214 related articles for article (PubMed ID: 23335989)
1. OASIS/CREB3L1 is induced by endoplasmic reticulum stress in human glioma cell lines and contributes to the unfolded protein response, extracellular matrix production and cell migration.
Vellanki RN; Zhang L; Volchuk A
PLoS One; 2013; 8(1):e54060. PubMed ID: 23335989
[TBL] [Abstract][Full Text] [Related]
2. OASIS/CREB3L1 induces expression of genes involved in extracellular matrix production but not classical endoplasmic reticulum stress response genes in pancreatic beta-cells.
Vellanki RN; Zhang L; Guney MA; Rocheleau JV; Gannon M; Volchuk A
Endocrinology; 2010 Sep; 151(9):4146-57. PubMed ID: 20668028
[TBL] [Abstract][Full Text] [Related]
3. Cleavage of the membrane-bound transcription factor OASIS in response to endoplasmic reticulum stress.
Murakami T; Kondo S; Ogata M; Kanemoto S; Saito A; Wanaka A; Imaizumi K
J Neurochem; 2006 Feb; 96(4):1090-100. PubMed ID: 16417584
[TBL] [Abstract][Full Text] [Related]
4. Induction of the unfolded protein response drives enhanced metabolism and chemoresistance in glioma cells.
Epple LM; Dodd RD; Merz AL; Dechkovskaia AM; Herring M; Winston BA; Lencioni AM; Russell RL; Madsen H; Nega M; Dusto NL; White J; Bigner DD; Nicchitta CV; Serkova NJ; Graner MW
PLoS One; 2013; 8(8):e73267. PubMed ID: 24039668
[TBL] [Abstract][Full Text] [Related]
5. OASIS/CREB3L1 is epigenetically silenced in human bladder cancer facilitating tumor cell spreading and migration in vitro.
Rose M; Schubert C; Dierichs L; Gaisa NT; Heer M; Heidenreich A; Knüchel R; Dahl E
Epigenetics; 2014 Dec; 9(12):1626-40. PubMed ID: 25625847
[TBL] [Abstract][Full Text] [Related]
6. OASIS regulates chondroitin 6-O-sulfotransferase 1 gene transcription in the injured adult mouse cerebral cortex.
Okuda H; Tatsumi K; Horii-Hayashi N; Morita S; Okuda-Yamamoto A; Imaizumi K; Wanaka A
J Neurochem; 2014 Sep; 130(5):612-25. PubMed ID: 24716865
[TBL] [Abstract][Full Text] [Related]
7. The tumorigenic role of DSPP and its potential regulation of the unfolded protein response and ER stress in oral cancer cells.
Gkouveris I; Nikitakis NG; Aseervatham J; Ogbureke KUE
Int J Oncol; 2018 Oct; 53(4):1743-1751. PubMed ID: 30015841
[TBL] [Abstract][Full Text] [Related]
8. Physiological unfolded protein response regulated by OASIS family members, transmembrane bZIP transcription factors.
Kondo S; Saito A; Asada R; Kanemoto S; Imaizumi K
IUBMB Life; 2011 Apr; 63(4):233-9. PubMed ID: 21438114
[TBL] [Abstract][Full Text] [Related]
9. Activation of OASIS family, ER stress transducers, is dependent on its stabilization.
Kondo S; Hino SI; Saito A; Kanemoto S; Kawasaki N; Asada R; Izumi S; Iwamoto H; Oki M; Miyagi H; Kaneko M; Nomura Y; Urano F; Imaizumi K
Cell Death Differ; 2012 Dec; 19(12):1939-49. PubMed ID: 22705851
[TBL] [Abstract][Full Text] [Related]
10. The endoplasmic reticulum stress transducer OASIS is involved in the terminal differentiation of goblet cells in the large intestine.
Asada R; Saito A; Kawasaki N; Kanemoto S; Iwamoto H; Oki M; Miyagi H; Izumi S; Imaizumi K
J Biol Chem; 2012 Mar; 287(11):8144-53. PubMed ID: 22262831
[TBL] [Abstract][Full Text] [Related]
11. Signalling mediated by the endoplasmic reticulum stress transducer OASIS is involved in bone formation.
Murakami T; Saito A; Hino S; Kondo S; Kanemoto S; Chihara K; Sekiya H; Tsumagari K; Ochiai K; Yoshinaga K; Saitoh M; Nishimura R; Yoneda T; Kou I; Furuichi T; Ikegawa S; Ikawa M; Okabe M; Wanaka A; Imaizumi K
Nat Cell Biol; 2009 Oct; 11(10):1205-11. PubMed ID: 19767743
[TBL] [Abstract][Full Text] [Related]
12. Deficiency for the ER-stress transducer OASIS causes severe recessive osteogenesis imperfecta in humans.
Symoens S; Malfait F; D'hondt S; Callewaert B; Dheedene A; Steyaert W; Bächinger HP; De Paepe A; Kayserili H; Coucke PJ
Orphanet J Rare Dis; 2013 Sep; 8():154. PubMed ID: 24079343
[TBL] [Abstract][Full Text] [Related]
13. Physiological functions of endoplasmic reticulum stress transducer OASIS in central nervous system.
Saito A
Anat Sci Int; 2014 Jan; 89(1):11-20. PubMed ID: 24242870
[TBL] [Abstract][Full Text] [Related]
14. Transcriptional regulation of VEGFA by the endoplasmic reticulum stress transducer OASIS in ARPE-19 cells.
Miyagi H; Kanemoto S; Saito A; Asada R; Iwamoto H; Izumi S; Kido M; Gomi F; Nishida K; Kiuchi Y; Imaizumi K
PLoS One; 2013; 8(1):e55155. PubMed ID: 23383089
[TBL] [Abstract][Full Text] [Related]
15. Homozygosity for CREB3L1 premature stop codon in first case of recessive osteogenesis imperfecta associated with OASIS-deficiency to survive infancy.
Lindahl K; Åström E; Dragomir A; Symoens S; Coucke P; Larsson S; Paschalis E; Roschger P; Gamsjaeger S; Klaushofer K; Fratzl-Zelman N; Kindmark A
Bone; 2018 Sep; 114():268-277. PubMed ID: 29936144
[TBL] [Abstract][Full Text] [Related]
16. The endoplasmic reticulum stress transducer old astrocyte specifically induced substance positively regulates glial scar formation in spinal cord injury.
Sumida Y; Kamei N; Suga N; Ochi M; Adachi N
Neuroreport; 2018 Dec; 29(17):1443-1448. PubMed ID: 30273224
[TBL] [Abstract][Full Text] [Related]
17. Roles of the endoplasmic reticulum stress transducer OASIS in fracture healing.
Funamoto T; Sekimoto T; Murakami T; Kurogi S; Imaizumi K; Chosa E
Bone; 2011 Oct; 49(4):724-32. PubMed ID: 21708301
[TBL] [Abstract][Full Text] [Related]
18. Comparison of expression patterns between CREB family transcription factor OASIS and proteoglycan core protein genes during murine tooth development.
Hikake T; Mori T; Iseki K; Hagino S; Zhang Y; Takagi H; Yokoya S; Wanaka A
Anat Embryol (Berl); 2003 Apr; 206(5):373-80. PubMed ID: 12684764
[TBL] [Abstract][Full Text] [Related]
19. Distinct mechanisms are responsible for osteopenia and growth retardation in OASIS-deficient mice.
Murakami T; Hino S; Nishimura R; Yoneda T; Wanaka A; Imaizumi K
Bone; 2011 Mar; 48(3):514-23. PubMed ID: 21047569
[TBL] [Abstract][Full Text] [Related]
20. [The regulation of unfolded protein response by OASIS, a transmembrane bZIP transcription factor, in astrocytes].
Imaizumi K; Tohyama M
Nihon Yakurigaku Zasshi; 2004 Dec; 124(6):383-90. PubMed ID: 15572842
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]