137 related articles for article (PubMed ID: 23178458)
1. Role of glutamate and nitric oxide in onset of motor neuron degeneration in neurolathyrism.
Khandare AL; Ankulu M; Aparna N
Neurotoxicology; 2013 Jan; 34():269-74. PubMed ID: 23178458
[TBL] [Abstract][Full Text] [Related]
2. [Research in Motor Neuron Diseases Caused by Natural Substances: Focus on Pathological Mechanisms of Neurolathyrism].
Kusama-Eguchi K
Yakugaku Zasshi; 2019; 139(4):609-615. PubMed ID: 30930396
[TBL] [Abstract][Full Text] [Related]
3. L-β-N-oxalyl-α,β-diaminopropionic acid toxicity in motor neurons.
Van Moorhem M; Decrock E; De Vuyst E; De Bock M; Wang N; Lambein F; Van Den Bosch L; Leybaert L
Neuroreport; 2011 Feb; 22(3):131-5. PubMed ID: 21341395
[TBL] [Abstract][Full Text] [Related]
4. Unraveling the mechanism of β-N-oxalyl-α,β-diaminopropionic acid (β-ODAP) induced excitotoxicity and oxidative stress, relevance for neurolathyrism prevention.
Van Moorhem M; Lambein F; Leybaert L
Food Chem Toxicol; 2011 Mar; 49(3):550-5. PubMed ID: 20510327
[TBL] [Abstract][Full Text] [Related]
5. The Lathyrus excitotoxin beta-N-oxalyl-L-alpha,beta-diaminopropionic acid is a substrate of the L-cystine/L-glutamate exchanger system xc-.
Warren BA; Patel SA; Nunn PB; Bridges RJ
Toxicol Appl Pharmacol; 2004 Oct; 200(2):83-92. PubMed ID: 15476861
[TBL] [Abstract][Full Text] [Related]
6. New insights into the mechanism of neurolathyrism: L-β-ODAP triggers [Ca2+]i accumulation and cell death in primary motor neurons through transient receptor potential channels and metabotropic glutamate receptors.
Kusama-Eguchi K; Miyano T; Yamamoto M; Suda A; Ito Y; Ishige K; Ishii M; Ogawa Y; Watanabe K; Ikegami F; Kusama T
Food Chem Toxicol; 2014 May; 67():113-22. PubMed ID: 24582715
[TBL] [Abstract][Full Text] [Related]
7. Sulfur amino acids deficiency caused by grass pea diet plays an important role in the toxicity of L-β-ODAP by increasing the oxidative stress: studies on a motor neuron cell line.
Kusama-Eguchi K; Yoshino N; Minoura A; Watanabe K; Kusama T; Lambein F; Ikegami F
Food Chem Toxicol; 2011 Mar; 49(3):636-43. PubMed ID: 20691243
[TBL] [Abstract][Full Text] [Related]
8. Hind-limb paraparesis in a rat model for neurolathyrism associated with apoptosis and an impaired vascular endothelial growth factor system in the spinal cord.
Kusama-Eguchi K; Yamazaki Y; Ueda T; Suda A; Hirayama Y; Ikegami F; Watanabe K; May M; Lambein F; Kusama T
J Comp Neurol; 2010 Mar; 518(6):928-42. PubMed ID: 20058324
[TBL] [Abstract][Full Text] [Related]
9. Vascular insult accompanied by overexpressed heme oxygenase-1 as a pathophysiological mechanism in experimental neurolathyrism with hind-leg paraparesis.
Kawaguchi K; Lambein F; Kusama-Eguchi K
Biochem Biophys Res Commun; 2012 Nov; 428(1):160-6. PubMed ID: 23068093
[TBL] [Abstract][Full Text] [Related]
10. Stress-related over-enhancement of the hypothalamic-pituitary-adrenal axis causes experimental neurolathyrism in rats.
Minagawa K; Yamada SI; Suzuki A; Ta S; Kumai T; Lambein F; Kusama-Eguchi K
Environ Toxicol Pharmacol; 2019 Nov; 72():103245. PubMed ID: 31499324
[TBL] [Abstract][Full Text] [Related]
11. Downregulation of glutaredoxin but not glutathione loss leads to mitochondrial dysfunction in female mice CNS: implications in excitotoxicity.
Diwakar L; Kenchappa RS; Annepu J; Ravindranath V
Neurochem Int; 2007 Jul; 51(1):37-46. PubMed ID: 17512091
[TBL] [Abstract][Full Text] [Related]
12. ALS-linked Cu/Zn-SOD mutation increases vulnerability of motor neurons to excitotoxicity by a mechanism involving increased oxidative stress and perturbed calcium homeostasis.
Kruman II; Pedersen WA; Springer JE; Mattson MP
Exp Neurol; 1999 Nov; 160(1):28-39. PubMed ID: 10630188
[TBL] [Abstract][Full Text] [Related]
13. Determination of β-N-oxalyl-L-α,β-diaminopropionic acid and homoarginine in Lathyrus sativus and Lathyrus cicera by capillary zone electrophoresis.
Sacristán M; Varela A; Pedrosa MM; Burbano C; Cuadrado C; Legaz ME; Muzquiz M
J Sci Food Agric; 2015 May; 95(7):1414-20. PubMed ID: 24954190
[TBL] [Abstract][Full Text] [Related]
14. A rat model of neurolathyrism: repeated injection of L: -beta-ODAP induces the paraparesis of the hind legs.
Kusama-Eguchi K; Ikegami F; Kusama T; Suda A; Ogawa Y; Igarashi K; Watanabe K
Amino Acids; 2005 Mar; 28(2):139-43. PubMed ID: 15714257
[TBL] [Abstract][Full Text] [Related]
15. Brain glutathione as a target for aetiological factors in neurolathyrism and konzo.
Nunn PB; Lyddiard JR; Christopher Perera KP
Food Chem Toxicol; 2011 Mar; 49(3):662-7. PubMed ID: 20816718
[TBL] [Abstract][Full Text] [Related]
16. Current scenario of consumption of Lathyrus sativus and lathyrism in three districts of Chhattisgarh State, India.
Khandare AL; Kumar RH; Meshram II; Arlappa N; Laxmaiah A; Venkaiah K; Rao PA; Validandi V; Toteja GS
Toxicon; 2018 Aug; 150():228-234. PubMed ID: 29908260
[TBL] [Abstract][Full Text] [Related]
17. Neurolathyrism: mitochondrial dysfunction in excitotoxicity mediated by L-beta-oxalyl aminoalanine.
Ravindranath V
Neurochem Int; 2002 May; 40(6):505-9. PubMed ID: 11850107
[TBL] [Abstract][Full Text] [Related]
18. A look at the brighter facets of β-N-oxalyl-l-α,β-diaminopropionic acid, homoarginine and the grass pea.
Rao SL
Food Chem Toxicol; 2011 Mar; 49(3):620-2. PubMed ID: 20654679
[TBL] [Abstract][Full Text] [Related]
19. L-beta-ODAP alters mitochondrial Ca2+ handling as an early event in excitotoxicity.
Van Moorhem M; Decrock E; Coussee E; Faes L; De Vuyst E; Vranckx K; De Bock M; Wang N; D'Herde K; Lambein F; Callewaert G; Leybaert L
Cell Calcium; 2010 Mar; 47(3):287-96. PubMed ID: 20129666
[TBL] [Abstract][Full Text] [Related]
20. Loss of nitric oxide-mediated down-regulation of NMDA receptors in neurofilament aggregate-bearing motor neurons in vitro: implications for motor neuron disease.
Sanelli T; Strong MJ
Free Radic Biol Med; 2007 Jan; 42(1):143-51. PubMed ID: 17157201
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
