BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

219 related articles for article (PubMed ID: 22884843)

  • 1. Transgenic overexpression of matrix metalloproteinase-9 in macrophages attenuates the inflammatory response and improves left ventricular function post-myocardial infarction.
    Zamilpa R; Ibarra J; de Castro Brás LE; Ramirez TA; Nguyen N; Halade GV; Zhang J; Dai Q; Dayah T; Chiao YA; Lowell W; Ahuja SS; D'Armiento J; Jin YF; Lindsey ML
    J Mol Cell Cardiol; 2012 Nov; 53(5):599-608. PubMed ID: 22884843
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Macrophage overexpression of matrix metalloproteinase-9 in aged mice improves diastolic physiology and cardiac wound healing after myocardial infarction.
    Meschiari CA; Jung M; Iyer RP; Yabluchanskiy A; Toba H; Garrett MR; Lindsey ML
    Am J Physiol Heart Circ Physiol; 2018 Feb; 314(2):H224-H235. PubMed ID: 29030341
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Matrix metalloproteinase-28 deletion exacerbates cardiac dysfunction and rupture after myocardial infarction in mice by inhibiting M2 macrophage activation.
    Ma Y; Halade GV; Zhang J; Ramirez TA; Levin D; Voorhees A; Jin YF; Han HC; Manicone AM; Lindsey ML
    Circ Res; 2013 Feb; 112(4):675-88. PubMed ID: 23261783
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Deficiency of ataxia telangiectasia mutated kinase delays inflammatory response in the heart following myocardial infarction.
    Daniel LL; Daniels CR; Harirforoosh S; Foster CR; Singh M; Singh K
    J Am Heart Assoc; 2014 Dec; 3(6):e001286. PubMed ID: 25520329
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Early matrix metalloproteinase-9 inhibition post-myocardial infarction worsens cardiac dysfunction by delaying inflammation resolution.
    Iyer RP; de Castro Brás LE; Patterson NL; Bhowmick M; Flynn ER; Asher M; Cannon PL; Deleon-Pennell KY; Fields GB; Lindsey ML
    J Mol Cell Cardiol; 2016 Nov; 100():109-117. PubMed ID: 27746126
    [TBL] [Abstract][Full Text] [Related]  

  • 6. SPARC mediates early extracellular matrix remodeling following myocardial infarction.
    McCurdy SM; Dai Q; Zhang J; Zamilpa R; Ramirez TA; Dayah T; Nguyen N; Jin YF; Bradshaw AD; Lindsey ML
    Am J Physiol Heart Circ Physiol; 2011 Aug; 301(2):H497-505. PubMed ID: 21602472
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Aliskiren and valsartan mediate left ventricular remodeling post-myocardial infarction in mice through MMP-9 effects.
    Ramirez TA; Iyer RP; Ghasemi O; Lopez EF; Levin DB; Zhang J; Zamilpa R; Chou YM; Jin YF; Lindsey ML
    J Mol Cell Cardiol; 2014 Jul; 72():326-35. PubMed ID: 24768766
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Myocardial injury after ischemia-reperfusion in mice deficient in Akt2 is associated with increased cardiac macrophage density.
    Li X; Mikhalkova D; Gao E; Zhang J; Myers V; Zincarelli C; Lei Y; Song J; Koch WJ; Peppel K; Cheung JY; Feldman AM; Chan TO
    Am J Physiol Heart Circ Physiol; 2011 Nov; 301(5):H1932-40. PubMed ID: 21890689
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Exogenous CXCL4 infusion inhibits macrophage phagocytosis by limiting CD36 signalling to enhance post-myocardial infarction cardiac dilation and mortality.
    Lindsey ML; Jung M; Yabluchanskiy A; Cannon PL; Iyer RP; Flynn ER; DeLeon-Pennell KY; Valerio FM; Harrison CL; Ripplinger CM; Hall ME; Ma Y
    Cardiovasc Res; 2019 Feb; 115(2):395-408. PubMed ID: 30169632
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Calpastatin overexpression impairs postinfarct scar healing in mice by compromising reparative immune cell recruitment and activation.
    Wan F; Letavernier E; Le Saux CJ; Houssaini A; Abid S; Czibik G; Sawaki D; Marcos E; Dubois-Rande JL; Baud L; Adnot S; Derumeaux G; Gellen B
    Am J Physiol Heart Circ Physiol; 2015 Dec; 309(11):H1883-93. PubMed ID: 26453333
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Mapping macrophage polarization over the myocardial infarction time continuum.
    Mouton AJ; DeLeon-Pennell KY; Rivera Gonzalez OJ; Flynn ER; Freeman TC; Saucerman JJ; Garrett MR; Ma Y; Harmancey R; Lindsey ML
    Basic Res Cardiol; 2018 Jun; 113(4):26. PubMed ID: 29868933
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Protective Effects of Activated Myofibroblasts in the Pressure-Overloaded Myocardium Are Mediated Through Smad-Dependent Activation of a Matrix-Preserving Program.
    Russo I; Cavalera M; Huang S; Su Y; Hanna A; Chen B; Shinde AV; Conway SJ; Graff J; Frangogiannis NG
    Circ Res; 2019 Apr; 124(8):1214-1227. PubMed ID: 30686120
    [TBL] [Abstract][Full Text] [Related]  

  • 13. P. gingivalis lipopolysaccharide intensifies inflammation post-myocardial infarction through matrix metalloproteinase-9.
    DeLeon-Pennell KY; de Castro Brás LE; Iyer RP; Bratton DR; Jin YF; Ripplinger CM; Lindsey ML
    J Mol Cell Cardiol; 2014 Nov; 76():218-26. PubMed ID: 25240641
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Myocardial Infarction Superimposed on Aging: MMP-9 Deletion Promotes M2 Macrophage Polarization.
    Yabluchanskiy A; Ma Y; DeLeon-Pennell KY; Altara R; Halade GV; Voorhees AP; Nguyen NT; Jin YF; Winniford MD; Hall ME; Han HC; Lindsey ML
    J Gerontol A Biol Sci Med Sci; 2016 Apr; 71(4):475-83. PubMed ID: 25878031
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Selective spatiotemporal induction of matrix metalloproteinase-2 and matrix metalloproteinase-9 transcription after myocardial infarction.
    Mukherjee R; Mingoia JT; Bruce JA; Austin JS; Stroud RE; Escobar GP; McClister DM; Allen CM; Alfonso-Jaume MA; Fini ME; Lovett DH; Spinale FG
    Am J Physiol Heart Circ Physiol; 2006 Nov; 291(5):H2216-28. PubMed ID: 16766634
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Targeted deletion of class A macrophage scavenger receptor increases the risk of cardiac rupture after experimental myocardial infarction.
    Tsujita K; Kaikita K; Hayasaki T; Honda T; Kobayashi H; Sakashita N; Suzuki H; Kodama T; Ogawa H; Takeya M
    Circulation; 2007 Apr; 115(14):1904-11. PubMed ID: 17389263
    [TBL] [Abstract][Full Text] [Related]  

  • 17. TIMP2 deficiency accelerates adverse post-myocardial infarction remodeling because of enhanced MT1-MMP activity despite lack of MMP2 activation.
    Kandalam V; Basu R; Abraham T; Wang X; Soloway PD; Jaworski DM; Oudit GY; Kassiri Z
    Circ Res; 2010 Mar; 106(4):796-808. PubMed ID: 20056917
    [TBL] [Abstract][Full Text] [Related]  

  • 18. The receptor for activated complement factor 5 (C5aR) conveys myocardial ischemic damage by mediating neutrophil transmigration.
    Mueller M; Herzog C; Larmann J; Schmitz M; Hilfiker-Kleiner D; Gessner JE; Theilmeier G
    Immunobiology; 2013 Sep; 218(9):1131-8. PubMed ID: 23642836
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Toll-like receptor 7 deficiency promotes survival and reduces adverse left ventricular remodelling after myocardial infarction.
    de Kleijn DPV; Chong SY; Wang X; Yatim SMJM; Fairhurst AM; Vernooij F; Zharkova O; Chan MY; Foo RSY; Timmers L; Lam CSP; Wang JW
    Cardiovasc Res; 2019 Oct; 115(12):1791-1803. PubMed ID: 30830156
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Transgenic overexpression of macrophage matrix metalloproteinase-9 exacerbates age-related cardiac hypertrophy, vessel rarefaction, inflammation, and fibrosis.
    Toba H; Cannon PL; Yabluchanskiy A; Iyer RP; D'Armiento J; Lindsey ML
    Am J Physiol Heart Circ Physiol; 2017 Mar; 312(3):H375-H383. PubMed ID: 28011588
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 11.