These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
384 related articles for article (PubMed ID: 22971774)
1. The Eyes Absent proteins in development and disease. Tadjuidje E; Hegde RS Cell Mol Life Sci; 2013 Jun; 70(11):1897-913. PubMed ID: 22971774 [TBL] [Abstract][Full Text] [Related]
2. Using Drosophila to decipher how mutations associated with human branchio-oto-renal syndrome and optical defects compromise the protein tyrosine phosphatase and transcriptional functions of eyes absent. Mutsuddi M; Chaffee B; Cassidy J; Silver SJ; Tootle TL; Rebay I Genetics; 2005 Jun; 170(2):687-95. PubMed ID: 15802522 [TBL] [Abstract][Full Text] [Related]
3. Branchio-oto-renal syndrome associated mutations in Eyes Absent 1 result in loss of phosphatase activity. Rayapureddi JP; Hegde RS FEBS Lett; 2006 Jul; 580(16):3853-9. PubMed ID: 16797546 [TBL] [Abstract][Full Text] [Related]
5. Biochemical and functional characterization of six SIX1 Branchio-oto-renal syndrome mutations. Patrick AN; Schiemann BJ; Yang K; Zhao R; Ford HL J Biol Chem; 2009 Jul; 284(31):20781-90. PubMed ID: 19497856 [TBL] [Abstract][Full Text] [Related]
6. Eyes absent proteins: characterization of substrate specificity and phosphatase activity of mutants associated with branchial, otic and renal anomalies. Musharraf A; Markschies N; Teichmann K; Pankratz S; Landgraf K; Englert C; Imhof D Chembiochem; 2008 Sep; 9(14):2285-94. PubMed ID: 18759246 [TBL] [Abstract][Full Text] [Related]
7. Structure-function analyses of the human SIX1-EYA2 complex reveal insights into metastasis and BOR syndrome. Patrick AN; Cabrera JH; Smith AL; Chen XS; Ford HL; Zhao R Nat Struct Mol Biol; 2013 Apr; 20(4):447-53. PubMed ID: 23435380 [TBL] [Abstract][Full Text] [Related]
8. The Eyes Absent family: At the intersection of DNA repair, mitosis, and replication. Nelson CB; Wells JK; Pickett HA DNA Repair (Amst); 2024 Sep; 141():103729. PubMed ID: 39089192 [TBL] [Abstract][Full Text] [Related]
9. Distinct Biochemical Activities of Eyes absent During Drosophila Eye Development. Jin M; Mardon G Sci Rep; 2016 Mar; 6():23228. PubMed ID: 26980695 [TBL] [Abstract][Full Text] [Related]
10. Molecular effects of Eya1 domain mutations causing organ defects in BOR syndrome. Buller C; Xu X; Marquis V; Schwanke R; Xu PX Hum Mol Genet; 2001 Nov; 10(24):2775-81. PubMed ID: 11734542 [TBL] [Abstract][Full Text] [Related]
11. A human homologue of the Drosophila eyes absent gene underlies branchio-oto-renal (BOR) syndrome and identifies a novel gene family. Abdelhak S; Kalatzis V; Heilig R; Compain S; Samson D; Vincent C; Weil D; Cruaud C; Sahly I; Leibovici M; Bitner-Glindzicz M; Francis M; Lacombe D; Vigneron J; Charachon R; Boven K; Bedbeder P; Van Regemorter N; Weissenbach J; Petit C Nat Genet; 1997 Feb; 15(2):157-64. PubMed ID: 9020840 [TBL] [Abstract][Full Text] [Related]
12. All eyes on Eya: A unique transcriptional co-activator and phosphatase in cancer. Hughes CJ; Alderman C; Wolin AR; Fields KM; Zhao R; Ford HL Biochim Biophys Acta Rev Cancer; 2024 May; 1879(3):189098. PubMed ID: 38555001 [TBL] [Abstract][Full Text] [Related]
13. Impaired interactions between mouse Eyal harboring mutations found in patients with branchio-oto-renal syndrome and Six, Dach, and G proteins. Ozaki H; Watanabe Y; Ikeda K; Kawakami K J Hum Genet; 2002; 47(3):107-16. PubMed ID: 11950062 [TBL] [Abstract][Full Text] [Related]
14. The EYA tyrosine phosphatase activity is pro-angiogenic and is inhibited by benzbromarone. Tadjuidje E; Wang TS; Pandey RN; Sumanas S; Lang RA; Hegde RS PLoS One; 2012; 7(4):e34806. PubMed ID: 22545090 [TBL] [Abstract][Full Text] [Related]
15. Branchio-oto-renal syndrome. Kochhar A; Fischer SM; Kimberling WJ; Smith RJ Am J Med Genet A; 2007 Jul; 143A(14):1671-8. PubMed ID: 17238186 [TBL] [Abstract][Full Text] [Related]
16. The C. elegans eyes absent ortholog EYA-1 is required for tissue differentiation and plays partially redundant roles with PAX-6. Furuya M; Qadota H; Chisholm AD; Sugimoto A Dev Biol; 2005 Oct; 286(2):452-63. PubMed ID: 16154558 [TBL] [Abstract][Full Text] [Related]
17. Eya1-deficient mice lack ears and kidneys and show abnormal apoptosis of organ primordia. Xu PX; Adams J; Peters H; Brown MC; Heaney S; Maas R Nat Genet; 1999 Sep; 23(1):113-7. PubMed ID: 10471511 [TBL] [Abstract][Full Text] [Related]
18. The Eya phosphatase: Its unique role in cancer. Zhou H; Zhang L; Vartuli RL; Ford HL; Zhao R Int J Biochem Cell Biol; 2018 Mar; 96():165-170. PubMed ID: 28887153 [TBL] [Abstract][Full Text] [Related]
19. The Eyes Absent proteins in development and in developmental disorders. Soni UK; Roychoudhury K; Hegde RS Biochem Soc Trans; 2021 Jun; 49(3):1397-1408. PubMed ID: 34196366 [TBL] [Abstract][Full Text] [Related]
20. SIX1 mutations cause branchio-oto-renal syndrome by disruption of EYA1-SIX1-DNA complexes. Ruf RG; Xu PX; Silvius D; Otto EA; Beekmann F; Muerb UT; Kumar S; Neuhaus TJ; Kemper MJ; Raymond RM; Brophy PD; Berkman J; Gattas M; Hyland V; Ruf EM; Schwartz C; Chang EH; Smith RJ; Stratakis CA; Weil D; Petit C; Hildebrandt F Proc Natl Acad Sci U S A; 2004 May; 101(21):8090-5. PubMed ID: 15141091 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]