187 related articles for article (PubMed ID: 23146978)
1. A novel target for diuretic therapy.
Soleimani M
Iran J Kidney Dis; 2012 Nov; 6(6):419-25. PubMed ID: 23146978
[TBL] [Abstract][Full Text] [Related]
2. Double knockout of pendrin and Na-Cl cotransporter (NCC) causes severe salt wasting, volume depletion, and renal failure.
Soleimani M; Barone S; Xu J; Shull GE; Siddiqui F; Zahedi K; Amlal H
Proc Natl Acad Sci U S A; 2012 Aug; 109(33):13368-73. PubMed ID: 22847418
[TBL] [Abstract][Full Text] [Related]
3. Pendrin as a novel target for diuretic therapy.
Amlal H; Soleimani M
Cell Physiol Biochem; 2011; 28(3):521-6. PubMed ID: 22116366
[TBL] [Abstract][Full Text] [Related]
4. Double knockout of carbonic anhydrase II (CAII) and Na(+)-Cl(-) cotransporter (NCC) causes salt wasting and volume depletion.
Xu J; Barone S; Brooks MB; Soleimani M
Cell Physiol Biochem; 2013; 32(7):173-83. PubMed ID: 24429824
[TBL] [Abstract][Full Text] [Related]
5. The Role of Epithelial Sodium Channel ENaC and the Apical Cl-/HCO3- Exchanger Pendrin in Compensatory Salt Reabsorption in the Setting of Na-Cl Cotransporter (NCC) Inactivation.
Patel-Chamberlin M; Varasteh Kia M; Xu J; Barone S; Zahedi K; Soleimani M
PLoS One; 2016; 11(3):e0150918. PubMed ID: 26963391
[TBL] [Abstract][Full Text] [Related]
6. The multiple roles of pendrin in the kidney.
Soleimani M
Nephrol Dial Transplant; 2015 Aug; 30(8):1257-66. PubMed ID: 25281699
[TBL] [Abstract][Full Text] [Related]
7. Prostaglandin-E2 Mediated Increase in Calcium and Phosphate Excretion in a Mouse Model of Distal Nephron Salt Wasting.
Soleimani M; Barone S; Xu J; Alshahrani S; Brooks M; McCormack FX; Smith RD; Zahedi K
PLoS One; 2016; 11(7):e0159804. PubMed ID: 27442254
[TBL] [Abstract][Full Text] [Related]
8. Two Mineralocorticoid Receptor-Mediated Mechanisms of Pendrin Activation in Distal Nephrons.
Ayuzawa N; Nishimoto M; Ueda K; Hirohama D; Kawarazaki W; Shimosawa T; Marumo T; Fujita T
J Am Soc Nephrol; 2020 Apr; 31(4):748-764. PubMed ID: 32034107
[TBL] [Abstract][Full Text] [Related]
9. Small-Molecule Inhibitors of Pendrin Potentiate the Diuretic Action of Furosemide.
Cil O; Haggie PM; Phuan PW; Tan JA; Verkman AS
J Am Soc Nephrol; 2016 Dec; 27(12):3706-3714. PubMed ID: 27153921
[TBL] [Abstract][Full Text] [Related]
10. Ablation of the Cl-/HCO3- Exchanger Pendrin Enhances Hydrochlorothiazide-Induced Diuresis.
Alshahrani S; Soleimani M
Kidney Blood Press Res; 2017; 42(3):444-455. PubMed ID: 28750403
[TBL] [Abstract][Full Text] [Related]
11. The chloride-bicarbonate exchanger pendrin is increased in the kidney of the pregnant rat.
West CA; Verlander JW; Wall SM; Baylis C
Exp Physiol; 2015 Oct; 100(10):1177-86. PubMed ID: 26260990
[TBL] [Abstract][Full Text] [Related]
12. Enhanced passive Ca2+ reabsorption and reduced Mg2+ channel abundance explains thiazide-induced hypocalciuria and hypomagnesemia.
Nijenhuis T; Vallon V; van der Kemp AW; Loffing J; Hoenderop JG; Bindels RJ
J Clin Invest; 2005 Jun; 115(6):1651-8. PubMed ID: 15902302
[TBL] [Abstract][Full Text] [Related]
13. Reduced ENaC protein abundance contributes to the lower blood pressure observed in pendrin-null mice.
Kim YH; Pech V; Spencer KB; Beierwaltes WH; Everett LA; Green ED; Shin W; Verlander JW; Sutliff RL; Wall SM
Am J Physiol Renal Physiol; 2007 Oct; 293(4):F1314-24. PubMed ID: 17686956
[TBL] [Abstract][Full Text] [Related]
14. A mouse model of pseudohypoaldosteronism type II reveals a novel mechanism of renal tubular acidosis.
López-Cayuqueo KI; Chavez-Canales M; Pillot A; Houillier P; Jayat M; Baraka-Vidot J; Trepiccione F; Baudrie V; Büsst C; Soukaseum C; Kumai Y; Jeunemaître X; Hadchouel J; Eladari D; Chambrey R
Kidney Int; 2018 Sep; 94(3):514-523. PubMed ID: 30146013
[TBL] [Abstract][Full Text] [Related]
15. The role of pendrin in renal physiology.
Wall SM; Lazo-Fernandez Y
Annu Rev Physiol; 2015; 77():363-78. PubMed ID: 25668022
[TBL] [Abstract][Full Text] [Related]
16. Inactivation of the Na-Cl co-transporter (NCC) gene is associated with high BMD through both renal and bone mechanisms: analysis of patients with Gitelman syndrome and Ncc null mice.
Nicolet-Barousse L; Blanchard A; Roux C; Pietri L; Bloch-Faure M; Kolta S; Chappard C; Geoffroy V; Morieux C; Jeunemaitre X; Shull GE; Meneton P; Paillard M; Houillier P; De Vernejoul MC
J Bone Miner Res; 2005 May; 20(5):799-808. PubMed ID: 15824853
[TBL] [Abstract][Full Text] [Related]
17. Identification of a novel target of thiazide diuretics.
Eladari D; Chambrey R
J Nephrol; 2011; 24(4):391-4. PubMed ID: 21667455
[TBL] [Abstract][Full Text] [Related]
18. Potentiation of the effect of thiazide derivatives by carbonic anhydrase inhibitors: molecular mechanisms and potential clinical implications.
Zahedi K; Barone S; Xu J; Soleimani M
PLoS One; 2013; 8(11):e79327. PubMed ID: 24260196
[TBL] [Abstract][Full Text] [Related]
19. Generation and analysis of the thiazide-sensitive Na+ -Cl- cotransporter (Ncc/Slc12a3) Ser707X knockin mouse as a model of Gitelman syndrome.
Yang SS; Lo YF; Yu IS; Lin SW; Chang TH; Hsu YJ; Chao TK; Sytwu HK; Uchida S; Sasaki S; Lin SH
Hum Mutat; 2010 Dec; 31(12):1304-15. PubMed ID: 20848653
[TBL] [Abstract][Full Text] [Related]
20. Renal expression of parvalbumin is critical for NaCl handling and response to diuretics.
Belge H; Gailly P; Schwaller B; Loffing J; Debaix H; Riveira-Munoz E; Beauwens R; Devogelaer JP; Hoenderop JG; Bindels RJ; Devuyst O
Proc Natl Acad Sci U S A; 2007 Sep; 104(37):14849-54. PubMed ID: 17804801
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]