BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

223 related articles for article (PubMed ID: 22789906)

  • 1. Brain connexins in demyelinating diseases: therapeutic potential of glial targets.
    Cotrina ML; Nedergaard M
    Brain Res; 2012 Dec; 1487():61-8. PubMed ID: 22789906
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Gap junction communication in myelinating glia.
    Nualart-Marti A; Solsona C; Fields RD
    Biochim Biophys Acta; 2013 Jan; 1828(1):69-78. PubMed ID: 22326946
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Connexins in oligodendrocytes and astrocytes: Possible factors for demyelination in multiple sclerosis.
    Xia CY; Xu JK; Pan CH; Lian WW; Yan Y; Ma BZ; He J; Zhang WK
    Neurochem Int; 2020 Jun; 136():104731. PubMed ID: 32201280
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Curious and contradictory roles of glial connexins and pannexins in epilepsy.
    Carlen PL
    Brain Res; 2012 Dec; 1487():54-60. PubMed ID: 22796594
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Connexin-based intercellular communication and astrocyte heterogeneity.
    Theis M; Giaume C
    Brain Res; 2012 Dec; 1487():88-98. PubMed ID: 22789907
    [TBL] [Abstract][Full Text] [Related]  

  • 6. [Connexin astrocytopathy and novel therapeutic strategy targeting connexin hemichannels in demyelinating disease].
    Masaki K
    Rinsho Shinkeigaku; 2012; 52(11):1354-6. PubMed ID: 23196616
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Severe Convulsions and Dysmyelination in Both Jimpy and Cx32/47
    Chaban YHG; Chen Y; Hertz E; Hertz L
    Neurochem Res; 2017 Jun; 42(6):1747-1766. PubMed ID: 28214987
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Connexin32 mutations cause loss of function in Schwann cells and oligodendrocytes leading to PNS and CNS myelination defects.
    Sargiannidou I; Vavlitou N; Aristodemou S; Hadjisavvas A; Kyriacou K; Scherer SS; Kleopa KA
    J Neurosci; 2009 Apr; 29(15):4736-49. PubMed ID: 19369543
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Connexins are critical for normal myelination in the CNS.
    Menichella DM; Goodenough DA; Sirkowski E; Scherer SS; Paul DL
    J Neurosci; 2003 Jul; 23(13):5963-73. PubMed ID: 12843301
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Connexins and gap junctions of astrocytes and oligodendrocytes in the CNS.
    Nagy JI; Rash JE
    Brain Res Brain Res Rev; 2000 Apr; 32(1):29-44. PubMed ID: 10751655
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Diseases of connexins expressed in myelinating glia.
    Abrams CK
    Neurosci Lett; 2019 Mar; 695():91-99. PubMed ID: 28545922
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Connexins in neuromyelitis optica: a link between astrocytopathy and demyelination.
    Richard C; Ruiz A; Cavagna S; Bigotte M; Vukusic S; Masaki K; Suenaga T; Kira JI; Giraudon P; Marignier R
    Brain; 2020 Sep; 143(9):2721-2732. PubMed ID: 32889550
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Molecular mechanisms of gap junction mutations in myelinating cells.
    Sargiannidou I; Markoullis K; Kleopa KA
    Histol Histopathol; 2010 Sep; 25(9):1191-206. PubMed ID: 20607661
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Oligodendrocytes are damaged by neuromyelitis optica immunoglobulin G via astrocyte injury.
    Marignier R; Nicolle A; Watrin C; Touret M; Cavagna S; Varrin-Doyer M; Cavillon G; Rogemond V; Confavreux C; Honnorat J; Giraudon P
    Brain; 2010 Sep; 133(9):2578-91. PubMed ID: 20688809
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Nitric oxide as a potential pathological mechanism in demyelination: its differential effects on primary glial cells in vitro.
    Mitrovic B; Ignarro LJ; Montestruque S; Smoll A; Merrill JE
    Neuroscience; 1994 Aug; 61(3):575-85. PubMed ID: 7969931
    [TBL] [Abstract][Full Text] [Related]  

  • 16. The relationship between type-1 astrocytes, Schwann cells and oligodendrocytes following transplantation of glial cell cultures into demyelinating lesions in the adult rat spinal cord.
    Blakemore WF; Crang AJ
    J Neurocytol; 1989 Aug; 18(4):519-28. PubMed ID: 2809635
    [TBL] [Abstract][Full Text] [Related]  

  • 17. The response of oligodendrocytes to chemical injury.
    Blakemore WF
    Acta Neurol Scand Suppl; 1984; 100():33-8. PubMed ID: 6385605
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Connexin32-null mice develop demyelinating peripheral neuropathy.
    Scherer SS; Xu YT; Nelles E; Fischbeck K; Willecke K; Bone LJ
    Glia; 1998 Sep; 24(1):8-20. PubMed ID: 9700485
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Deletion of astrocyte connexins 43 and 30 leads to a dysmyelinating phenotype and hippocampal CA1 vacuolation.
    Lutz SE; Zhao Y; Gulinello M; Lee SC; Raine CS; Brosnan CF
    J Neurosci; 2009 Jun; 29(24):7743-52. PubMed ID: 19535586
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Oligodendrocytes in mouse corpus callosum are coupled via gap junction channels formed by connexin47 and connexin32.
    Maglione M; Tress O; Haas B; Karram K; Trotter J; Willecke K; Kettenmann H
    Glia; 2010 Jul; 58(9):1104-17. PubMed ID: 20468052
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 12.