These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

773 related articles for article (PubMed ID: 22803956)

  • 1. Relationship between helix stability and binding affinities: molecular dynamics simulations of Bfl-1/A1-binding pro-apoptotic BH3 peptide helices in explicit solvent.
    Modi V; Lama D; Sankararamakrishnan R
    J Biomol Struct Dyn; 2013; 31(1):65-77. PubMed ID: 22803956
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Anti-apoptotic Bcl-XL protein in complex with BH3 peptides of pro-apoptotic Bak, Bad, and Bim proteins: comparative molecular dynamics simulations.
    Lama D; Sankararamakrishnan R
    Proteins; 2008 Nov; 73(2):492-514. PubMed ID: 18452209
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Structural biology of the Bcl-2 family of proteins.
    Petros AM; Olejniczak ET; Fesik SW
    Biochim Biophys Acta; 2004 Mar; 1644(2-3):83-94. PubMed ID: 14996493
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Behavior of solvent-exposed hydrophobic groove in the anti-apoptotic Bcl-XL protein: clues for its ability to bind diverse BH3 ligands from MD simulations.
    Lama D; Modi V; Sankararamakrishnan R
    PLoS One; 2013; 8(2):e54397. PubMed ID: 23468841
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Synthesis and helical structure of lactam bridged BH3 peptides derived from pro-apoptotic Bcl-2 family proteins.
    Yang B; Liu D; Huang Z
    Bioorg Med Chem Lett; 2004 Mar; 14(6):1403-6. PubMed ID: 15006371
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Bcl-2 family member Bfl-1/A1 sequesters truncated bid to inhibit is collaboration with pro-apoptotic Bak or Bax.
    Werner AB; de Vries E; Tait SW; Bontjer I; Borst J
    J Biol Chem; 2002 Jun; 277(25):22781-8. PubMed ID: 11929871
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Structure of the BH3 domains from the p53-inducible BH3-only proteins Noxa and Puma in complex with Mcl-1.
    Day CL; Smits C; Fan FC; Lee EF; Fairlie WD; Hinds MG
    J Mol Biol; 2008 Jul; 380(5):958-71. PubMed ID: 18589438
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Membrane-insertion fragments of Bcl-xL, Bax, and Bid.
    García-Sáez AJ; Mingarro I; Pérez-Payá E; Salgado J
    Biochemistry; 2004 Aug; 43(34):10930-43. PubMed ID: 15323553
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Molecular dynamics simulation and automated docking of the pro-apoptotic Bax protein and its complex with a peptide designed from the Bax-binding domain of anti-apoptotic Ku70.
    Mancinelli F; Caraglia M; Budillon A; Abbruzzese A; Bismuto E
    J Cell Biochem; 2006 Sep; 99(1):305-18. PubMed ID: 16619258
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Characterization of the anti-apoptotic mechanism of Bcl-B.
    Zhai D; Ke N; Zhang H; Ladror U; Joseph M; Eichinger A; Godzik A; Ng SC; Reed JC
    Biochem J; 2003 Nov; 376(Pt 1):229-36. PubMed ID: 12921534
    [TBL] [Abstract][Full Text] [Related]  

  • 11. The structural basis of Bcl-2 mediated cell death regulation in hydra.
    Banjara S; D Sa J; Hinds MG; Kvansakul M
    Biochem J; 2020 Sep; 477(17):3287-3297. PubMed ID: 32776134
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Terphenyl-Based Bak BH3 alpha-helical proteomimetics as low-molecular-weight antagonists of Bcl-xL.
    Yin H; Lee GI; Sedey KA; Kutzki O; Park HS; Orner BP; Ernst JT; Wang HG; Sebti SM; Hamilton AD
    J Am Chem Soc; 2005 Jul; 127(29):10191-6. PubMed ID: 16028929
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Minimal BH3 peptides promote cell death by antagonizing anti-apoptotic proteins.
    Moreau C; Cartron PF; Hunt A; Meflah K; Green DR; Evan G; Vallette FM; Juin P
    J Biol Chem; 2003 May; 278(21):19426-35. PubMed ID: 12642586
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Functional dissection of Bfl-1, a Bcl-2 homolog: anti-apoptosis, oncogene-cooperation and cell proliferation activities.
    D'Sa-Eipper C; Chinnadurai G
    Oncogene; 1998 Jun; 16(24):3105-14. PubMed ID: 9671389
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Differential targeting of prosurvival Bcl-2 proteins by their BH3-only ligands allows complementary apoptotic function.
    Chen L; Willis SN; Wei A; Smith BJ; Fletcher JI; Hinds MG; Colman PM; Day CL; Adams JM; Huang DC
    Mol Cell; 2005 Feb; 17(3):393-403. PubMed ID: 15694340
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Peptides derived from apoptotic Bax and Bid reproduce the poration activity of the parent full-length proteins.
    García-Sáez AJ; Coraiola M; Dalla Serra M; Mingarro I; Menestrina G; Salgado J
    Biophys J; 2005 Jun; 88(6):3976-90. PubMed ID: 15778450
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Structural basis of the specificity and interaction mechanism of Bmf binding to pro-survival Bcl-2 family proteins.
    Wang H; Guo M; Wei H; Chen Y
    Comput Struct Biotechnol J; 2023; 21():3760-3767. PubMed ID: 37560128
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Sequence and helicity requirements for the proapoptotic activity of Bax BH3 peptides.
    Shangary S; Oliver CL; Tillman TS; Cascio M; Johnson DE
    Mol Cancer Ther; 2004 Nov; 3(11):1343-54. PubMed ID: 15542773
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Vaccinia virus anti-apoptotic F1L is a novel Bcl-2-like domain-swapped dimer that binds a highly selective subset of BH3-containing death ligands.
    Kvansakul M; Yang H; Fairlie WD; Czabotar PE; Fischer SF; Perugini MA; Huang DC; Colman PM
    Cell Death Differ; 2008 Oct; 15(10):1564-71. PubMed ID: 18551131
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Peptides derived from BH3 domains of Bcl-2 family members: a comparative analysis of inhibition of Bcl-2, Bcl-x(L) and Bax oligomerization, induction of cytochrome c release, and activation of cell death.
    Shangary S; Johnson DE
    Biochemistry; 2002 Jul; 41(30):9485-95. PubMed ID: 12135371
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 39.