These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
198 related articles for article (PubMed ID: 23589030)
1. Clathrin adaptor CALM/PICALM is associated with neurofibrillary tangles and is cleaved in Alzheimer's brains. Ando K; Brion JP; Stygelbout V; Suain V; Authelet M; Dedecker R; Chanut A; Lacor P; Lavaur J; Sazdovitch V; Rogaeva E; Potier MC; Duyckaerts C Acta Neuropathol; 2013 Jun; 125(6):861-78. PubMed ID: 23589030 [TBL] [Abstract][Full Text] [Related]
2. Level of PICALM, a key component of clathrin-mediated endocytosis, is correlated with levels of phosphotau and autophagy-related proteins and is associated with tau inclusions in AD, PSP and Pick disease. Ando K; Tomimura K; Sazdovitch V; Suain V; Yilmaz Z; Authelet M; Ndjim M; Vergara C; Belkouch M; Potier MC; Duyckaerts C; Brion JP Neurobiol Dis; 2016 Oct; 94():32-43. PubMed ID: 27260836 [TBL] [Abstract][Full Text] [Related]
3. PICALM and Alzheimer's Disease: An Update and Perspectives. Ando K; Nagaraj S; Küçükali F; de Fisenne MA; Kosa AC; Doeraene E; Lopez Gutierrez L; Brion JP; Leroy K Cells; 2022 Dec; 11(24):. PubMed ID: 36552756 [TBL] [Abstract][Full Text] [Related]
5. Dystrophic (senescent) rather than activated microglial cells are associated with tau pathology and likely precede neurodegeneration in Alzheimer's disease. Streit WJ; Braak H; Xue QS; Bechmann I Acta Neuropathol; 2009 Oct; 118(4):475-85. PubMed ID: 19513731 [TBL] [Abstract][Full Text] [Related]
6. Phosphorylation of tau protein at sites Ser(396-404) is one of the earliest events in Alzheimer's disease and Down syndrome. Mondragón-Rodríguez S; Perry G; Luna-Muñoz J; Acevedo-Aquino MC; Williams S Neuropathol Appl Neurobiol; 2014 Feb; 40(2):121-35. PubMed ID: 24033439 [TBL] [Abstract][Full Text] [Related]
7. Neurofibrillary pathology and aluminum in Alzheimer's disease. Shin RW; Lee VM; Trojanowski JQ Histol Histopathol; 1995 Oct; 10(4):969-78. PubMed ID: 8574016 [TBL] [Abstract][Full Text] [Related]
8. Biochemical and anatomical redistribution of tau protein in Alzheimer's disease. Mukaetova-Ladinska EB; Harrington CR; Roth M; Wischik CM Am J Pathol; 1993 Aug; 143(2):565-78. PubMed ID: 8342603 [TBL] [Abstract][Full Text] [Related]
9. Relationship between genetic risk factors and markers for Alzheimer's disease pathology. Elias-Sonnenschein LS; Bertram L; Visser PJ Biomark Med; 2012 Aug; 6(4):477-95. PubMed ID: 22917148 [TBL] [Abstract][Full Text] [Related]
10. Carboxyl-terminal fragments of presenilin-1 are closely related to cytoskeletal abnormalities in Alzheimer's brains. Tomidokoro Y; Ishiguro K; Igeta Y; Matsubara E; Kanai M; Shizuka M; Kawarabayashi T; Harigaya Y; Kawakatsu S; Ii K; Ikeda M; St George-Hyslop PH; Hirai S; Okamoto K; Shoji M Biochem Biophys Res Commun; 1999 Mar; 256(3):512-8. PubMed ID: 10080929 [TBL] [Abstract][Full Text] [Related]
11. 14-3-3 protein beta isoform is associated with 3-repeat tau neurofibrillary tangles in Alzheimer's disease. Sugimori K; Kobayashi K; Kitamura T; Sudo S; Koshino Y Psychiatry Clin Neurosci; 2007 Apr; 61(2):159-67. PubMed ID: 17362433 [TBL] [Abstract][Full Text] [Related]
12. The role of PTB domain containing adaptor proteins on PICALM-mediated APP endocytosis and localization. Merthan L; Haller A; Thal DR; von Einem B; von Arnim CAF Biochem J; 2019 Jul; 476(14):2093-2109. PubMed ID: 31300465 [TBL] [Abstract][Full Text] [Related]
13. Regions with abundant neurofibrillary pathology in human brain exhibit a selective reduction in levels of binding-competent tau and accumulation of abnormal tau-isoforms (A68 proteins). Bramblett GT; Trojanowski JQ; Lee VM Lab Invest; 1992 Feb; 66(2):212-22. PubMed ID: 1735956 [TBL] [Abstract][Full Text] [Related]
14. Non-tau based neuronal degeneration in Alzheimer's disease -- an immunocytochemical and quantitative study in the supragranular layers of the middle temporal neocortex. van de Nes JA; Nafe R; Schlote W Brain Res; 2008 Jun; 1213():152-65. PubMed ID: 18455153 [TBL] [Abstract][Full Text] [Related]
15. Neurofibrillary tangles may interfere with Smad 2/3 signaling in neurons. Chalmers KA; Love S J Neuropathol Exp Neurol; 2007 Feb; 66(2):158-67. PubMed ID: 17279001 [TBL] [Abstract][Full Text] [Related]
16. Proteolytic cleavage of polymeric tau protein by caspase-3: implications for Alzheimer disease. Jarero-Basulto JJ; Luna-Muñoz J; Mena R; Kristofikova Z; Ripova D; Perry G; Binder LI; Garcia-Sierra F J Neuropathol Exp Neurol; 2013 Dec; 72(12):1145-61. PubMed ID: 24226268 [TBL] [Abstract][Full Text] [Related]
17. Molecular dissection of the neurofibrillary lesions of Alzheimer's disease. Goedert M Arzneimittelforschung; 1995 Mar; 45(3A):403-9. PubMed ID: 7763334 [TBL] [Abstract][Full Text] [Related]
18. Reduction of aggregated Tau in neuronal processes but not in the cell bodies after Abeta42 immunisation in Alzheimer's disease. Boche D; Donald J; Love S; Harris S; Neal JW; Holmes C; Nicoll JA Acta Neuropathol; 2010 Jul; 120(1):13-20. PubMed ID: 20532897 [TBL] [Abstract][Full Text] [Related]
19. Immunohistochemical study of tau accumulation in early stages of Alzheimer-type neurofibrillary lesions. Togo T; Akiyama H; Iseki E; Uchikado H; Kondo H; Ikeda K; Tsuchiya K; de Silva R; Lees A; Kosaka K Acta Neuropathol; 2004 Jun; 107(6):504-8. PubMed ID: 15024583 [TBL] [Abstract][Full Text] [Related]
20. Evolutional aspects of Alzheimer's disease pathogenesis. Braak H; Del Tredici K J Alzheimers Dis; 2013; 33 Suppl 1():S155-61. PubMed ID: 22699850 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]