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8. TAZ encodes tafazzin, a transacylase essential for cardiolipin formation and central to the etiology of Barth syndrome. Garlid AO; Schaffer CT; Kim J; Bhatt H; Guevara-Gonzalez V; Ping P Gene; 2020 Feb; 726():144148. PubMed ID: 31647997 [TBL] [Abstract][Full Text] [Related]
9. Loss of tafazzin results in decreased myoblast differentiation in C2C12 cells: A myoblast model of Barth syndrome and cardiolipin deficiency. Lou W; Reynolds CA; Li Y; Liu J; Hüttemann M; Schlame M; Stevenson D; Strathdee D; Greenberg ML Biochim Biophys Acta Mol Cell Biol Lipids; 2018 Aug; 1863(8):857-865. PubMed ID: 29694924 [TBL] [Abstract][Full Text] [Related]
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20. The human TAZ gene complements mitochondrial dysfunction in the yeast taz1Delta mutant. Implications for Barth syndrome. Ma L; Vaz FM; Gu Z; Wanders RJ; Greenberg ML J Biol Chem; 2004 Oct; 279(43):44394-9. PubMed ID: 15304507 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]