These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
79 related articles for article (PubMed ID: 23206802)
41. Mitochondrial encephalocardio-myopathy with early neonatal onset due to TMEM70 mutation. Honzík T; Tesarová M; Mayr JA; Hansíková H; Jesina P; Bodamer O; Koch J; Magner M; Freisinger P; Huemer M; Kostková O; van Coster R; Kmoch S; Houstêk J; Sperl W; Zeman J Arch Dis Child; 2010 Apr; 95(4):296-301. PubMed ID: 20335238 [TBL] [Abstract][Full Text] [Related]
42. MELAS phenotype associated with m.3302A>G mutation in mitochondrial tRNA(Leu(UUR)) gene. Goto M; Komaki H; Saito T; Saito Y; Nakagawa E; Sugai K; Sasaki M; Nishino I; Goto Y Brain Dev; 2014 Feb; 36(2):180-2. PubMed ID: 23582502 [TBL] [Abstract][Full Text] [Related]
43. The mutation at nt 8993 of mitochondrial DNA is a common cause of Leigh's syndrome. Santorelli FM; Shanske S; Macaya A; DeVivo DC; DiMauro S Ann Neurol; 1993 Dec; 34(6):827-34. PubMed ID: 8250532 [TBL] [Abstract][Full Text] [Related]
44. Reduced respiratory control with ADP and changed pattern of respiratory chain enzymes as a result of selective deficiency of the mitochondrial ATP synthase. Mayr JA; Paul J; Pecina P; Kurnik P; Förster H; Fötschl U; Sperl W; Houstek J Pediatr Res; 2004 Jun; 55(6):988-94. PubMed ID: 15155867 [TBL] [Abstract][Full Text] [Related]
45. Late-onset Leigh syndrome with m.9176T>C mutation in the mitochondrial ATPase 6 gene. Ichikawa K; Tsuyusaki Y; Shimbo H; Goto T Pediatr Int; 2019 Oct; 61(10):1055-1056. PubMed ID: 31625254 [No Abstract] [Full Text] [Related]
46. At environmental doses, dietary methylmercury inhibits mitochondrial energy metabolism in skeletal muscles of the zebra fish (Danio rerio). Cambier S; Bénard G; Mesmer-Dudons N; Gonzalez P; Rossignol R; Brèthes D; Bourdineaud JP Int J Biochem Cell Biol; 2009 Apr; 41(4):791-9. PubMed ID: 18765295 [TBL] [Abstract][Full Text] [Related]
47. Deep sequencing discovery of causal mtDNA mutations in a patient with unspecific neurological disease. Spangenberg L; Graña M; Mansilla S; Martínez J; Tapié A; Greif G; Montano N; Vaglio A; Gueçaimburú R; Robello C; Castro L; Quijano C; Raggio V; Naya H Mitochondrion; 2019 May; 46():337-344. PubMed ID: 30227252 [TBL] [Abstract][Full Text] [Related]
48. Adult-onset spinocerebellar ataxia due to MTATP6 mutations: are they more common than previously thought? Pulkes T J Neurol Neurosurg Psychiatry; 2012 Sep; 83(9):857-8. PubMed ID: 22773856 [No Abstract] [Full Text] [Related]
49. Facial Dysmorphism, Hirsutism, and Failure to Thrive as Manifestation of Leigh Syndrome in a Child with Baskaran D; Hussain N J Pediatr Neurosci; 2020; 15(2):108-110. PubMed ID: 33042241 [TBL] [Abstract][Full Text] [Related]
50. Motor Neuron Syndrome as a New Phenotypic Manifestation of Mutation 9185T>C in Gene MTATP6. Brum M; Semedo C; Guerreiro R; Pinto Marques J Case Rep Neurol Med; 2014; 2014():701761. PubMed ID: 25548692 [TBL] [Abstract][Full Text] [Related]
51. [Mitochondrial ATP synthase deficiency due to 8993T > G mutation on ATP6 gene]. Ma YY; Wu TF; Liu YP Zhonghua Er Ke Za Zhi; 2011 Jul; 49(7):557-8. PubMed ID: 22088191 [No Abstract] [Full Text] [Related]
52. Abnormal mitochondria in the Rett syndrome. Eeg-Olofsson O; al-Zuhair AG; Teebi AS; al-Essa MM Brain Dev; 1988; 10(4):260-2. PubMed ID: 3218707 [TBL] [Abstract][Full Text] [Related]
53. m.8993T>G-Associated Leigh Syndrome with Hypocitrullinemia on Newborn Screening. Mori M; Mytinger JR; Martin LC; Bartholomew D; Hickey S JIMD Rep; 2014; 17():47-51. PubMed ID: 25240982 [TBL] [Abstract][Full Text] [Related]
54. An evolutionary insight into mutation of ATPase6 gene in primary ovarian insufficiency. Venkatesh S; Dada R Arch Gynecol Obstet; 2011 Jul; 284(1):251-2. PubMed ID: 21165743 [No Abstract] [Full Text] [Related]
55. Hyperammonemic crisis in a child with ATP synthase deficiency caused by mtDNA mutation m.8851T>C. Dvorakova V; Magner M; Honzik T Mol Genet Metab Rep; 2015 Mar; 2():46. PubMed ID: 28649526 [No Abstract] [Full Text] [Related]
56. Defective mitochondrial ATPase due to rare mtDNA m.8969G>A mutation-causing lactic acidosis, intellectual disability, and poor growth. Isohanni P; Carroll CJ; Jackson CB; Pohjanpelto M; Lönnqvist T; Suomalainen A Neurogenetics; 2018 Jan; 19(1):49-53. PubMed ID: 29350304 [TBL] [Abstract][Full Text] [Related]
57. Identification of G8969>A in mitochondrial ATP6 gene that severely compromises ATP synthase function in a patient with IgA nephropathy. Wen S; Niedzwiecka K; Zhao W; Xu S; Liang S; Zhu X; Xie H; Tribouillard-Tanvier D; Giraud MF; Zeng C; Dautant A; Kucharczyk R; Liu Z; di Rago JP; Chen H Sci Rep; 2016 Nov; 6():36313. PubMed ID: 27812026 [TBL] [Abstract][Full Text] [Related]
58. Mitochondrial myopathy, lactic acidosis, and sideroblastic anemia (MLASA) plus associated with a novel de novo mutation (m.8969G>A) in the mitochondrial encoded ATP6 gene. Burrage LC; Tang S; Wang J; Donti TR; Walkiewicz M; Luchak JM; Chen LC; Schmitt ES; Niu Z; Erana R; Hunter JV; Graham BH; Wong LJ; Scaglia F Mol Genet Metab; 2014 Nov; 113(3):207-12. PubMed ID: 25037980 [TBL] [Abstract][Full Text] [Related]