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25. Kinetic mechanism of DNA unwinding by the BLM helicase core and molecular basis for its low processivity. Yang Y; Dou SX; Xu YN; Bazeille N; Wang PY; Rigolet P; Xu HQ; Xi XG Biochemistry; 2010 Feb; 49(4):656-68. PubMed ID: 20028084 [TBL] [Abstract][Full Text] [Related]
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29. Topo IIIalpha and BLM act within the Fanconi anemia pathway in response to DNA-crosslinking agents. Hemphill AW; Akkari Y; Newell AH; Schultz RA; Grompe M; North PS; Hickson ID; Jakobs PM; Rennie S; Pauw D; Hejna J; Olson SB; Moses RE Cytogenet Genome Res; 2009; 125(3):165-75. PubMed ID: 19738377 [TBL] [Abstract][Full Text] [Related]
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31. The Bloom's syndrome helicase suppresses crossing over during homologous recombination. Wu L; Hickson ID Nature; 2003 Dec; 426(6968):870-4. PubMed ID: 14685245 [TBL] [Abstract][Full Text] [Related]
32. Localization of the Bloom syndrome helicase to punctate nuclear structures and the nuclear matrix and regulation during the cell cycle: comparison with the Werner's syndrome helicase. Gharibyan V; Youssoufian H Mol Carcinog; 1999 Dec; 26(4):261-73. PubMed ID: 10569803 [TBL] [Abstract][Full Text] [Related]
33. BLM helicase unwinds lagging strand substrates to assemble the ALT telomere damage response. Jiang H; Zhang T; Kaur H; Shi T; Krishnan A; Kwon Y; Sung P; Greenberg RA Mol Cell; 2024 May; 84(9):1684-1698.e9. PubMed ID: 38593805 [TBL] [Abstract][Full Text] [Related]
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