265 related articles for article (PubMed ID: 23530069)
1. FANCJ couples replication past natural fork barriers with maintenance of chromatin structure.
Schwab RA; Nieminuszczy J; Shin-ya K; Niedzwiedz W
J Cell Biol; 2013 Apr; 201(1):33-48. PubMed ID: 23530069
[TBL] [Abstract][Full Text] [Related]
2. A minimal threshold of FANCJ helicase activity is required for its response to replication stress or double-strand break repair.
Bharti SK; Sommers JA; Awate S; Bellani MA; Khan I; Bradley L; King GA; Seol Y; Vidhyasagar V; Wu Y; Abe T; Kobayashi K; Shin-Ya K; Kitao H; Wold MS; Branzei D; Neuman KC; Brosh RM
Nucleic Acids Res; 2018 Jul; 46(12):6238-6256. PubMed ID: 29788478
[TBL] [Abstract][Full Text] [Related]
3. MutSβ-MutLβ-FANCJ axis mediates the restart of DNA replication after fork stalling at cotranscriptional G4/R-loops.
Isik E; Shukla K; Pospisilova M; König C; Andrs M; Rao S; Rosano V; Dobrovolna J; Krejci L; Janscak P
Sci Adv; 2024 Feb; 10(6):eadk2685. PubMed ID: 38324687
[TBL] [Abstract][Full Text] [Related]
4. FANCJ promotes DNA synthesis through G-quadruplex structures.
Castillo Bosch P; Segura-Bayona S; Koole W; van Heteren JT; Dewar JM; Tijsterman M; Knipscheer P
EMBO J; 2014 Nov; 33(21):2521-33. PubMed ID: 25193968
[TBL] [Abstract][Full Text] [Related]
5. ATR activation and replication fork restart are defective in FANCM-deficient cells.
Schwab RA; Blackford AN; Niedzwiedz W
EMBO J; 2010 Feb; 29(4):806-18. PubMed ID: 20057355
[TBL] [Abstract][Full Text] [Related]
6. The vertebrate Hef ortholog is a component of the Fanconi anemia tumor-suppressor pathway.
Mosedale G; Niedzwiedz W; Alpi A; Perrina F; Pereira-Leal JB; Johnson M; Langevin F; Pace P; Patel KJ
Nat Struct Mol Biol; 2005 Sep; 12(9):763-71. PubMed ID: 16116434
[TBL] [Abstract][Full Text] [Related]
7. Specialization among iron-sulfur cluster helicases to resolve G-quadruplex DNA structures that threaten genomic stability.
Bharti SK; Sommers JA; George F; Kuper J; Hamon F; Shin-ya K; Teulade-Fichou MP; Kisker C; Brosh RM
J Biol Chem; 2013 Sep; 288(39):28217-29. PubMed ID: 23935105
[TBL] [Abstract][Full Text] [Related]
8. FANCM: fork pause, rewind and play.
Collis SJ; Boulton SJ
EMBO J; 2010 Feb; 29(4):703-5. PubMed ID: 20160754
[TBL] [Abstract][Full Text] [Related]
9. Purification and biochemical characterization of the G4 resolvase and DNA helicase FANCJ.
Kulikowicz T; Sommers JA; Fuchs KF; Wu Y; Brosh RM
Methods Enzymol; 2024; 695():1-27. PubMed ID: 38521581
[TBL] [Abstract][Full Text] [Related]
10. FANCD2, FANCJ and BRCA2 cooperate to promote replication fork recovery independently of the Fanconi Anemia core complex.
Raghunandan M; Chaudhury I; Kelich SL; Hanenberg H; Sobeck A
Cell Cycle; 2015; 14(3):342-53. PubMed ID: 25659033
[TBL] [Abstract][Full Text] [Related]
11. Multistep mechanism of G-quadruplex resolution during DNA replication.
Sato K; Martin-Pintado N; Post H; Altelaar M; Knipscheer P
Sci Adv; 2021 Sep; 7(39):eabf8653. PubMed ID: 34559566
[TBL] [Abstract][Full Text] [Related]
12. FANCJ (BACH1) helicase forms DNA damage inducible foci with replication protein A and interacts physically and functionally with the single-stranded DNA-binding protein.
Gupta R; Sharma S; Sommers JA; Kenny MK; Cantor SB; Brosh RM
Blood; 2007 Oct; 110(7):2390-8. PubMed ID: 17596542
[TBL] [Abstract][Full Text] [Related]
13. FANCJ helicase defective in Fanconia anemia and breast cancer unwinds G-quadruplex DNA to defend genomic stability.
Wu Y; Shin-ya K; Brosh RM
Mol Cell Biol; 2008 Jun; 28(12):4116-28. PubMed ID: 18426915
[TBL] [Abstract][Full Text] [Related]
14. Insight into the roles of helicase motif Ia by characterizing Fanconi anemia group J protein (FANCJ) patient mutations.
Guo M; Vidhyasagar V; Ding H; Wu Y
J Biol Chem; 2014 Apr; 289(15):10551-10565. PubMed ID: 24573678
[TBL] [Abstract][Full Text] [Related]
15. Opposing Roles of FANCJ and HLTF Protect Forks and Restrain Replication during Stress.
Peng M; Cong K; Panzarino NJ; Nayak S; Calvo J; Deng B; Zhu LJ; Morocz M; Hegedus L; Haracska L; Cantor SB
Cell Rep; 2018 Sep; 24(12):3251-3261. PubMed ID: 30232006
[TBL] [Abstract][Full Text] [Related]
16. Escherichia coli PriA helicase: fork binding orients the helicase to unwind the lagging strand side of arrested replication forks.
Jones JM; Nakai H
J Mol Biol; 2001 Oct; 312(5):935-47. PubMed ID: 11580240
[TBL] [Abstract][Full Text] [Related]
17. G-quadruplex recognition and remodeling by the FANCJ helicase.
Wu CG; Spies M
Nucleic Acids Res; 2016 Oct; 44(18):8742-8753. PubMed ID: 27342280
[TBL] [Abstract][Full Text] [Related]
18. Molecular and cellular functions of the FANCJ DNA helicase defective in cancer and in Fanconi anemia.
Brosh RM; Cantor SB
Front Genet; 2014; 5():372. PubMed ID: 25374583
[TBL] [Abstract][Full Text] [Related]
19. The HIRAN domain of helicase-like transcription factor positions the DNA translocase motor to drive efficient DNA fork regression.
Chavez DA; Greer BH; Eichman BF
J Biol Chem; 2018 Jun; 293(22):8484-8494. PubMed ID: 29643183
[TBL] [Abstract][Full Text] [Related]
20. FANCJ helicase uniquely senses oxidative base damage in either strand of duplex DNA and is stimulated by replication protein A to unwind the damaged DNA substrate in a strand-specific manner.
Suhasini AN; Sommers JA; Mason AC; Voloshin ON; Camerini-Otero RD; Wold MS; Brosh RM
J Biol Chem; 2009 Jul; 284(27):18458-70. PubMed ID: 19419957
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
