These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
6. TWEAK/Fn14 promotes pro-inflammatory cytokine secretion in hepatic stellate cells via NF-κB/STAT3 pathways. Wang A; Zhang F; Xu H; Xu M; Cao Y; Wang C; Xu Y; Su M; Zhang M; Zhuge Y Mol Immunol; 2017 Jul; 87():67-75. PubMed ID: 28411440 [TBL] [Abstract][Full Text] [Related]
7. Functional expression of TWEAK and the receptor Fn14 in human malignant ovarian tumors: possible implication for ovarian tumor intervention. Gu L; Dai L; Cao C; Zhu J; Ding C; Xu HB; Qiu L; Di W PLoS One; 2013; 8(3):e57436. PubMed ID: 23469193 [TBL] [Abstract][Full Text] [Related]
8. The cytokine tumor necrosis factor-like weak inducer of apoptosis and its receptor fibroblast growth factor-inducible 14 have a neuroprotective effect in the central nervous system. Echeverry R; Wu F; Haile WB; Wu J; Yepes M J Neuroinflammation; 2012 Mar; 9():45. PubMed ID: 22394384 [TBL] [Abstract][Full Text] [Related]
9. Identification of aurintricarboxylic acid as a selective inhibitor of the TWEAK-Fn14 signaling pathway in glioblastoma cells. Roos A; Dhruv HD; Mathews IT; Inge LJ; Tuncali S; Hartman LK; Chow D; Millard N; Yin HH; Kloss J; Loftus JC; Winkles JA; Berens ME; Tran NL Oncotarget; 2017 Feb; 8(7):12234-12246. PubMed ID: 28103571 [TBL] [Abstract][Full Text] [Related]
10. The TWEAK-Fn14 system as a potential drug target. Wajant H Br J Pharmacol; 2013 Oct; 170(4):748-64. PubMed ID: 23957828 [TBL] [Abstract][Full Text] [Related]
11. Tumor necrosis factor-like weak inducer of apoptosis and fibroblast growth factor-inducible 14 mediate cerebral ischemia-induced poly(ADP-ribose) polymerase-1 activation and neuronal death. Haile WB; Echeverry R; Wu F; Guzman J; An J; Wu J; Yepes M Neuroscience; 2010 Dec; 171(4):1256-64. PubMed ID: 20955770 [TBL] [Abstract][Full Text] [Related]
12. Studies of binding of tumor necrosis factor (TNF)-like weak inducer of apoptosis (TWEAK) to fibroblast growth factor inducible 14 (Fn14). Fick A; Lang I; Schäfer V; Seher A; Trebing J; Weisenberger D; Wajant H J Biol Chem; 2012 Jan; 287(1):484-495. PubMed ID: 22081603 [TBL] [Abstract][Full Text] [Related]
13. Interaction of TWEAK with Fn14 leads to the progression of fibrotic liver disease by directly modulating hepatic stellate cell proliferation. Wilhelm A; Shepherd EL; Amatucci A; Munir M; Reynolds G; Humphreys E; Resheq Y; Adams DH; Hübscher S; Burkly LC; Weston CJ; Afford SC J Pathol; 2016 May; 239(1):109-21. PubMed ID: 26924336 [TBL] [Abstract][Full Text] [Related]
15. TWEAK-independent Fn14 self-association and NF-κB activation is mediated by the C-terminal region of the Fn14 cytoplasmic domain. Brown SA; Cheng E; Williams MS; Winkles JA PLoS One; 2013; 8(6):e65248. PubMed ID: 23750247 [TBL] [Abstract][Full Text] [Related]
16. TWEAK mediates signal transduction and differentiation of RAW264.7 cells in the absence of Fn14/TweakR. Evidence for a second TWEAK receptor. Polek TC; Talpaz M; Darnay BG; Spivak-Kroizman T J Biol Chem; 2003 Aug; 278(34):32317-23. PubMed ID: 12794080 [TBL] [Abstract][Full Text] [Related]
17. TWEAK/Fn14 activation induces keratinocyte proliferation under psoriatic inflammation. Cheng H; Xu M; Liu X; Zou X; Zhan N; Xia Y Exp Dermatol; 2016 Jan; 25(1):32-7. PubMed ID: 26264384 [TBL] [Abstract][Full Text] [Related]
18. Tumour necrosis factor-like weak inducer of apoptosis (TWEAK) and its receptor Fn14 during cardiac remodelling in rats. Mustonen E; Säkkinen H; Tokola H; Isopoussu E; Aro J; Leskinen H; Ruskoaho H; Rysä J Acta Physiol (Oxf); 2010 May; 199(1):11-22. PubMed ID: 20082609 [TBL] [Abstract][Full Text] [Related]