These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

111 related articles for article (PubMed ID: 2373149)

  • 1. The effects of aminoguanidine on the glycation (non-enzymic glycosylation) of lens proteins.
    Lewis BS; Harding JJ
    Exp Eye Res; 1990 May; 50(5):463-7. PubMed ID: 2373149
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Ibuprofen, a putative anti-cataract drug, protects the lens against cyanate and galactose.
    Roberts KA; Harding JJ
    Exp Eye Res; 1990 Feb; 50(2):157-64. PubMed ID: 2311679
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Glycation-induced crosslinking of calf lens crystallins.
    van Boekel MA; Hoenders HJ
    Exp Eye Res; 1991 Jul; 53(1):89-94. PubMed ID: 1879506
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Non-enzymic glycosylation (glycation) of lens proteins by galactose and protection by aspirin and reduced glutathione.
    Huby R; Harding JJ
    Exp Eye Res; 1988 Jul; 47(1):53-9. PubMed ID: 3409987
    [TBL] [Abstract][Full Text] [Related]  

  • 5. The major metabolite of bendazac inhibits the glycosylation of soluble lens proteins: a possible mechanism for a delay in cataractogenesis.
    Lewis BS; Harding JJ
    Exp Eye Res; 1988 Aug; 47(2):217-25. PubMed ID: 3409993
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Aminoguanidine inhibits protein browning without extensive Amadori carbonyl blocking.
    Requena JR; Vidal P; Cabezas-Cerrato J
    Diabetes Res Clin Pract; 1993 Jan; 19(1):23-30. PubMed ID: 8472617
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Aspirin prevents the nonenzymatic glycosylation and carbamylation of the human eye lens crystallins in vitro.
    Rao GN; Cotlier E
    Biochem Biophys Res Commun; 1988 Mar; 151(3):991-6. PubMed ID: 3355566
    [TBL] [Abstract][Full Text] [Related]  

  • 8. The effectiveness of putative anti-cataract agents in the prevention of protein glycation.
    Stevens A
    J Am Optom Assoc; 1995 Dec; 66(12):744-9. PubMed ID: 8557952
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Aspirin prevents carbamylation of soluble lens proteins and prevents cyanate-induced phase separation opacities in vitro: a possible mechanism by which aspirin could prevent cataract.
    Crompton M; Rixon KC; Harding JJ
    Exp Eye Res; 1985 Feb; 40(2):297-311. PubMed ID: 3979467
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Binding of glucose, galactose and pyridoxal phosphate to lens crystallins.
    Ganea E; Rixon KC; Harding JJ
    Biochim Biophys Acta; 1994 Jul; 1226(3):286-90. PubMed ID: 8054360
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Bendazac prevents cyanate binding to soluble lens proteins and cyanate-induced phase-separation opacities in vitro: a possible mechanism by which bendazac could delay cataract.
    Lewis BS; Rixon KC; Harding JJ
    Exp Eye Res; 1986 Dec; 43(6):973-9. PubMed ID: 3817034
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Inhibition of 6-phosphogluconate dehydrogenase by carbamylation and protection by alpha-crystallin, a chaperone-like protein.
    Ganea E; Harding JJ
    Biochem Biophys Res Commun; 1996 May; 222(2):626-31. PubMed ID: 8670255
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Diabetic cataract formation: potential role of glycosylation of lens crystallins.
    Stevens VJ; Rouzer CA; Monnier VM; Cerami A
    Proc Natl Acad Sci U S A; 1978 Jun; 75(6):2918-22. PubMed ID: 275862
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Assessment of structure, stability and aggregation of soluble lens proteins and alpha-crystallin upon non-enzymatic glycation: The pathomechanisms underlying cataract development in diabetic patients.
    Yousefi R; Javadi S; Amirghofran S; Oryan A; Moosavi-Movahedi AA
    Int J Biol Macromol; 2016 Jan; 82():328-38. PubMed ID: 26478093
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Free lysine, glycine, alanine, glutamic acid and aspartic acid reduce the glycation of human lens proteins by galactose.
    Ramakrishnan S; Sulochana KN; Punitham R
    Indian J Biochem Biophys; 1997 Dec; 34(6):518-23. PubMed ID: 9594433
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Mechanistic studies of advanced glycosylation end product inhibition by aminoguanidine.
    Edelstein D; Brownlee M
    Diabetes; 1992 Jan; 41(1):26-9. PubMed ID: 1727735
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Effects of nonenzymatic glycosylation of mesangial matrix on proliferation of mesangial cells.
    Crowley ST; Brownlee M; Edelstein D; Satriano JA; Mori T; Singhal PC; Schlondorff DO
    Diabetes; 1991 May; 40(5):540-7. PubMed ID: 1708734
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Free alanine, aspartic acid, or glutamic acid reduce the glycation of human lens proteins.
    Ramakrishnan S; Sulochana KN; Punitham R; Arunagiri K
    Glycoconj J; 1996 Aug; 13(4):519-23. PubMed ID: 8872107
    [TBL] [Abstract][Full Text] [Related]  

  • 19. The non-enzymic glycosylation of bovine lens proteins by glucosamine and its inhibition by aspirin, ibuprofen and glutathione.
    Ajiboye R; Harding JJ
    Exp Eye Res; 1989 Jul; 49(1):31-41. PubMed ID: 2759189
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Decrease in glycation of lens proteins by lysine and glycine by scavenging of glucose and possible mitigation of cataractogenesis.
    Ramakrishnan S; Sulochana KN
    Exp Eye Res; 1993 Nov; 57(5):623-8. PubMed ID: 8282049
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 6.