232 related articles for article (PubMed ID: 23755184)
1. Polycyclic aromatic hydrocarbon-induced signaling events relevant to inflammation and tumorigenesis in lung cells are dependent on molecular structure.
Osgood RS; Upham BL; Hill T; Helms KL; Velmurugan K; Babica P; Bauer AK
PLoS One; 2014; 8(6):e65150. PubMed ID: 23755184
[TBL] [Abstract][Full Text] [Related]
2. Secondhand Smoke-Prevalent Polycyclic Aromatic Hydrocarbon Binary Mixture-Induced Specific Mitogenic and Pro-inflammatory Cell Signaling Events in Lung Epithelial Cells.
Osgood RS; Upham BL; Bushel PR; Velmurugan K; Xiong KN; Bauer AK
Toxicol Sci; 2017 May; 157(1):156-171. PubMed ID: 28329830
[TBL] [Abstract][Full Text] [Related]
3. Polycyclic Aromatic Hydrocarbons and Endocrine Disruption: Role of Testicular Gap Junctional Intercellular Communication and Connexins.
Kubincová P; Sychrová E; Raška J; Basu A; Yawer A; Dydowiczová A; Babica P; Sovadinová I
Toxicol Sci; 2019 May; 169(1):70-83. PubMed ID: 30668803
[TBL] [Abstract][Full Text] [Related]
4. Interactive effects of inflammatory cytokine and abundant low-molecular-weight PAHs on inhibition of gap junctional intercellular communication, disruption of cell proliferation control, and the AhR-dependent transcription.
Kabátková M; Svobodová J; Pěnčíková K; Mohatad DS; Šmerdová L; Kozubík A; Machala M; Vondráček J
Toxicol Lett; 2015 Jan; 232(1):113-21. PubMed ID: 25268939
[TBL] [Abstract][Full Text] [Related]
5. Environmentally prevalent polycyclic aromatic hydrocarbons can elicit co-carcinogenic properties in an in vitro murine lung epithelial cell model.
Bauer AK; Velmurugan K; Plöttner S; Siegrist KJ; Romo D; Welge P; Brüning T; Xiong KN; Käfferlein HU
Arch Toxicol; 2018 Mar; 92(3):1311-1322. PubMed ID: 29170806
[TBL] [Abstract][Full Text] [Related]
6. Early Mechanistic Events Induced by Low Molecular Weight Polycyclic Aromatic Hydrocarbons in Mouse Lung Epithelial Cells: A Role for Eicosanoid Signaling.
Siegrist KJ; Romo D; Upham BL; Armstrong M; Quinn K; Vanderlinden L; Osgood RS; Velmurugan K; Elie M; Manke J; Reinhold D; Reisdorph N; Saba L; Bauer AK
Toxicol Sci; 2019 May; 169(1):180-193. PubMed ID: 30690640
[TBL] [Abstract][Full Text] [Related]
7. Airborne PAHs inhibit gap junctional intercellular communication and activate MAPKs in human bronchial epithelial cell line.
Brózman O; Novák J; Bauer AK; Babica P
Environ Toxicol Pharmacol; 2020 Oct; 79():103422. PubMed ID: 32492535
[TBL] [Abstract][Full Text] [Related]
8. Tumor promoting properties of a cigarette smoke prevalent polycyclic aromatic hydrocarbon as indicated by the inhibition of gap junctional intercellular communication via phosphatidylcholine-specific phospholipase C.
Upham BL; Bláha L; Babica P; Park JS; Sovadinova I; Pudrith C; Rummel AM; Weis LM; Sai K; Tithof PK; Guzvić M; Vondrácek J; Machala M; Trosko JE
Cancer Sci; 2008 Apr; 99(4):696-705. PubMed ID: 18377422
[TBL] [Abstract][Full Text] [Related]
9. Polycyclic aromatic hydrocarbons with bay-like regions inhibited gap junctional intercellular communication and stimulated MAPK activity.
Rummel AM; Trosko JE; Wilson MR; Upham BL
Toxicol Sci; 1999 Jun; 49(2):232-40. PubMed ID: 10416268
[TBL] [Abstract][Full Text] [Related]
10. Bay or baylike regions of polycyclic aromatic hydrocarbons were potent inhibitors of Gap junctional intercellular communication.
Weis LM; Rummel AM; Masten SJ; Trosko JE; Upham BL
Environ Health Perspect; 1998 Jan; 106(1):17-22. PubMed ID: 9417772
[TBL] [Abstract][Full Text] [Related]
11. Cigarette smoke components inhibited intercellular communication and differentiation in human pancreatic ductal epithelial cells.
Tai MH; Upham BL; Olson LK; Tsao MS; Reed DN; Trosko JE
Int J Cancer; 2007 May; 120(9):1855-62. PubMed ID: 17266035
[TBL] [Abstract][Full Text] [Related]
12. Inhibition of gap-junctional intercellular communication by environmentally occurring polycyclic aromatic hydrocarbons.
Bláha L; Kapplová P; Vondrácek J; Upham B; Machala M
Toxicol Sci; 2002 Jan; 65(1):43-51. PubMed ID: 11752684
[TBL] [Abstract][Full Text] [Related]
13. Aberrant Connexin 43 endocytosis by the carcinogen lindane involves activation of the ERK/mitogen-activated protein kinase pathway.
Mograbi B; Corcelle E; Defamie N; Samson M; Nebout M; Segretain D; Fénichel P; Pointis G
Carcinogenesis; 2003 Aug; 24(8):1415-23. PubMed ID: 12807735
[TBL] [Abstract][Full Text] [Related]
14. Modulated gap junctional intercellular communication as a biomarker of PAH epigenetic toxicity: structure-function relationship.
Upham BL; Weis LM; Trosko JE
Environ Health Perspect; 1998 Aug; 106 Suppl 4(Suppl 4):975-81. PubMed ID: 9703481
[TBL] [Abstract][Full Text] [Related]
15. Nerve growth factor increases connexin43 phosphorylation and gap junctional intercellular communication.
Cushing P; Bhalla R; Johnson AM; Rushlow WJ; Meakin SO; Belliveau DJ
J Neurosci Res; 2005 Dec; 82(6):788-801. PubMed ID: 16302187
[TBL] [Abstract][Full Text] [Related]
16. Dysregulation of Gap Junction Function and Cytokine Production in Response to Non-Genotoxic Polycyclic Aromatic Hydrocarbons in an In Vitro Lung Cell Model.
Romo D; Velmurugan K; Upham BL; Dwyer-Nield LD; Bauer AK
Cancers (Basel); 2019 Apr; 11(4):. PubMed ID: 31018556
[TBL] [Abstract][Full Text] [Related]
17. Regulation of epidermal growth factor-induced connexin 43 gap junction communication by big mitogen-activated protein kinase1/ERK5 but not ERK1/2 kinase activation.
Cameron SJ; Malik S; Akaike M; Lerner-Marmarosh N; Yan C; Lee JD; Abe J; Yang J
J Biol Chem; 2003 May; 278(20):18682-8. PubMed ID: 12637502
[TBL] [Abstract][Full Text] [Related]
18. Pressure induces loss of gap junction communication and redistribution of connexin 43 in astrocytes.
Malone P; Miao H; Parker A; Juarez S; Hernandez MR
Glia; 2007 Aug; 55(10):1085-98. PubMed ID: 17551925
[TBL] [Abstract][Full Text] [Related]
19. The roles of ERK1/2 and p38 MAP kinases in the preventive mechanisms of mushroom Phellinus linteus against the inhibition of gap junctional intercellular communication by hydrogen peroxide.
Cho JH; Cho SD; Hu H; Kim SH; Lee SK; Lee YS; Kang KS
Carcinogenesis; 2002 Jul; 23(7):1163-9. PubMed ID: 12117774
[TBL] [Abstract][Full Text] [Related]
20. Aryl hydrocarbon receptor-mediated disruption of contact inhibition is associated with connexin43 downregulation and inhibition of gap junctional intercellular communication.
Andrysík Z; Procházková J; Kabátková M; Umannová L; Simečková P; Kohoutek J; Kozubík A; Machala M; Vondráček J
Arch Toxicol; 2013 Mar; 87(3):491-503. PubMed ID: 23085979
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]