BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

434 related articles for article (PubMed ID: 23555296)

  • 1. The role of ATM in the deficiency in nonhomologous end-joining near telomeres in a human cancer cell line.
    Muraki K; Han L; Miller D; Murnane JP
    PLoS Genet; 2013 Mar; 9(3):e1003386. PubMed ID: 23555296
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Cell cycle-dependent role of MRN at dysfunctional telomeres: ATM signaling-dependent induction of nonhomologous end joining (NHEJ) in G1 and resection-mediated inhibition of NHEJ in G2.
    Dimitrova N; de Lange T
    Mol Cell Biol; 2009 Oct; 29(20):5552-63. PubMed ID: 19667071
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Differences in the recruitment of DNA repair proteins at subtelomeric and interstitial I-SceI endonuclease-induced DNA double-strand breaks.
    Alcaraz Silva B; Jones TJ; Murnane JP
    DNA Repair (Amst); 2017 Jan; 49():1-8. PubMed ID: 27842255
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Multiple roles for MRE11 at uncapped telomeres.
    Deng Y; Guo X; Ferguson DO; Chang S
    Nature; 2009 Aug; 460(7257):914-8. PubMed ID: 19633651
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Subtelomeric regions in mammalian cells are deficient in DNA double-strand break repair.
    Miller D; Reynolds GE; Mejia R; Stark JM; Murnane JP
    DNA Repair (Amst); 2011 May; 10(5):536-44. PubMed ID: 21466975
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Processing by MRE11 is involved in the sensitivity of subtelomeric regions to DNA double-strand breaks.
    Muraki K; Han L; Miller D; Murnane JP
    Nucleic Acids Res; 2015 Sep; 43(16):7911-30. PubMed ID: 26209132
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Protection of telomeres through independent control of ATM and ATR by TRF2 and POT1.
    Denchi EL; de Lange T
    Nature; 2007 Aug; 448(7157):1068-71. PubMed ID: 17687332
    [TBL] [Abstract][Full Text] [Related]  

  • 8. DNA-damage response and repair activities at uncapped telomeres depend on RNF8.
    Peuscher MH; Jacobs JJ
    Nat Cell Biol; 2011 Aug; 13(9):1139-45. PubMed ID: 21857671
    [TBL] [Abstract][Full Text] [Related]  

  • 9. No overt nucleosome eviction at deprotected telomeres.
    Wu P; de Lange T
    Mol Cell Biol; 2008 Sep; 28(18):5724-35. PubMed ID: 18625717
    [TBL] [Abstract][Full Text] [Related]  

  • 10. The DNA damage response at dysfunctional telomeres, and at interstitial and subtelomeric DNA double-strand breaks.
    Muraki K; Murnane JP
    Genes Genet Syst; 2018 Jan; 92(3):135-152. PubMed ID: 29162774
    [TBL] [Abstract][Full Text] [Related]  

  • 11. ATM limits incorrect end utilization during non-homologous end joining of multiple chromosome breaks.
    Bennardo N; Stark JM
    PLoS Genet; 2010 Nov; 6(11):e1001194. PubMed ID: 21079684
    [TBL] [Abstract][Full Text] [Related]  

  • 12. DNA processing is not required for ATM-mediated telomere damage response after TRF2 deletion.
    Celli GB; de Lange T
    Nat Cell Biol; 2005 Jul; 7(7):712-8. PubMed ID: 15968270
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Removal of shelterin reveals the telomere end-protection problem.
    Sfeir A; de Lange T
    Science; 2012 May; 336(6081):593-7. PubMed ID: 22556254
    [TBL] [Abstract][Full Text] [Related]  

  • 14. 53BP1 deficiency combined with telomere dysfunction activates ATR-dependent DNA damage response.
    Martínez P; Flores JM; Blasco MA
    J Cell Biol; 2012 Apr; 197(2):283-300. PubMed ID: 22508511
    [TBL] [Abstract][Full Text] [Related]  

  • 15. ATM damage response and XLF repair factor are functionally redundant in joining DNA breaks.
    Zha S; Guo C; Boboila C; Oksenych V; Cheng HL; Zhang Y; Wesemann DR; Yuen G; Patel H; Goff PH; Dubois RL; Alt FW
    Nature; 2011 Jan; 469(7329):250-4. PubMed ID: 21160472
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Increased sensitivity of subtelomeric regions to DNA double-strand breaks in a human cancer cell line.
    Zschenker O; Kulkarni A; Miller D; Reynolds GE; Granger-Locatelli M; Pottier G; Sabatier L; Murnane JP
    DNA Repair (Amst); 2009 Aug; 8(8):886-900. PubMed ID: 19540174
    [TBL] [Abstract][Full Text] [Related]  

  • 17. NBS1 Phosphorylation Status Dictates Repair Choice of Dysfunctional Telomeres.
    Rai R; Hu C; Broton C; Chen Y; Lei M; Chang S
    Mol Cell; 2017 Mar; 65(5):801-817.e4. PubMed ID: 28216226
    [TBL] [Abstract][Full Text] [Related]  

  • 18. The shelterin protein TRF2 inhibits Chk2 activity at telomeres in the absence of DNA damage.
    Buscemi G; Zannini L; Fontanella E; Lecis D; Lisanti S; Delia D
    Curr Biol; 2009 May; 19(10):874-9. PubMed ID: 19375317
    [TBL] [Abstract][Full Text] [Related]  

  • 19. MAD2L2 controls DNA repair at telomeres and DNA breaks by inhibiting 5' end resection.
    Boersma V; Moatti N; Segura-Bayona S; Peuscher MH; van der Torre J; Wevers BA; Orthwein A; Durocher D; Jacobs JJL
    Nature; 2015 May; 521(7553):537-540. PubMed ID: 25799990
    [TBL] [Abstract][Full Text] [Related]  

  • 20. The influence of heterochromatin on DNA double strand break repair: Getting the strong, silent type to relax.
    Goodarzi AA; Jeggo P; Lobrich M
    DNA Repair (Amst); 2010 Dec; 9(12):1273-82. PubMed ID: 21036673
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 22.