BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

187 related articles for article (PubMed ID: 23146978)

  • 1. A novel target for diuretic therapy.
    Soleimani M
    Iran J Kidney Dis; 2012 Nov; 6(6):419-25. PubMed ID: 23146978
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Double knockout of pendrin and Na-Cl cotransporter (NCC) causes severe salt wasting, volume depletion, and renal failure.
    Soleimani M; Barone S; Xu J; Shull GE; Siddiqui F; Zahedi K; Amlal H
    Proc Natl Acad Sci U S A; 2012 Aug; 109(33):13368-73. PubMed ID: 22847418
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Pendrin as a novel target for diuretic therapy.
    Amlal H; Soleimani M
    Cell Physiol Biochem; 2011; 28(3):521-6. PubMed ID: 22116366
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Double knockout of carbonic anhydrase II (CAII) and Na(+)-Cl(-) cotransporter (NCC) causes salt wasting and volume depletion.
    Xu J; Barone S; Brooks MB; Soleimani M
    Cell Physiol Biochem; 2013; 32(7):173-83. PubMed ID: 24429824
    [TBL] [Abstract][Full Text] [Related]  

  • 5. The Role of Epithelial Sodium Channel ENaC and the Apical Cl-/HCO3- Exchanger Pendrin in Compensatory Salt Reabsorption in the Setting of Na-Cl Cotransporter (NCC) Inactivation.
    Patel-Chamberlin M; Varasteh Kia M; Xu J; Barone S; Zahedi K; Soleimani M
    PLoS One; 2016; 11(3):e0150918. PubMed ID: 26963391
    [TBL] [Abstract][Full Text] [Related]  

  • 6. The multiple roles of pendrin in the kidney.
    Soleimani M
    Nephrol Dial Transplant; 2015 Aug; 30(8):1257-66. PubMed ID: 25281699
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Prostaglandin-E2 Mediated Increase in Calcium and Phosphate Excretion in a Mouse Model of Distal Nephron Salt Wasting.
    Soleimani M; Barone S; Xu J; Alshahrani S; Brooks M; McCormack FX; Smith RD; Zahedi K
    PLoS One; 2016; 11(7):e0159804. PubMed ID: 27442254
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Two Mineralocorticoid Receptor-Mediated Mechanisms of Pendrin Activation in Distal Nephrons.
    Ayuzawa N; Nishimoto M; Ueda K; Hirohama D; Kawarazaki W; Shimosawa T; Marumo T; Fujita T
    J Am Soc Nephrol; 2020 Apr; 31(4):748-764. PubMed ID: 32034107
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Small-Molecule Inhibitors of Pendrin Potentiate the Diuretic Action of Furosemide.
    Cil O; Haggie PM; Phuan PW; Tan JA; Verkman AS
    J Am Soc Nephrol; 2016 Dec; 27(12):3706-3714. PubMed ID: 27153921
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Ablation of the Cl-/HCO3- Exchanger Pendrin Enhances Hydrochlorothiazide-Induced Diuresis.
    Alshahrani S; Soleimani M
    Kidney Blood Press Res; 2017; 42(3):444-455. PubMed ID: 28750403
    [TBL] [Abstract][Full Text] [Related]  

  • 11. The chloride-bicarbonate exchanger pendrin is increased in the kidney of the pregnant rat.
    West CA; Verlander JW; Wall SM; Baylis C
    Exp Physiol; 2015 Oct; 100(10):1177-86. PubMed ID: 26260990
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Enhanced passive Ca2+ reabsorption and reduced Mg2+ channel abundance explains thiazide-induced hypocalciuria and hypomagnesemia.
    Nijenhuis T; Vallon V; van der Kemp AW; Loffing J; Hoenderop JG; Bindels RJ
    J Clin Invest; 2005 Jun; 115(6):1651-8. PubMed ID: 15902302
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Reduced ENaC protein abundance contributes to the lower blood pressure observed in pendrin-null mice.
    Kim YH; Pech V; Spencer KB; Beierwaltes WH; Everett LA; Green ED; Shin W; Verlander JW; Sutliff RL; Wall SM
    Am J Physiol Renal Physiol; 2007 Oct; 293(4):F1314-24. PubMed ID: 17686956
    [TBL] [Abstract][Full Text] [Related]  

  • 14. A mouse model of pseudohypoaldosteronism type II reveals a novel mechanism of renal tubular acidosis.
    López-Cayuqueo KI; Chavez-Canales M; Pillot A; Houillier P; Jayat M; Baraka-Vidot J; Trepiccione F; Baudrie V; Büsst C; Soukaseum C; Kumai Y; Jeunemaître X; Hadchouel J; Eladari D; Chambrey R
    Kidney Int; 2018 Sep; 94(3):514-523. PubMed ID: 30146013
    [TBL] [Abstract][Full Text] [Related]  

  • 15. The role of pendrin in renal physiology.
    Wall SM; Lazo-Fernandez Y
    Annu Rev Physiol; 2015; 77():363-78. PubMed ID: 25668022
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Inactivation of the Na-Cl co-transporter (NCC) gene is associated with high BMD through both renal and bone mechanisms: analysis of patients with Gitelman syndrome and Ncc null mice.
    Nicolet-Barousse L; Blanchard A; Roux C; Pietri L; Bloch-Faure M; Kolta S; Chappard C; Geoffroy V; Morieux C; Jeunemaitre X; Shull GE; Meneton P; Paillard M; Houillier P; De Vernejoul MC
    J Bone Miner Res; 2005 May; 20(5):799-808. PubMed ID: 15824853
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Identification of a novel target of thiazide diuretics.
    Eladari D; Chambrey R
    J Nephrol; 2011; 24(4):391-4. PubMed ID: 21667455
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Potentiation of the effect of thiazide derivatives by carbonic anhydrase inhibitors: molecular mechanisms and potential clinical implications.
    Zahedi K; Barone S; Xu J; Soleimani M
    PLoS One; 2013; 8(11):e79327. PubMed ID: 24260196
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Generation and analysis of the thiazide-sensitive Na+ -Cl- cotransporter (Ncc/Slc12a3) Ser707X knockin mouse as a model of Gitelman syndrome.
    Yang SS; Lo YF; Yu IS; Lin SW; Chang TH; Hsu YJ; Chao TK; Sytwu HK; Uchida S; Sasaki S; Lin SH
    Hum Mutat; 2010 Dec; 31(12):1304-15. PubMed ID: 20848653
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Renal expression of parvalbumin is critical for NaCl handling and response to diuretics.
    Belge H; Gailly P; Schwaller B; Loffing J; Debaix H; Riveira-Munoz E; Beauwens R; Devogelaer JP; Hoenderop JG; Bindels RJ; Devuyst O
    Proc Natl Acad Sci U S A; 2007 Sep; 104(37):14849-54. PubMed ID: 17804801
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 10.