These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
173 related articles for article (PubMed ID: 23212364)
1. Cooperation between different RNA virus genomes produces a new phenotype. Shirogane Y; Watanabe S; Yanagi Y Nat Commun; 2012; 3():1235. PubMed ID: 23212364 [TBL] [Abstract][Full Text] [Related]
2. Cooperation: another mechanism of viral evolution. Shirogane Y; Watanabe S; Yanagi Y Trends Microbiol; 2013 Jul; 21(7):320-4. PubMed ID: 23764388 [TBL] [Abstract][Full Text] [Related]
3. Mutations in the stalk region of the measles virus hemagglutinin inhibit syncytium formation but not virus entry. Ennis MK; Hu C; Naik SK; Hallak LK; Peng KW; Russell SJ; Dingli D J Virol; 2010 Oct; 84(20):10913-7. PubMed ID: 20702637 [TBL] [Abstract][Full Text] [Related]
4. Importance of the cytoplasmic tails of the measles virus glycoproteins for fusogenic activity and the generation of recombinant measles viruses. Moll M; Klenk HD; Maisner A J Virol; 2002 Jul; 76(14):7174-86. PubMed ID: 12072517 [TBL] [Abstract][Full Text] [Related]
5. Attenuation of recombinant vesicular stomatitis viruses encoding mutant glycoproteins demonstrate a critical role for maintaining a high pH threshold for membrane fusion in viral fitness. Fredericksen BL; Whitt MA Virology; 1998 Jan; 240(2):349-58. PubMed ID: 9454708 [TBL] [Abstract][Full Text] [Related]
6. A single amino acid substitution in the measles virus F₂ protein reciprocally modulates membrane fusion activity in pathogenic and oncolytic strains. Heidmeier S; Hanauer JR; Friedrich K; Prüfer S; Schneider IC; Buchholz CJ; Cichutek K; Mühlebach MD Virus Res; 2014 Feb; 180():43-8. PubMed ID: 24368277 [TBL] [Abstract][Full Text] [Related]
8. A recombinant measles vaccine virus expressing wild-type glycoproteins: consequences for viral spread and cell tropism. Johnston IC; ter Meulen V; Schneider-Schaulies J; Schneider-Schaulies S J Virol; 1999 Aug; 73(8):6903-15. PubMed ID: 10400788 [TBL] [Abstract][Full Text] [Related]
9. Resistance of a measles virus mutant to fusion inhibitory oligopeptides is not associated with mutations in the fusion peptide. Hull JD; Krah DL; Choppin PW Virology; 1987 Aug; 159(2):368-72. PubMed ID: 2887065 [TBL] [Abstract][Full Text] [Related]
10. Mutant fusion proteins with enhanced fusion activity promote measles virus spread in human neuronal cells and brains of suckling hamsters. Watanabe S; Shirogane Y; Suzuki SO; Ikegame S; Koga R; Yanagi Y J Virol; 2013 Mar; 87(5):2648-59. PubMed ID: 23255801 [TBL] [Abstract][Full Text] [Related]
11. Cell fusion by the envelope glycoproteins of persistent measles viruses which caused lethal human brain disease. Cattaneo R; Rose JK J Virol; 1993 Mar; 67(3):1493-502. PubMed ID: 8437226 [TBL] [Abstract][Full Text] [Related]
12. Measles Virus Bearing Measles Inclusion Body Encephalitis-Derived Fusion Protein Is Pathogenic after Infection via the Respiratory Route. Mathieu C; Ferren M; Jurgens E; Dumont C; Rybkina K; Harder O; Stelitano D; Madeddu S; Sanna G; Schwartz D; Biswas S; Hardie D; Hashiguchi T; Moscona A; Horvat B; Niewiesk S; Porotto M J Virol; 2019 Apr; 93(8):. PubMed ID: 30728259 [TBL] [Abstract][Full Text] [Related]
13. Cooperation between different variants: A unique potential for virus evolution. Shirogane Y; Watanabe S; Yanagi Y Virus Res; 2019 Apr; 264():68-73. PubMed ID: 30822430 [TBL] [Abstract][Full Text] [Related]
14. Analysis of a Subacute Sclerosing Panencephalitis Genotype B3 Virus from the 2009-2010 South African Measles Epidemic Shows That Hyperfusogenic F Proteins Contribute to Measles Virus Infection in the Brain. Angius F; Smuts H; Rybkina K; Stelitano D; Eley B; Wilmshurst J; Ferren M; Lalande A; Mathieu C; Moscona A; Horvat B; Hashiguchi T; Porotto M; Hardie D J Virol; 2019 Feb; 93(4):. PubMed ID: 30487282 [TBL] [Abstract][Full Text] [Related]
15. A recombinant vesicular stomatitis virus bearing a lethal mutation in the glycoprotein gene uncovers a second site suppressor that restores fusion. Stanifer ML; Cureton DK; Whelan SP J Virol; 2011 Aug; 85(16):8105-15. PubMed ID: 21680501 [TBL] [Abstract][Full Text] [Related]
16. Leucine at position 278 of the AIK-C measles virus vaccine strain fusion protein is responsible for reduced syncytium formation. Nakayama T; Komase K; Uzuka R; Hoshi A; Okafuji T J Gen Virol; 2001 Sep; 82(Pt 9):2143-2150. PubMed ID: 11514723 [TBL] [Abstract][Full Text] [Related]
17. Cell-to-Cell Measles Virus Spread between Human Neurons Is Dependent on Hemagglutinin and Hyperfusogenic Fusion Protein. Sato Y; Watanabe S; Fukuda Y; Hashiguchi T; Yanagi Y; Ohno S J Virol; 2018 Mar; 92(6):. PubMed ID: 29298883 [TBL] [Abstract][Full Text] [Related]
18. The measles virus fusion protein transmembrane region modulates availability of an active glycoprotein complex and fusion efficiency. Mühlebach MD; Leonard VH; Cattaneo R J Virol; 2008 Nov; 82(22):11437-45. PubMed ID: 18786999 [TBL] [Abstract][Full Text] [Related]
19. The many mechanisms of viral membrane fusion proteins. Earp LJ; Delos SE; Park HE; White JM Curr Top Microbiol Immunol; 2005; 285():25-66. PubMed ID: 15609500 [TBL] [Abstract][Full Text] [Related]
20. Mutations in the putative HR-C region of the measles virus F2 glycoprotein modulate syncytium formation. Plemper RK; Compans RW J Virol; 2003 Apr; 77(7):4181-90. PubMed ID: 12634376 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]