Razzaque MS
Contrib Nephrol; 2013; 180():1-13. PubMed ID: 23652546
[TBL] [Abstract][Full Text] [Related]
20. Transgenic mice expressing fibroblast growth factor 23 under the control of the alpha1(I) collagen promoter exhibit growth retardation, osteomalacia, and disturbed phosphate homeostasis.
Larsson T; Marsell R; Schipani E; Ohlsson C; Ljunggren O; Tenenhouse HS; Jüppner H; Jonsson KB
Endocrinology; 2004 Jul; 145(7):3087-94. PubMed ID: 14988389
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
