142 related articles for article (PubMed ID: 23750002)
21. Ku counteracts mobilization of PARP1 and MRN in chromatin damaged with DNA double-strand breaks.
Cheng Q; Barboule N; Frit P; Gomez D; Bombarde O; Couderc B; Ren GS; Salles B; Calsou P
Nucleic Acids Res; 2011 Dec; 39(22):9605-19. PubMed ID: 21880593
[TBL] [Abstract][Full Text] [Related]
22. RAD18 and poly(ADP-ribose) polymerase independently suppress the access of nonhomologous end joining to double-strand breaks and facilitate homologous recombination-mediated repair.
Saberi A; Hochegger H; Szuts D; Lan L; Yasui A; Sale JE; Taniguchi Y; Murakawa Y; Zeng W; Yokomori K; Helleday T; Teraoka H; Arakawa H; Buerstedde JM; Takeda S
Mol Cell Biol; 2007 Apr; 27(7):2562-71. PubMed ID: 17242200
[TBL] [Abstract][Full Text] [Related]
23. PARP-1 and Ku compete for repair of DNA double strand breaks by distinct NHEJ pathways.
Wang M; Wu W; Wu W; Rosidi B; Zhang L; Wang H; Iliakis G
Nucleic Acids Res; 2006; 34(21):6170-82. PubMed ID: 17088286
[TBL] [Abstract][Full Text] [Related]
24. Toward a unified nomenclature for mammalian ADP-ribosyltransferases.
Hottiger MO; Hassa PO; Lüscher B; Schüler H; Koch-Nolte F
Trends Biochem Sci; 2010 Apr; 35(4):208-19. PubMed ID: 20106667
[TBL] [Abstract][Full Text] [Related]
25. PARP-3 and APLF function together to accelerate nonhomologous end-joining.
Rulten SL; Fisher AE; Robert I; Zuma MC; Rouleau M; Ju L; Poirier G; Reina-San-Martin B; Caldecott KW
Mol Cell; 2011 Jan; 41(1):33-45. PubMed ID: 21211721
[TBL] [Abstract][Full Text] [Related]
26. The absence of Ku but not defects in classical non-homologous end-joining is required to trigger PARP1-dependent end-joining.
Mansour WY; Borgmann K; Petersen C; Dikomey E; Dahm-Daphi J
DNA Repair (Amst); 2013 Dec; 12(12):1134-42. PubMed ID: 24210699
[TBL] [Abstract][Full Text] [Related]
27. Poly (ADP-ribose) polymerase (PARP) is not involved in base excision repair but PARP inhibition traps a single-strand intermediate.
Ström CE; Johansson F; Uhlén M; Szigyarto CA; Erixon K; Helleday T
Nucleic Acids Res; 2011 Apr; 39(8):3166-75. PubMed ID: 21183466
[TBL] [Abstract][Full Text] [Related]
28. PARP activation regulates the RNA-binding protein NONO in the DNA damage response to DNA double-strand breaks.
Krietsch J; Caron MC; Gagné JP; Ethier C; Vignard J; Vincent M; Rouleau M; Hendzel MJ; Poirier GG; Masson JY
Nucleic Acids Res; 2012 Nov; 40(20):10287-301. PubMed ID: 22941645
[TBL] [Abstract][Full Text] [Related]
29. Emerging models for DNA repair: Dictyostelium discoideum as a model for nonhomologous end-joining.
Pears CJ; Lakin ND
DNA Repair (Amst); 2014 May; 17():121-31. PubMed ID: 24548787
[TBL] [Abstract][Full Text] [Related]
30. Involvement of poly(ADP-ribose) polymerase-1 and XRCC1/DNA ligase III in an alternative route for DNA double-strand breaks rejoining.
Audebert M; Salles B; Calsou P
J Biol Chem; 2004 Dec; 279(53):55117-26. PubMed ID: 15498778
[TBL] [Abstract][Full Text] [Related]
31. Cells deficient in PARP-1 show an accelerated accumulation of DNA single strand breaks, but not AP sites, over the PARP-1-proficient cells exposed to MMS.
Pachkowski BF; Tano K; Afonin V; Elder RH; Takeda S; Watanabe M; Swenberg JA; Nakamura J
Mutat Res; 2009 Dec; 671(1-2):93-9. PubMed ID: 19778542
[TBL] [Abstract][Full Text] [Related]
32. Inhibition of poly (ADP-ribose) polymerase activates ATM which is required for subsequent homologous recombination repair.
Bryant HE; Helleday T
Nucleic Acids Res; 2006; 34(6):1685-91. PubMed ID: 16556909
[TBL] [Abstract][Full Text] [Related]
33. Homology and enzymatic requirements of microhomology-dependent alternative end joining.
Sharma S; Javadekar SM; Pandey M; Srivastava M; Kumari R; Raghavan SC
Cell Death Dis; 2015 Mar; 6(3):e1697. PubMed ID: 25789972
[TBL] [Abstract][Full Text] [Related]
34. DNA damage response factors from diverse pathways, including DNA crosslink repair, mediate alternative end joining.
Howard SM; Yanez DA; Stark JM
PLoS Genet; 2015 Jan; 11(1):e1004943. PubMed ID: 25629353
[TBL] [Abstract][Full Text] [Related]
35. Marked contribution of alternative end-joining to chromosome-translocation-formation by stochastically induced DNA double-strand-breaks in G2-phase human cells.
Soni A; Siemann M; Pantelias GE; Iliakis G
Mutat Res Genet Toxicol Environ Mutagen; 2015 Nov; 793():2-8. PubMed ID: 26520366
[TBL] [Abstract][Full Text] [Related]
36. Alternative NHEJ Pathway Components Are Therapeutic Targets in High-Risk Neuroblastoma.
Newman EA; Lu F; Bashllari D; Wang L; Opipari AW; Castle VP
Mol Cancer Res; 2015 Mar; 13(3):470-82. PubMed ID: 25563294
[TBL] [Abstract][Full Text] [Related]
37. XRCC1 and DNA polymerase beta in cellular protection against cytotoxic DNA single-strand breaks.
Horton JK; Watson M; Stefanick DF; Shaughnessy DT; Taylor JA; Wilson SH
Cell Res; 2008 Jan; 18(1):48-63. PubMed ID: 18166976
[TBL] [Abstract][Full Text] [Related]
38. SnapShot: ADP-Ribosylation Signaling.
Hottiger MO
Mol Cell; 2015 Jun; 58(6):1134-1134.e1. PubMed ID: 26091348
[TBL] [Abstract][Full Text] [Related]
39. Substrate-assisted catalysis by PARP10 limits its activity to mono-ADP-ribosylation.
Kleine H; Poreba E; Lesniewicz K; Hassa PO; Hottiger MO; Litchfield DW; Shilton BH; Lüscher B
Mol Cell; 2008 Oct; 32(1):57-69. PubMed ID: 18851833
[TBL] [Abstract][Full Text] [Related]
40. Towards a structural understanding of PARP1 activation and related signalling ADP-ribosyl-transferases.
Hassler M; Ladurner AG
Curr Opin Struct Biol; 2012 Dec; 22(6):721-9. PubMed ID: 22985748
[TBL] [Abstract][Full Text] [Related]
[Previous] [Next] [New Search]
