BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

388 related articles for article (PubMed ID: 23261783)

  • 1. Matrix metalloproteinase-28 deletion exacerbates cardiac dysfunction and rupture after myocardial infarction in mice by inhibiting M2 macrophage activation.
    Ma Y; Halade GV; Zhang J; Ramirez TA; Levin D; Voorhees A; Jin YF; Han HC; Manicone AM; Lindsey ML
    Circ Res; 2013 Feb; 112(4):675-88. PubMed ID: 23261783
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Early matrix metalloproteinase-12 inhibition worsens post-myocardial infarction cardiac dysfunction by delaying inflammation resolution.
    Iyer RP; Patterson NL; Zouein FA; Ma Y; Dive V; de Castro Brás LE; Lindsey ML
    Int J Cardiol; 2015 Apr; 185():198-208. PubMed ID: 25797678
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Building a better infarct: Modulation of collagen cross-linking to increase infarct stiffness and reduce left ventricular dilation post-myocardial infarction.
    Voorhees AP; DeLeon-Pennell KY; Ma Y; Halade GV; Yabluchanskiy A; Iyer RP; Flynn E; Cates CA; Lindsey ML; Han HC
    J Mol Cell Cardiol; 2015 Aug; 85():229-39. PubMed ID: 26080361
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Reduced scar maturation and contractility lead to exaggerated left ventricular dilation after myocardial infarction in mice lacking AMPKα1.
    Noppe G; Dufeys C; Buchlin P; Marquet N; Castanares-Zapatero D; Balteau M; Hermida N; Bouzin C; Esfahani H; Viollet B; Bertrand L; Balligand JL; Vanoverschelde JL; Beauloye C; Horman S
    J Mol Cell Cardiol; 2014 Sep; 74():32-43. PubMed ID: 24805196
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Macrophage overexpression of matrix metalloproteinase-9 in aged mice improves diastolic physiology and cardiac wound healing after myocardial infarction.
    Meschiari CA; Jung M; Iyer RP; Yabluchanskiy A; Toba H; Garrett MR; Lindsey ML
    Am J Physiol Heart Circ Physiol; 2018 Feb; 314(2):H224-H235. PubMed ID: 29030341
    [TBL] [Abstract][Full Text] [Related]  

  • 6. P. gingivalis lipopolysaccharide intensifies inflammation post-myocardial infarction through matrix metalloproteinase-9.
    DeLeon-Pennell KY; de Castro Brás LE; Iyer RP; Bratton DR; Jin YF; Ripplinger CM; Lindsey ML
    J Mol Cell Cardiol; 2014 Nov; 76():218-26. PubMed ID: 25240641
    [TBL] [Abstract][Full Text] [Related]  

  • 7. MK5 haplodeficiency decreases collagen deposition and scar size during post-myocardial infarction wound repair.
    Nawaito SA; Sahadevan P; Clavet-Lanthier MÉ; Pouliot P; Sahmi F; Shi Y; Gillis MA; Lesage F; Gaestel M; Sirois MG; Calderone A; Tardif JC; Allen BG
    Am J Physiol Heart Circ Physiol; 2019 Jun; 316(6):H1281-H1296. PubMed ID: 30901279
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Early matrix metalloproteinase-9 inhibition post-myocardial infarction worsens cardiac dysfunction by delaying inflammation resolution.
    Iyer RP; de Castro Brás LE; Patterson NL; Bhowmick M; Flynn ER; Asher M; Cannon PL; Deleon-Pennell KY; Fields GB; Lindsey ML
    J Mol Cell Cardiol; 2016 Nov; 100():109-117. PubMed ID: 27746126
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Age-related differences in postinfarct left ventricular rupture and remodeling.
    Yang Y; Ma Y; Han W; Li J; Xiang Y; Liu F; Ma X; Zhang J; Fu Z; Su YD; Du XJ; Gao XM
    Am J Physiol Heart Circ Physiol; 2008 Apr; 294(4):H1815-22. PubMed ID: 18263717
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Transgenic overexpression of matrix metalloproteinase-9 in macrophages attenuates the inflammatory response and improves left ventricular function post-myocardial infarction.
    Zamilpa R; Ibarra J; de Castro Brás LE; Ramirez TA; Nguyen N; Halade GV; Zhang J; Dai Q; Dayah T; Chiao YA; Lowell W; Ahuja SS; D'Armiento J; Jin YF; Lindsey ML
    J Mol Cell Cardiol; 2012 Nov; 53(5):599-608. PubMed ID: 22884843
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Excessive tumor necrosis factor activation after infarction contributes to susceptibility of myocardial rupture and left ventricular dysfunction.
    Sun M; Dawood F; Wen WH; Chen M; Dixon I; Kirshenbaum LA; Liu PP
    Circulation; 2004 Nov; 110(20):3221-8. PubMed ID: 15533863
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Epilysin (matrix metalloproteinase-28) joins the matrix metalloproteinase team on the field of postmyocardial infarction remodeling.
    Spinale FG
    Circ Res; 2013 Feb; 112(4):579-82. PubMed ID: 23410875
    [No Abstract]   [Full Text] [Related]  

  • 13. Interleukin-35 Promotes Macrophage Survival and Improves Wound Healing After Myocardial Infarction in Mice.
    Jia D; Jiang H; Weng X; Wu J; Bai P; Yang W; Wang Z; Hu K; Sun A; Ge J
    Circ Res; 2019 Apr; 124(9):1323-1336. PubMed ID: 30832557
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Ataxia telangiectasia-mutated kinase deficiency exacerbates left ventricular dysfunction and remodeling late after myocardial infarction.
    Daniel LL; Scofield SL; Thrasher P; Dalal S; Daniels CR; Foster CR; Singh M; Singh K
    Am J Physiol Heart Circ Physiol; 2016 Aug; 311(2):H445-52. PubMed ID: 27288435
    [TBL] [Abstract][Full Text] [Related]  

  • 15. SPARC mediates early extracellular matrix remodeling following myocardial infarction.
    McCurdy SM; Dai Q; Zhang J; Zamilpa R; Ramirez TA; Dayah T; Nguyen N; Jin YF; Bradshaw AD; Lindsey ML
    Am J Physiol Heart Circ Physiol; 2011 Aug; 301(2):H497-505. PubMed ID: 21602472
    [TBL] [Abstract][Full Text] [Related]  

  • 16. TIMP2 deficiency accelerates adverse post-myocardial infarction remodeling because of enhanced MT1-MMP activity despite lack of MMP2 activation.
    Kandalam V; Basu R; Abraham T; Wang X; Soloway PD; Jaworski DM; Oudit GY; Kassiri Z
    Circ Res; 2010 Mar; 106(4):796-808. PubMed ID: 20056917
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Targeted deletion of class A macrophage scavenger receptor increases the risk of cardiac rupture after experimental myocardial infarction.
    Tsujita K; Kaikita K; Hayasaki T; Honda T; Kobayashi H; Sakashita N; Suzuki H; Kodama T; Ogawa H; Takeya M
    Circulation; 2007 Apr; 115(14):1904-11. PubMed ID: 17389263
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Targeted deletion of MMP-2 attenuates early LV rupture and late remodeling after experimental myocardial infarction.
    Hayashidani S; Tsutsui H; Ikeuchi M; Shiomi T; Matsusaka H; Kubota T; Imanaka-Yoshida K; Itoh T; Takeshita A
    Am J Physiol Heart Circ Physiol; 2003 Sep; 285(3):H1229-35. PubMed ID: 12775562
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Genetic deletion of 12/15 lipoxygenase promotes effective resolution of inflammation following myocardial infarction.
    Kain V; Ingle KA; Kabarowski J; Barnes S; Limdi NA; Prabhu SD; Halade GV
    J Mol Cell Cardiol; 2018 May; 118():70-80. PubMed ID: 29526491
    [TBL] [Abstract][Full Text] [Related]  

  • 20. ADAM15 is required for optimal collagen cross-linking and scar formation following myocardial infarction.
    Chute M; Aujla PK; Li Y; Jana S; Zhabyeyev P; Rasmuson J; Owen CA; Abraham T; Oudit GY; Kassiri Z
    Matrix Biol; 2022 Jan; 105():127-143. PubMed ID: 34995785
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 20.