281 related articles for article (PubMed ID: 23144634)
1. CtIP is required to initiate replication-dependent interstrand crosslink repair.
Duquette ML; Zhu Q; Taylor ER; Tsay AJ; Shi LZ; Berns MW; McGowan CH
PLoS Genet; 2012; 8(11):e1003050. PubMed ID: 23144634
[TBL] [Abstract][Full Text] [Related]
2. FANCD2 binds CtIP and regulates DNA-end resection during DNA interstrand crosslink repair.
Unno J; Itaya A; Taoka M; Sato K; Tomida J; Sakai W; Sugasawa K; Ishiai M; Ikura T; Isobe T; Kurumizaka H; Takata M
Cell Rep; 2014 May; 7(4):1039-47. PubMed ID: 24794430
[TBL] [Abstract][Full Text] [Related]
3. FANCD2 and CtIP cooperate to repair DNA interstrand crosslinks.
Murina O; von Aesch C; Karakus U; Ferretti LP; Bolck HA; Hänggi K; Sartori AA
Cell Rep; 2014 May; 7(4):1030-8. PubMed ID: 24794434
[TBL] [Abstract][Full Text] [Related]
4. Cooperation of the NEIL3 and Fanconi anemia/BRCA pathways in interstrand crosslink repair.
Li N; Wang J; Wallace SS; Chen J; Zhou J; D'Andrea AD
Nucleic Acids Res; 2020 Apr; 48(6):3014-3028. PubMed ID: 31980815
[TBL] [Abstract][Full Text] [Related]
5. The interaction of CtIP and Nbs1 connects CDK and ATM to regulate HR-mediated double-strand break repair.
Wang H; Shi LZ; Wong CC; Han X; Hwang PY; Truong LN; Zhu Q; Shao Z; Chen DJ; Berns MW; Yates JR; Chen L; Wu X
PLoS Genet; 2013; 9(2):e1003277. PubMed ID: 23468639
[TBL] [Abstract][Full Text] [Related]
6. CtIP-BRCA1 modulates the choice of DNA double-strand-break repair pathway throughout the cell cycle.
Yun MH; Hiom K
Nature; 2009 May; 459(7245):460-3. PubMed ID: 19357644
[TBL] [Abstract][Full Text] [Related]
7. Chromosomal Integrity after UV Irradiation Requires FANCD2-Mediated Repair of Double Strand Breaks.
Federico MB; Vallerga MB; Radl A; Paviolo NS; Bocco JL; Di Giorgio M; Soria G; Gottifredi V
PLoS Genet; 2016 Jan; 12(1):e1005792. PubMed ID: 26765540
[TBL] [Abstract][Full Text] [Related]
8. ATM-dependent MiR-335 targets CtIP and modulates the DNA damage response.
Martin NT; Nakamura K; Davies R; Nahas SA; Brown C; Tunuguntla R; Gatti RA; Hu H
PLoS Genet; 2013 May; 9(5):e1003505. PubMed ID: 23696749
[TBL] [Abstract][Full Text] [Related]
9. CtIP mediates replication fork recovery in a FANCD2-regulated manner.
Yeo JE; Lee EH; Hendrickson EA; Sobeck A
Hum Mol Genet; 2014 Jul; 23(14):3695-705. PubMed ID: 24556218
[TBL] [Abstract][Full Text] [Related]
10. Cell cycle-dependent complex formation of BRCA1.CtIP.MRN is important for DNA double-strand break repair.
Chen L; Nievera CJ; Lee AY; Wu X
J Biol Chem; 2008 Mar; 283(12):7713-20. PubMed ID: 18171670
[TBL] [Abstract][Full Text] [Related]
11. The SNM1B/APOLLO DNA nuclease functions in resolution of replication stress and maintenance of common fragile site stability.
Mason JM; Das I; Arlt M; Patel N; Kraftson S; Glover TW; Sekiguchi JM
Hum Mol Genet; 2013 Dec; 22(24):4901-13. PubMed ID: 23863462
[TBL] [Abstract][Full Text] [Related]
12. Preventing over-resection by DNA2 helicase/nuclease suppresses repair defects in Fanconi anemia cells.
Karanja KK; Lee EH; Hendrickson EA; Campbell JL
Cell Cycle; 2014; 13(10):1540-50. PubMed ID: 24626199
[TBL] [Abstract][Full Text] [Related]
13. CtIP protein dimerization is critical for its recruitment to chromosomal DNA double-stranded breaks.
Wang H; Shao Z; Shi LZ; Hwang PY; Truong LN; Berns MW; Chen DJ; Wu X
J Biol Chem; 2012 Jun; 287(25):21471-80. PubMed ID: 22544744
[TBL] [Abstract][Full Text] [Related]
14. Replication Protein A (RPA) deficiency activates the Fanconi anemia DNA repair pathway.
Jang SW; Jung JK; Kim JM
Cell Cycle; 2016 Sep; 15(17):2336-45. PubMed ID: 27398742
[TBL] [Abstract][Full Text] [Related]
15. Collaborative action of Brca1 and CtIP in elimination of covalent modifications from double-strand breaks to facilitate subsequent break repair.
Nakamura K; Kogame T; Oshiumi H; Shinohara A; Sumitomo Y; Agama K; Pommier Y; Tsutsui KM; Tsutsui K; Hartsuiker E; Ogi T; Takeda S; Taniguchi Y
PLoS Genet; 2010 Jan; 6(1):e1000828. PubMed ID: 20107609
[TBL] [Abstract][Full Text] [Related]
16. Impaired 53BP1/RIF1 DSB mediated end-protection stimulates CtIP-dependent end resection and switches the repair to PARP1-dependent end joining in G1.
Bakr A; Köcher S; Volquardsen J; Petersen C; Borgmann K; Dikomey E; Rothkamm K; Mansour WY
Oncotarget; 2016 Sep; 7(36):57679-57693. PubMed ID: 27494840
[TBL] [Abstract][Full Text] [Related]
17. HP1 regulates the localization of FANCJ at sites of DNA double-strand breaks.
Wu W; Togashi Y; Johmura Y; Miyoshi Y; Nobuoka S; Nakanishi M; Ohta T
Cancer Sci; 2016 Oct; 107(10):1406-1415. PubMed ID: 27399284
[TBL] [Abstract][Full Text] [Related]
18. Fancd2 functions in a double strand break repair pathway that is distinct from non-homologous end joining.
Houghtaling S; Newell A; Akkari Y; Taniguchi T; Olson S; Grompe M
Hum Mol Genet; 2005 Oct; 14(20):3027-33. PubMed ID: 16135554
[TBL] [Abstract][Full Text] [Related]
19. Novel characteristics of CtIP at damage-induced foci following the initiation of DNA end resection.
Fujisawa H; Fujimori A; Okayasu R; Uesaka M; Yajima H
Mutat Res; 2015 Jan; 771():36-44. PubMed ID: 25771978
[TBL] [Abstract][Full Text] [Related]
20. C. elegans FANCD2 responds to replication stress and functions in interstrand cross-link repair.
Collis SJ; Barber LJ; Ward JD; Martin JS; Boulton SJ
DNA Repair (Amst); 2006 Nov; 5(11):1398-406. PubMed ID: 16914393
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
