These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
410 related articles for article (PubMed ID: 23372648)
1. Accumulation of intraneuronal β-amyloid 42 peptides is associated with early changes in microtubule-associated protein 2 in neurites and synapses. Takahashi RH; Capetillo-Zarate E; Lin MT; Milner TA; Gouras GK PLoS One; 2013; 8(1):e51965. PubMed ID: 23372648 [TBL] [Abstract][Full Text] [Related]
3. Oligomerization of Alzheimer's beta-amyloid within processes and synapses of cultured neurons and brain. Takahashi RH; Almeida CG; Kearney PF; Yu F; Lin MT; Milner TA; Gouras GK J Neurosci; 2004 Apr; 24(14):3592-9. PubMed ID: 15071107 [TBL] [Abstract][Full Text] [Related]
4. Co-occurrence of Alzheimer's disease ß-amyloid and τ pathologies at synapses. Takahashi RH; Capetillo-Zarate E; Lin MT; Milner TA; Gouras GK Neurobiol Aging; 2010 Jul; 31(7):1145-52. PubMed ID: 18771816 [TBL] [Abstract][Full Text] [Related]
5. Amyloid β oligomers suppress excitatory transmitter release via presynaptic depletion of phosphatidylinositol-4,5-bisphosphate. He Y; Wei M; Wu Y; Qin H; Li W; Ma X; Cheng J; Ren J; Shen Y; Chen Z; Sun B; Huang FD; Shen Y; Zhou YD Nat Commun; 2019 Mar; 10(1):1193. PubMed ID: 30867420 [TBL] [Abstract][Full Text] [Related]
6. Familial Alzheimer's disease mutations at position 22 of the amyloid β-peptide sequence differentially affect synaptic loss, tau phosphorylation and neuronal cell death in an ex vivo system. Tackenberg C; Kulic L; Nitsch RM PLoS One; 2020; 15(9):e0239584. PubMed ID: 32966331 [TBL] [Abstract][Full Text] [Related]
7. Presynaptic dystrophic neurites surrounding amyloid plaques are sites of microtubule disruption, BACE1 elevation, and increased Aβ generation in Alzheimer's disease. Sadleir KR; Kandalepas PC; Buggia-Prévot V; Nicholson DA; Thinakaran G; Vassar R Acta Neuropathol; 2016 Aug; 132(2):235-256. PubMed ID: 26993139 [TBL] [Abstract][Full Text] [Related]
8. Soluble Conformers of Aβ and Tau Alter Selective Proteins Governing Axonal Transport. Sherman MA; LaCroix M; Amar F; Larson ME; Forster C; Aguzzi A; Bennett DA; Ramsden M; Lesné SE J Neurosci; 2016 Sep; 36(37):9647-58. PubMed ID: 27629715 [TBL] [Abstract][Full Text] [Related]
9. Tau-tubulin kinase 1 and amyloid-β peptide induce phosphorylation of collapsin response mediator protein-2 and enhance neurite degeneration in Alzheimer disease mouse models. Ikezu S; Ingraham Dixie KL; Koro L; Watanabe T; Kaibuchi K; Ikezu T Acta Neuropathol Commun; 2020 Feb; 8(1):12. PubMed ID: 32019603 [TBL] [Abstract][Full Text] [Related]
10. Dimeric amyloid beta protein rapidly accumulates in lipid rafts followed by apolipoprotein E and phosphorylated tau accumulation in the Tg2576 mouse model of Alzheimer's disease. Kawarabayashi T; Shoji M; Younkin LH; Wen-Lang L; Dickson DW; Murakami T; Matsubara E; Abe K; Ashe KH; Younkin SG J Neurosci; 2004 Apr; 24(15):3801-9. PubMed ID: 15084661 [TBL] [Abstract][Full Text] [Related]
11. The inside-out amyloid hypothesis and synapse pathology in Alzheimer's disease. Gouras GK; Willén K; Faideau M Neurodegener Dis; 2014; 13(2-3):142-6. PubMed ID: 24080821 [TBL] [Abstract][Full Text] [Related]
12. Active full-length DNA Aβ Rosenberg RN; Fu M; Lambracht-Washington D Alzheimers Res Ther; 2018 Nov; 10(1):115. PubMed ID: 30454039 [TBL] [Abstract][Full Text] [Related]
14. Reduction of aggregated Tau in neuronal processes but not in the cell bodies after Abeta42 immunisation in Alzheimer's disease. Boche D; Donald J; Love S; Harris S; Neal JW; Holmes C; Nicoll JA Acta Neuropathol; 2010 Jul; 120(1):13-20. PubMed ID: 20532897 [TBL] [Abstract][Full Text] [Related]
15. Heterogeneous Association of Alzheimer's Disease-Linked Amyloid-β and Amyloid-β Protein Precursor with Synapses. Willén K; Sroka A; Takahashi RH; Gouras GK J Alzheimers Dis; 2017; 60(2):511-524. PubMed ID: 28869466 [TBL] [Abstract][Full Text] [Related]
16. Synaptic Mitochondria: An Early Target of Amyloid-β and Tau in Alzheimer's Disease. Torres AK; Jara C; Park-Kang HS; Polanco CM; Tapia D; Alarcón F; de la Peña A; Llanquinao J; Vargas-Mardones G; Indo JA; Inestrosa NC; Tapia-Rojas C J Alzheimers Dis; 2021; 84(4):1391-1414. PubMed ID: 34719499 [TBL] [Abstract][Full Text] [Related]
17. Are N- and C-terminally truncated Aβ species key pathological triggers in Alzheimer's disease? Dunys J; Valverde A; Checler F J Biol Chem; 2018 Oct; 293(40):15419-15428. PubMed ID: 30143530 [TBL] [Abstract][Full Text] [Related]
18. RPS23RG1 reduces Aβ oligomer-induced synaptic and cognitive deficits. Yan L; Chen Y; Li W; Huang X; Badie H; Jian F; Huang T; Zhao Y; Cohen SN; Li L; Zhang YW; Luo H; Tu S; Xu H Sci Rep; 2016 Jan; 6():18668. PubMed ID: 26733416 [TBL] [Abstract][Full Text] [Related]
19. Insulin deficiency promotes formation of toxic amyloid-β42 conformer co-aggregating with hyper-phosphorylated tau oligomer in an Alzheimer's disease model. Imamura T; Yanagihara YT; Ohyagi Y; Nakamura N; Iinuma KM; Yamasaki R; Asai H; Maeda M; Murakami K; Irie K; Kira JI Neurobiol Dis; 2020 Apr; 137():104739. PubMed ID: 31927145 [TBL] [Abstract][Full Text] [Related]
20. Postsynaptic degeneration as revealed by PSD-95 reduction occurs after advanced Aβ and tau pathology in transgenic mouse models of Alzheimer's disease. Shao CY; Mirra SS; Sait HB; Sacktor TC; Sigurdsson EM Acta Neuropathol; 2011 Sep; 122(3):285-92. PubMed ID: 21630115 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]