117 related articles for article (PubMed ID: 22899723)
1. Unrepairable DNA double-strand breaks that are generated by ionising radiation determine the fate of normal human cells.
Noda A; Hirai Y; Hamasaki K; Mitani H; Nakamura N; Kodama Y
J Cell Sci; 2012 Nov; 125(Pt 22):5280-7. PubMed ID: 22899723
[TBL] [Abstract][Full Text] [Related]
2. Regulation of ATM in DNA double strand break repair accounts for the radiosensitivity in human cells exposed to high linear energy transfer ionizing radiation.
Xue L; Yu D; Furusawa Y; Okayasu R; Tong J; Cao J; Fan S
Mutat Res; 2009 Nov; 670(1-2):15-23. PubMed ID: 19583974
[TBL] [Abstract][Full Text] [Related]
3. Modulation of the DNA-damage response to HZE particles by shielding.
Mukherjee B; Camacho CV; Tomimatsu N; Miller J; Burma S
DNA Repair (Amst); 2008 Oct; 7(10):1717-30. PubMed ID: 18672098
[TBL] [Abstract][Full Text] [Related]
4. Evidence of an adaptive response targeting DNA nonhomologous end joining and its transmission to bystander cells.
Klammer H; Kadhim M; Iliakis G
Cancer Res; 2010 Nov; 70(21):8498-506. PubMed ID: 20861183
[TBL] [Abstract][Full Text] [Related]
5. Inter-individual variation in DNA double-strand break repair in human fibroblasts before and after exposure to low doses of ionizing radiation.
Wilson PF; Nham PB; Urbin SS; Hinz JM; Jones IM; Thompson LH
Mutat Res; 2010 Jan; 683(1-2):91-7. PubMed ID: 19896956
[TBL] [Abstract][Full Text] [Related]
6. DNA double-strand breaks induced by very low X-ray doses are largely due to bystander effects.
Ojima M; Ban N; Kai M
Radiat Res; 2008 Sep; 170(3):365-71. PubMed ID: 18763860
[TBL] [Abstract][Full Text] [Related]
7. Persistence of unrepaired DNA double strand breaks caused by inhibition of ATM does not lead to radio-sensitisation in the absence of NF-κB activation.
Veuger SJ; Durkacz BW
DNA Repair (Amst); 2011 Feb; 10(2):235-44. PubMed ID: 21144805
[TBL] [Abstract][Full Text] [Related]
8. The impact of heterochromatin on DSB repair.
Goodarzi AA; Noon AT; Jeggo PA
Biochem Soc Trans; 2009 Jun; 37(Pt 3):569-76. PubMed ID: 19442252
[TBL] [Abstract][Full Text] [Related]
9. Identification of phosphorylation sites on transcription factor Sp1 in response to DNA damage and its accumulation at damaged sites.
Iwahori S; Yasui Y; Kudoh A; Sato Y; Nakayama S; Murata T; Isomura H; Tsurumi T
Cell Signal; 2008 Oct; 20(10):1795-803. PubMed ID: 18619531
[TBL] [Abstract][Full Text] [Related]
10. In vivo γ-irradiation low dose threshold for suppression of DNA double strand breaks below the spontaneous level in mouse blood and spleen cells.
Osipov AN; Buleeva G; Arkhangelskaya E; Klokov D
Mutat Res; 2013 Aug; 756(1-2):141-5. PubMed ID: 23664857
[TBL] [Abstract][Full Text] [Related]
11. Radiation-induced unrepairable DSBs: their role in the late effects of radiation and possible applications to biodosimetry.
Noda A
J Radiat Res; 2018 Apr; 59(suppl_2):ii114-ii120. PubMed ID: 29281054
[TBL] [Abstract][Full Text] [Related]
12. Development of a high-content high-throughput screening assay for the discovery of ATM signaling inhibitors.
Bardelle C; Boros J
J Biomol Screen; 2012 Aug; 17(7):912-20. PubMed ID: 22653913
[TBL] [Abstract][Full Text] [Related]
13. Accumulation of DNA double-strand breaks in normal tissues after fractionated irradiation.
Rübe CE; Fricke A; Wendorf J; Stützel A; Kühne M; Ong MF; Lipp P; Rübe C
Int J Radiat Oncol Biol Phys; 2010 Mar; 76(4):1206-13. PubMed ID: 20206019
[TBL] [Abstract][Full Text] [Related]
14. Regulation of the cellular DNA double-strand break response.
Cann KL; Hicks GG
Biochem Cell Biol; 2007 Dec; 85(6):663-74. PubMed ID: 18059525
[TBL] [Abstract][Full Text] [Related]
15. A DNA double-strand break defective fibroblast cell line (180BR) derived from a radiosensitive patient represents a new mutant phenotype.
Badie C; Goodhardt M; Waugh A; Doyen N; Foray N; Calsou P; Singleton B; Gell D; Salles B; Jeggo P; Arlett CF; Malaise EP
Cancer Res; 1997 Oct; 57(20):4600-7. PubMed ID: 9377575
[TBL] [Abstract][Full Text] [Related]
16. Three-dimensional cell growth confers radioresistance by chromatin density modification.
Storch K; Eke I; Borgmann K; Krause M; Richter C; Becker K; Schröck E; Cordes N
Cancer Res; 2010 May; 70(10):3925-34. PubMed ID: 20442295
[TBL] [Abstract][Full Text] [Related]
17. [Cellular repair potential in families of ataxia-telangiectasia patients].
Polubotko EA; Shatrova AN; Pleskach NM; Mikhel'son VM; Spivak IM
Tsitologiia; 2009; 51(12):978-85. PubMed ID: 20141033
[TBL] [Abstract][Full Text] [Related]
18. ATM-deficient human fibroblast cells are resistant to low levels of DNA double-strand break induced apoptosis and subsequently undergo drug-induced premature senescence.
Park J; Jo YH; Cho CH; Choe W; Kang I; Baik HH; Yoon KS
Biochem Biophys Res Commun; 2013 Jan; 430(1):429-35. PubMed ID: 23178571
[TBL] [Abstract][Full Text] [Related]
19. c-Jun-deficient cells undergo premature senescence as a result of spontaneous DNA damage accumulation.
MacLaren A; Black EJ; Clark W; Gillespie DA
Mol Cell Biol; 2004 Oct; 24(20):9006-18. PubMed ID: 15456874
[TBL] [Abstract][Full Text] [Related]
20. Constitutive phosphorylation of ATM in lymphoblastoid cell lines from patients with ICF syndrome without downstream kinase activity.
Goldstine JV; Nahas S; Gamo K; Gartler SM; Hansen RS; Roelfsema JH; Gatti RA; Marahrens Y
DNA Repair (Amst); 2006 Apr; 5(4):432-43. PubMed ID: 16426903
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]